Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Conner J. Hansen"'
Autor:
Zohar Sachs, David A. Largaespada, Chad L. Myers, Michael A. Linden, Matthew L. Schomaker, Matthew M. Meredith, Michael R. Verneris, Rachel J. Bergerson, Fatma K. Karadag, Conner J. Hansen, Ngoc A. Ha, Ian R. Nykaza, Alexandria L. Hillesheim, Connor M. Navis, Yoonkyu Lee, Daniel Chang, Marie Lue Antony, Rebecca S. LaRue, Klara E. Noble-Orcutt, Aaron L. Sarver, Karen Sachs
Supplementary Materials and Methods and Figures. Suppl Figs: SF1. Workflow; SF2: Cell Surface Markers; SF3: HSPC Markers; SF4: Human AML, SF5: Sorting Strategy, SF6: Post-sort; SF7: Viability and Immunophenotype; SF8: CD36 and CD69 in Human AML
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::25a68d2ddb5981e42b2ca9e713153494
https://doi.org/10.1158/0008-5472.22424277.v1
https://doi.org/10.1158/0008-5472.22424277.v1
Autor:
Zohar Sachs, David A. Largaespada, Chad L. Myers, Michael A. Linden, Matthew L. Schomaker, Matthew M. Meredith, Michael R. Verneris, Rachel J. Bergerson, Fatma K. Karadag, Conner J. Hansen, Ngoc A. Ha, Ian R. Nykaza, Alexandria L. Hillesheim, Connor M. Navis, Yoonkyu Lee, Daniel Chang, Marie Lue Antony, Rebecca S. LaRue, Klara E. Noble-Orcutt, Aaron L. Sarver, Karen Sachs
Supplementary Tables: ST1: Differentially Expressed Genes; ST2: IPA, ST3: Clinical Data, ST4: Mutational Profiles, ST5: qPCR Gene Panel, ST6: qPCR Enriched, ST7: NGS Gene panel
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::60483820bbf9de505f52a46c5156587e
https://doi.org/10.1158/0008-5472.22424274.v1
https://doi.org/10.1158/0008-5472.22424274.v1
Autor:
Zohar Sachs, David A. Largaespada, Chad L. Myers, Michael A. Linden, Matthew L. Schomaker, Matthew M. Meredith, Michael R. Verneris, Rachel J. Bergerson, Fatma K. Karadag, Conner J. Hansen, Ngoc A. Ha, Ian R. Nykaza, Alexandria L. Hillesheim, Connor M. Navis, Yoonkyu Lee, Daniel Chang, Marie Lue Antony, Rebecca S. LaRue, Klara E. Noble-Orcutt, Aaron L. Sarver, Karen Sachs
Standard chemotherapy for acute myeloid leukemia (AML) targets proliferative cells and efficiently induces complete remission; however, many patients relapse and die of their disease. Relapse is caused by leukemia stem cells (LSC), the cells with sel
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6e81b3f262f9c3c84ead08aab90f0989
https://doi.org/10.1158/0008-5472.c.6511725.v1
https://doi.org/10.1158/0008-5472.c.6511725.v1