Zobrazeno 1 - 10
of 270
pro vyhledávání: '"Compensatory Hyperinsulinemia"'
Autor:
Marina B. Khamoshina, Yulia S. Artemenko, Ayshan A. Bayramova, Valentina A. Ryabova, Mekan R. Orazov
Publikováno v:
RUDN Journal of Medicine, Vol 26, Iss 4, Pp 382-395 (2022)
Polycystic ovary syndrome is a heterogeneous endocrine disease that affects women of childbearing age. The pathogenesis of polycystic ovary syndrome has not been fully studied to date, its paradigm considers the genetic determinism of the manifestati
Externí odkaz:
https://doaj.org/article/bcc3d123b0874ffa9001f122a8eaaf59
Publikováno v:
Journal of obstetrics and women's diseases. 70:51-63
BACKGROUND: The imbalance of vascular endothelial cell metabolism determines the clinical manifestations of preeclampsia; however, the molecular mechanisms underlying the vessel destabilization are not fully understood. In recent years, researchers h
Autor:
Sumitra Yadav, Manisha Gupta
Publikováno v:
Indian Journal of Obstetrics and Gynecology Research. 8:305-309
The importance of insulin resistance, compensatory hyperinsulinemia, and its effects, many of which have adverse effects on both the metabolic and reproductive organs. Treatment options for insulin resistance/hyperinsulinemia include lifestyle change
Publikováno v:
Ожирение и метаболизм, Vol 9, Iss 4, Pp 23-27 (2012)
The article presents the results of evaluation of vitamin D3, osteoprotegerin, carbohydrate and fat metabolic parameters in women with type 2 diabetes and obesity. The study subjects showed an increase of osteoprotegerin, decrease of vitamin D3, insu
Externí odkaz:
https://doaj.org/article/702bf0e5a8984064a29c4150e8b1c623
Autor:
Brett D. Hollingshead, Anna K. Kopec, Yutian Zhan, Timothy P. LaBranche, John W. Davis, Kyle Hayes, Srinivasa R. Mantena, Andrew W. Harrington, Laurence O. Whiteley, Zachary S. Stewart, Andrew D. Burdick
Publikováno v:
Toxicologic Pathology. 48:994-1007
Fatty liver disease is a potential risk factor for drug-induced liver injury (DILI). Despite advances in nonclinical in vitro and in vivo models to assess liver injury during drug development, the pharmaceutical industry is still plagued by idiosyncr
Publikováno v:
Clinical Endocrinology. 93:163-172
Context Polycystic ovary syndrome (PCOS) is a highly prevalent disorder associated with insulin resistance (IR) and compensatory hyperinsulinemia. Although variations in cardiometabolic risks across race and ethnicities have been reported in the gene
Autor:
Andrea Kelly, Diva D. De León, Saba Sheikh, Marissa J. Kilberg, Christina Kubrak, Denis Hadjiliadis, Darko Stefanovski, Ronald C. Rubenstein, Michael R. Rickels
Publikováno v:
J Cyst Fibros
BACKGROUND: Post-prandial and oral glucose tolerance test-related hypoglycemia is common in cystic fibrosis (CF); however, the underlying mechanisms are unclear. METHODS: To understand the relationship of hypoglycemia with meal-related glucose excurs
Autor:
Luisa Escobar, James González, Olga M. Echeverría, Nayeli Torres-Ramírez, Rosario Ortiz-Hernández, Emilio Espinoza-Simón, Jenifer Lizbet Hernández-Jiménez, David Barrera
Publikováno v:
Gynecological endocrinology : the official journal of the International Society of Gynecological Endocrinology. 38(1)
Polycystic ovary syndrome (PCOS) is a disease whose diagnosis is based on the detection of hyperandrogenism (HA) and ovulatory dysfunction. Women with PCOS frequently develop insulin resistance (IR), which generates a metabolic condition that involve
Autor:
Akio Ohashi, Ichiro Kishimoto
Publikováno v:
Nutrients
Volume 13
Issue 9
Nutrients, Vol 13, Iss 3092, p 3092 (2021)
Volume 13
Issue 9
Nutrients, Vol 13, Iss 3092, p 3092 (2021)
Data regarding hyperglycemia-related factors were scarce in people without diabetes. Fifty males (age 50–65 years) with overweight/obesity but without diagnosis of diabetes were recruited. After excluding participants with the 2 h plasma glucose va
Publikováno v:
Biomolecules, Vol 11, Iss 1350, p 1350 (2021)
Biomolecules
Biomolecules
Polycystic ovary syndrome (PCOS) is extremely heterogeneous in terms of clinical manifestations. The variability of the syndrome’s phenotype is derived from the genetic and molecular heterogeneity, with a great deal of environmental factors that ma