Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Colleen M. Urben"'
Autor:
S.A. Strugnell, Annemiete W.C.M. van der Kemp, René J. M. Bindels, Colleen M. Urben, Joost G. J. Hoenderop
Publikováno v:
Kidney International. 66:1082-1089
Effects of vitamin D compounds on renal and intestinal Ca 2+ transport proteins in 25-hydroxyvitamin D 3 -1α-hydroxylase knockout mice. Background Vitamin D compounds are used clinically to control secondary hyperparathyroidism (SHPT) due to renal f
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::25c810e14f249681d55424fd006d882d
https://doi.org/10.1111/j.1523-1755.2004.00858.x
https://doi.org/10.1111/j.1523-1755.2004.00858.x
Autor:
Glendon M, Zinser, Emily, Tribble, Meggan, Valrance, Colleen M, Urben, Joyce C, Knutson, Richard B, Mazess, Stephen A, Strugnell, Joellen, Welsh
Publikováno v:
Anticancer research. 25(1A)
1,24-Dihydroxyvitamin D2 (1,24(OH)2D2) is a naturally occurring metabolite of vitamin D2 with low calcemic activity and potent antiproliferative activity. We evaluated the activity of 1,24(OH)2D2 in breast cancer models.The antiproliferative activity
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::873b69094778b858bdaa8ce7dd6c0afa
https://pubmed.ncbi.nlm.nih.gov/15816543
https://pubmed.ncbi.nlm.nih.gov/15816543
Autor:
Joost G J, Hoenderop, Annemiete W C M, van der Kemp, Colleen M, Urben, Stephen A, Strugnell, René J M, Bindels
Publikováno v:
Kidney international. 66(3)
Vitamin D compounds are used clinically to control secondary hyperparathyroidism (SHPT) due to renal failure. Newer vitamin D compounds retain the suppressive action of 1,25(OH)(2)D(3) on the parathyroid glands and may have less Ca(2+)-mobilizing act
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::a3554935f6e10c63f638ce80f0b3c251
https://pubmed.ncbi.nlm.nih.gov/15327430
https://pubmed.ncbi.nlm.nih.gov/15327430
Publikováno v:
Brain research. Molecular brain research. 92(1-2)
Brain corticotropin-releasing factor (CRF) systems integrate various responses to stress. Pathological responses to stress may result from errors in CRF receptor regulation in response to changes in synaptic CRF levels. To establish an in vitro model
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0c9818725f4d2071e006effe38202ad3
https://pubmed.ncbi.nlm.nih.gov/11483248
https://pubmed.ncbi.nlm.nih.gov/11483248