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pro vyhledávání: '"Collados Rodriguez, Mila"'
Autor:
Pinto, Rute Maria, Bakshi, Siddharth, Lytras, Spyros, Zakaria, Mohammad Khalid, Swingler, Simon, Worrell, Julie C, Herder, Vanessa, Hargrave, Kerrie E, Varjak, Margus, Cameron-Ruiz, Natalia, Collados Rodriguez, Mila, Varela, Mariana, Wickenhagen, Arthur, Loney, Colin, Pei, Yanlong, Hughes, Joseph, Valette, Elise, Turnbull, Matthew L, Furnon, Wilhelm, Gu, Quan, Orr, Lauren, Taggart, Aislynn, Diebold, Ola, Davis, Chris, Boutell, Chris, Grey, Finn, Hutchinson, Edward, Digard, Paul, Monne, Isabella, Wootton, Sarah K, MacLeod, Megan K L, Wilson, Sam J, Palmarini, Massimo
Publikováno v:
Pinto, R M, Bakshi, S, Lytras, S, Zakaria, M K, Swingler, S, Worrell, J C, Herder, V, Hargrave, K E, Varjak, M, Cameron-Ruiz, N, Collados Rodriguez, M, Varela, M, Wickenhagen, A, Loney, C, Pei, Y, Hughes, J, Valette, E, Turnbull, M L, Furnon, W, Gu, Q, Orr, L, Taggart, A, Diebold, O, Davis, C, Boutell, C, Grey, F, Hutchinson, E, Digard, P, Monne, I, Wootton, S K, MacLeod, M K L, Wilson, S J & Palmarini, M 2023, ' BTN3A3 evasion promotes the zoonotic potential of influenza A viruses ', Nature, vol. 619, no. 7969, pp. 338-347 . https://doi.org/10.1038/s41586-023-06261-8
Spillover events of avian influenza A viruses (IAVs) to humans could represent the first step in a future pandemic1. Several factors that limit the transmission and replication of avian IAVs in mammals have been identified. There are several gaps in
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od______3094::5b6c7466df9e3bc3a262f90aed57c814
https://www.pure.ed.ac.uk/ws/files/362204359/s41586_023_06261_8_1_.pdf
https://www.pure.ed.ac.uk/ws/files/362204359/s41586_023_06261_8_1_.pdf
Autor:
Collados Rodriguez, Mila
Publikováno v:
Frontiers in Cellular and Infection Microbiology, Vol 10 (2021)
Mila Collados Rodríguez
Frontiers in Cellular and Infection Microbiology
Mila Collados Rodríguez
Frontiers in Cellular and Infection Microbiology
The constitutive expression ofSpeckled-100(Sp100) is known to restrict the replication of many clinically important DNA viruses. This pre-existing (intrinsic) immune defense to virus infection can be further upregulated uponinterferon(IFN) stimulatio