Zobrazeno 1 - 10
of 30
pro vyhledávání: '"Clovis de Carvalho Frimm"'
Publikováno v:
Arquivos Brasileiros de Cardiologia, Vol 80, Iss 5, Pp 515-520 (2003)
OBJECTIVE: To investigate the role of hemodynamic changes occurring during acute MI in subsequent fibrosis deposition within non-MI. METHODS: By using the rat model of MI, 3 groups of 7 rats each [sham, SMI (MI 30%)] were compared. Systemic and left
Externí odkaz:
https://doaj.org/article/2b1b49cd9f474dcc8b32cbb203cd375b
Autor:
Barbara Maria Ianni, Edmundo Arteaga, Clovis de Carvalho Frimm, Antonio Carlos Pereira Barretto, Charles Mady
Publikováno v:
Arquivos Brasileiros de Cardiologia, Vol 77, Iss 1, Pp 59-62 (2001)
OBJECTIVE: To identify and associate potential electrocardiographic and echocardiographic changes in patients with the indeterminate form of Chagas' disease during long-term follow-up. METHODS: One hundred sixty patients underwent standard electrocar
Externí odkaz:
https://doaj.org/article/bf13951e4f4d416ca5dc8f772002e5ea
Publikováno v:
Arquivos Brasileiros de Cardiologia, Vol 75, Iss 2, Pp 148-150 (2000)
A 59-year-old woman presented with an embolic transient ischemic attack and a history of controlled hypertension for 16 years. Both echocardiogram and MRI showed severe biventricular hypertrophy and an apical aneurysm with a thrombus. The occurrence
Externí odkaz:
https://doaj.org/article/a19869ef2bcb4f9aa3ee2216d5283321
Autor:
Mariana Cúri, Ana Clara Tudi Rodrigues, Valéria Fontenelle Angelim Pereira, Clovis de Carvalho Frimm
Publikováno v:
Journal of the American Society of Hypertension. 4:14-21
In hypertension, left ventricular (LV) hypertrophy develops as an adaptive mechanism to compensate for increased afterload and thus preserve systolic function. Associated structural changes such as microvascular disease might potentially interfere wi
Publikováno v:
International Journal of Experimental Pathology. 88:279-290
Summary Subendocardial remodelling of the left ventricular (LV) non-infarcted myocardium has been poorly investigated. Previously, we have demonstrated that low coronary driving pressure (CDP) early postinfarction was associated with the subsequent d
Publikováno v:
Clinical and Experimental Pharmacology and Physiology. 32:1131-1136
Bradykinin may interfere with myocardial remodelling by promoting inflammation and mast cell activation or, alternatively, by counteracting angiotensin II-dependent collagen accumulation. The aim of the present study was to investigate the role of br
Autor:
Wilson Mathias, Jeane M. Tsutsui, Edimar Alcides Bocchi, Fernando Bacal, Silvia G. Lage, Clovis de Carvalho Frimm, Ana Clara Tude Rodrigues, Vanessa Andreolli
Publikováno v:
International Journal of Cardiology. 103:201-206
The impact of allograft vasculopathy on the coronary circulation and consequently on cardiac outcome may be expressed by coronary flow reserve (CFR) impairment. Therefore, we aimed to evaluate CFR and its relation to cardiac events in heart transplan
Autor:
Ana Clara Tude Rodrigues, Clovis de Carvalho Frimm, Jeane M. Tsutsui, Mariana Cúri, Valéria Fontenelle Angelim Pereira
Publikováno v:
Clinical Cardiology. 28:19-24
BACKGROUND The pathophysiologic role of coronary reserve impairment in hypertensive cardiac dysfunction is still debated. Previously, we demonstrated that satisfactory coronary vasodilatation may coexist with ventricular systolic dysfunction. It is c
Publikováno v:
Revista do Hospital das Clínicas v.59 n.5 2004
Revista do Hospital das Clínicas
Universidade de São Paulo (USP)
instacron:USP
Revista do Hospital das Clínicas
Universidade de São Paulo (USP)
instacron:USP
Left ventricular hypertrophy following volume overload is regarded as an example of cardiac remodeling without increased fibrosis accumulation. However, infarction is associated with increased fibrosis within the noninfarcted, hypertrophied myocardiu
Publikováno v:
Revista do Hospital das Clínicas, Vol 59, Iss 5, Pp 228-235 (2004)
Left ventricular hypertrophy following volume overload is regarded as an example of cardiac remodeling without increased fibrosis accumulation. However, infarction is associated with increased fibrosis within the noninfarcted, hypertrophied myocardiu