Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Clement Kin-ming Tsui"'
Publikováno v:
IMA Fungus, Vol 15, Iss 1, Pp 1-15 (2024)
Abstract Chrysomyxa rusts cause significant damage to spruce in both natural forests and plantations. Particularly, Three Chrysomyxa species, Chrysomyxa deformans, Chrysomyxa qilianensis, and Chrysomyxa rhododendri, listed as National Forest Dangerou
Externí odkaz:
https://doaj.org/article/47b7a346403d429ea5740586989cee21
Autor:
Clement Kin-Ming Tsui, Lina Farfan, Amanda D Roe, Adrianne V Rice, Janice E K Cooke, Yousry A El-Kassaby, Richard C Hamelin
Publikováno v:
PLoS ONE, Vol 9, Iss 8, p e105455 (2014)
Over 18 million ha of forests have been destroyed in the past decade in Canada by the mountain pine beetle (MPB) and its fungal symbionts. Understanding their population dynamics is critical to improving modeling of beetle epidemics and providing pot
Externí odkaz:
https://doaj.org/article/af5db037774842bbb3552e718a3bec33
Autor:
Clement Kin-Ming Tsui, Fatma Ben Abid, Christi Lee McElheny, Muna Almuslamani, Ali S. Omrani, Yohei Doi
The emergence of carbapenem-resistant (CR) hypervirulent Klebsiella pneumoniae (hvKp) is a new threat to healthcare. In this study, we studied the molecular epidemiology of CR Klebsiella isolates in Qatar using whole genome sequence data. We also cha
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::174dd461e33f4f645d88b15705069b80
https://doi.org/10.1101/2022.06.02.494628
https://doi.org/10.1101/2022.06.02.494628
Autor:
Clement Kin-ming Tsui, Mary L. Berbee
Publikováno v:
Mycoscience. 51:144-148
Autor:
Susanne, Zeilinger, Vijai K, Gupta, Tanya E S, Dahms, Roberto N, Silva, Harikesh B, Singh, Ram S, Upadhyay, Eriston Vieira, Gomes, Clement Kin-Ming, Tsui, Chandra, Nayak S
Publikováno v:
FEMS Microbiology Reviews
Fungi interact with plants in various ways, with each interaction giving rise to different alterations in both partners. While fungal pathogens have detrimental effects on plant physiology, mutualistic fungi augment host defence responses to pathogen