Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Claudine P. Black"'
Autor:
Megan A. Ahern, Claudine P. Black, Gregory J. Seedorf, Christopher D. Baker, Douglas P. Shepherd
Publikováno v:
AIMS Biophysics, Vol 4, Iss 2, Pp 284-297 (2017)
Disruptions in the response of endothelial progenitor cells to changes in oxygen environment may present a possible mechanism behind multiple pediatric pulmonary disease models, such as bronchopulmonary dysplasia. Using high-throughput fixed single-c
Externí odkaz:
https://doaj.org/article/53fb01b6fe4e41e8bb1b51706e1af532
Autor:
Douglas P. Shepherd, Christopher D. Baker, Claudine P. Black, Megan A. Ahern, Gregory J. Seedorf
Publikováno v:
AIMS Biophysics, Vol 4, Iss 2, Pp 284-297 (2017)
Disruptions in the response of endothelial progenitor cells to changes in oxygen environment may present a possible mechanism behind multiple pediatric pulmonary disease models, such as bronchopulmonary dysplasia. Using high-throughput fixed single-c
Autor:
Steven H. Abman, Claudine P. Black, Gregory J. Seedorf, Benjamin L. Wisniewski, Christopher D. Baker, Sharon L. Ryan, Vivek Balasubramaniam
Publikováno v:
American Journal of Physiology-Lung Cellular and Molecular Physiology. 305:L73-L81
Late-outgrowth endothelial colony-forming cells (ECFCs), a type of circulating endothelial progenitor cell (EPC), may contribute to pulmonary angiogenesis during development. Cord blood ECFCs from preterm newborns proliferate more rapidly than term E
Autor:
Claudine P. Black, Diane L. Gumina, Virginia D. Winn, Vivek Balasubramaniam, Christopher D. Baker
Publikováno v:
Reproductive sciences (Thousand Oaks, Calif.). 24(7)
Preeclampsia (PE) is a pregnancy-specific disease characterized by the new onset of hypertension and proteinuria. Mothers with PE are known to develop endothelial dysfunction, but its effect on infants has been understudied, as newborns are often asy
Autor:
Douglas P. Shepherd, Gregory J. Seedorf, Christopher D. Baker, Jennifer Larmore, Claudine P. Black
Publikováno v:
Biophysical Journal. 110(3)
Disruptions to pro-growth, proliferation, and angiogenesis signaling pathways within endothelial progenitor cells (EPC) may play key role in the pathogenesis of multiple pediatric pulmonary diseases, such as bronchopulmonary dysplasia. Using high-thr
Autor:
Vivek Balasubramaniam, Peter M. Mourani, Marci K. Sontag, Claudine P. Black, Steven H. Abman, Sharon L. Ryan, Christopher D. Baker
Publikováno v:
European Respiratory Journal. 40:1516-1522
Bronchopulmonary dysplasia (BPD), the chronic lung disease of prematurity, is associated with impaired vascular and alveolar growth. Antenatal factors contribute to the risk for developing BPD by unclear mechanisms. Endothelial progenitor cells, such
Autor:
Steven H. Abman, Sharon L. Ryan, Claudine P. Black, Christopher D. Baker, Vivek Balasubramaniam
Publikováno v:
The Journal of Pediatrics. 163:905-907
Endothelial colony-forming cells (ECFCs) are decreased in the cord blood of preterm infants with moderate-to-severe bronchopulmonary dysplasia. We quantified ECFCs from infants with congenital diaphragmatic hernia, a neonatal disorder with severe lun
Autor:
Peter M. Mourani, Christopher D. Baker, Sharon L. Ryan, Steven H. Abman, Vivek Balasubramaniam, Claudine P. Black
Publikováno v:
B108. NEONATAL LUNG DISEASE AND BRONCHOPULMONARY DYSPLASIA.
Autor:
Kenneth H. Cowan, Rebecca S. Spieker, Phu T. Cao, Ying Yan, Claudine P. Black, Ryan Kolb, Alexandra M. Ristow, Jamie L. Haferbier
Publikováno v:
Cancer research. 68(13)
Previous studies from our laboratory have shown that the activation of G2-M checkpoint after exposure of MCF-7 breast cancer cells to γ-irradiation (IR) is dependent on the activation of extracellular signal-regulated kinase 1/2 (ERK1/2) signaling.