Zobrazeno 1 - 10 of 11 pro vyhledávání: '"Claudia Rebosio"'
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Akademický článek
Exocytosis regulates trafficking of GABA and glycine heterotransporters in spinal cord glutamatergic synapses: a mechanism for the excessive heterotransporter-induced release of glutamate in experimental amyotrophic lateral sclerosis
Autor: Marco Milanese, Tiziana Bonifacino, Ernesto Fedele, Claudia Rebosio, Luca Cattaneo, Fabio Benfenati, Cesare Usai, Giambattista Bonanno
Publikováno v: Neurobiology of Disease, Vol 74, Iss , Pp 314-324 (2015)
The impact of synaptic vesicle endo-exocytosis on the trafficking of nerve terminal heterotransporters was studied by monitoring membrane expression and function of the GABA transporter-1 (GAT-1) and of type-1/2 glycine (Gly) transporters (GlyT-1/2)
Externí odkaz: https://doaj.org/article/6c11d3ae9df84976b62a7c5fef535f17
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2
Altered glucose catabolism in the presynaptic and perisynaptic compartments of SOD1G93Amouse spinal cord and motor cortex indicates that mitochondria are the site of bioenergetic imbalance in ALS
Autor: Tiziana Bonifacino, Claudia Rebosio, Silvia Ravera, Isabella Panfoli, Francesca Provenzano, Giambattista Bonanno, Cesare Usai, Carola Torazza, Marco Milanese
Publikováno v: Journal of Neurochemistry. 151:336-350
Amyotrophic lateral sclerosis is an adult-onset neurodegenerative disease that develops because of motor neuron death. Several mechanisms occur supporting neurodegeneration, including mitochondrial dysfunction. Recently, we demonstrated that the syna
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5d9f3e848442f9a949b27ae006fc6ce9
https://doi.org/10.1111/jnc.14819
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3
Presynaptic GLP-1 receptors enhance the depolarization-evoked release of glutamate and GABA in the mouse cortex and hippocampus
Autor: Ernesto Fedele, Claudia Rebosio, Roberta Ricciarelli, Matilde Balbi, Mario Passalacqua
Publikováno v: BioFactors. 44:148-157
Glucagon-like peptide-1 receptors (GLP-1Rs) have been shown to mediate cognitive-enhancing and neuroprotective effects in the central nervous system. However, little is known about their physiological roles on central neurotransmission, especially at
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::19042054d8a0a77782596b03abaa2c5d
https://doi.org/10.1002/biof.1406
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4
Amyloid-β Peptide Is Needed for cGMP-Induced Long-Term Potentiation and Memory
Autor: Walter Gulisano, Elisa Calcagno, Ernesto Fedele, Maria Rosaria Tropea, Ottavio Arancio, Claudia Rebosio, Roberta Ricciarelli, Maria Adelaide Pronzato, Utpal Das, Subhojit Roy, Silvia Conti, Daniela Puzzo, Agostino Palmeri, Daniela Rivera
Publikováno v: The Journal of Neuroscience. 37:6926-6937
High levels of amyloid-β peptide (Aβ) have been related to Alzheimer9s disease pathogenesis. However, in the healthy brain, low physiologically relevant concentrations of Aβ are necessary for long-term potentiation (LTP) and memory. Because cGMP p
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c0c4ea990f295e0c80b6371ab386e78b
https://doi.org/10.1523/jneurosci.3607-16.2017
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5
Enhanced Function and Overexpression of Metabotropic Glutamate Receptors 1 and 5 in the Spinal Cord of the SOD1G93A Mouse Model of Amyotrophic Lateral Sclerosis during Disease Progression
Autor: Cesare Usai, Tiziana Bonifacino, Matilde Balbi, Francesca Provenzano, Carola Torazza, Luca Raiteri, Giambattista Bonanno, Claudia Rebosio, Marco Milanese, Ernesto Fedele
Publikováno v: International Journal of Molecular Sciences
International Journal of Molecular Sciences, Vol 20, Iss 18, p 4552 (2019)
Volume 20
Issue 18
Glutamate (Glu)-mediated excitotoxicity is a major cause of amyotrophic lateral sclerosis (ALS) and our previous work highlighted that abnormal Glu release may represent a leading mechanism for excessive synaptic Glu. We demonstrated that group I met
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4648a8dfecccfce359637b8d2ee39dfe
http://europepmc.org/articles/PMC6774337
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6
Altered glucose catabolism in the presynaptic and perisynaptic compartments of SOD1
Autor: Silvia, Ravera, Carola, Torazza, Tiziana, Bonifacino, Francesca, Provenzano, Claudia, Rebosio, Marco, Milanese, Cesare, Usai, Isabella, Panfoli, Giambattista, Bonanno
Publikováno v: Journal of neurochemistryReferences. 151(3)
Amyotrophic lateral sclerosis is an adult-onset neurodegenerative disease that develops because of motor neuron death. Several mechanisms occur supporting neurodegeneration, including mitochondrial dysfunction. Recently, we demonstrated that the syna
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::9d7e6248c8ebf467798d239b23aa37ee
https://pubmed.ncbi.nlm.nih.gov/31282572
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8
Presynaptic GLP-1 receptors enhance the depolarization-evoked release of Glutamate and GABA in the mouse cortex and hippocampus
Autor: Claudia, Rebosio, Matilde, Balbi, Mario, Passalacqua, Roberta, Ricciarelli, Ernesto, Fedele
Glucagon-like peptide-1 receptors (GLP-1Rs) have been shown to mediate cognitive-enhancing and neuroprotective effects in the central nervous system. However, little is known about their physiological roles on central neurotransmission, especially at
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::53e73905bcb7cd0cefcaa20ac93c4c66
http://hdl.handle.net/11567/884305
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9
Memory-enhancing effects of GEBR-32a, a new PDE4D inhibitor holding promise for the treatment of Alzheimer’s disease
Autor: Carla Villa, Jos Prickaerts, Chiara Brullo, Claudia Rebosio, Matilde Balbi, Roberta Ricciarelli, Elisa Calcagno, Britt T. J. van Hagen, Olga Bruno, Ottavio Arancio, Elentina K. Argyrousi, Ernesto Fedele, Hong Zhang, Maria Adelaide Pronzato
Publikováno v: Scientific Reports, 7:46320. Nature Publishing Group
Scientific Reports
Memory loss characterizes several neurodegenerative disorders, including Alzheimer’s disease (AD). Inhibition of type 4 phosphodiesterase (PDE4) and elevation of cyclic adenosine monophosphate (cAMP) has emerged as a promising therapeutic approach
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::820c4038ac860a6fcf67b3255acd9a54
https://doi.org/10.1038/srep46320
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10
Exocytosis regulates trafficking of GABA and glycine heterotransporters in spinal cord glutamatergic synapses: a mechanism for the excessive heterotransporter-induced release of glutamate in experimental amyotrophic lateral sclerosis
Autor: Claudia Rebosio, Tiziana Bonifacino, Luca Cattaneo, Ernesto Fedele, Giambattista Bonanno, Marco Milanese, Cesare Usai, Fabio Benfenati
Publikováno v: Neurobiology of disease 74 (2015): 314–324. doi:10.1016/j.nbd.2014.12.004
info:cnr-pdr/source/autori:Milanese M.; Bonifacino T.; Fedele E.; Rebosio C.; Cattaneo L.; Benfenati F.; Usai C.; Bonanno G./titolo:Exocytosis regulates trafficking of GABA and glycine heterotransporters in spinal cord glutamatergic synapses: A mechanism for the excessive heterotransporter-induced release of glutamate in experimental amyotrophic lateral sclerosis/doi:10.1016%2Fj.nbd.2014.12.004/rivista:Neurobiology of disease/anno:2015/pagina_da:314/pagina_a:324/intervallo_pagine:314–324/volume:74
Neurobiology of Disease, Vol 74, Iss, Pp 314-324 (2015)
The impact of synaptic vesicle endo-exocytosis on the trafficking of nerve terminal heterotransporters was studied by monitoring membrane expression and function of the GABA transporter-1 (GAT-1) and of type-1/2 glycine (Gly) transporters (GlyT-1/2)
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::10e103ba66a03050d8b6137ad19ff63a
https://hdl.handle.net/11567/768224
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