Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Claudia P. Yefi"'
Autor:
Héctor M. Ramos-Zaldívar, Iva Polakovicova, Edison Salas-Huenuleo, Alejandro H. Corvalán, Marcelo J. Kogan, Claudia P. Yefi, Marcelo E. Andia
Publikováno v:
Fluids and Barriers of the CNS, Vol 19, Iss 1, Pp 1-15 (2022)
Abstract Extracellular vesicles (EVs) are particles naturally released from cells that are delimited by a lipid bilayer and are unable to replicate. How the EVs cross the Blood–Brain barrier (BBB) in a bidirectional manner between the bloodstream a
Externí odkaz:
https://doaj.org/article/bb4df49d73b34f6f84d0f95f3812a6a4
Autor:
Viviana A Cavieres, Alexis González, Vanessa C Muñoz, Claudia P Yefi, Hianara A Bustamante, Rafael R Barraza, Cheril Tapia-Rojas, Carola Otth, María José Barrera, Carlos González, Gonzalo A Mardones, Nibaldo C Inestrosa, Patricia V Burgos
Publikováno v:
PLoS ONE, Vol 10, Iss 8, p e0136313 (2015)
Alzheimer's disease (AD) is a neurodegenerative disorder characterized by the accumulation of amyloid-β (Aβ) peptide. We have previously shown that the compound tetrahydrohyperforin (IDN5706) prevents accumulation of Aβ species in an in vivo model
Externí odkaz:
https://doaj.org/article/9775bf90c9e94a3aba8e18681276dc3a
Publikováno v:
Endocrine Research. 35:59-70
Pregnancy may be complicated by maternal diabetes. The following experiments were performed in an attempt to produce mouse models of insulin-resistant maternal diabetes.CD1 females received 200 mg/kg streptozocin (STZ) to model insulin-dependent diab
Autor:
Nibaldo C. Inestrosa, Viviana A Cavieres, Claudia P. Yefi, Alexis E González, Carola Otth, Cheril Tapia-Rojas, Carlos B. Gonzalez, Rafael R. Barraza, Hianara A. Bustamante, María-José Barrera, Gonzalo A. Mardones, Vanessa C. Muñoz, Patricia V. Burgos
Publikováno v:
PLoS ONE
Artículos CONICYT
CONICYT Chile
instacron:CONICYT
PLoS ONE, Vol 10, Iss 8, p e0136313 (2015)
Artículos CONICYT
CONICYT Chile
instacron:CONICYT
PLoS ONE, Vol 10, Iss 8, p e0136313 (2015)
Alzheimer's disease (AD) is a neurodegenerative disorder characterized by the accumulation of amyloid-β (Aβ) peptide. We have previously shown that the compound tetrahydrohyperforin (IDN5706) prevents accumulation of Aβ species in an in vivo model