Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Claudia L. M. Silva"'
Autor:
Ygor Marinho, Elizabeth S. Villarreal, Sammy Y. Aboagye, David L. Williams, Jun Sun, Claudia L. M. Silva, Sarah E. Lutz, Suellen D. Oliveira
Publikováno v:
Frontiers in Immunology, Vol 14 (2023)
Schistosomiasis-associated Pulmonary Arterial Hypertension (Sch-PAH) is a life-threatening complication of chronic S. mansoni infection that can lead to heart failure and death. During PAH, the expansion of apoptosis-resistant endothelial cells (ECs)
Externí odkaz:
https://doaj.org/article/82466b06107d45c1b4e65f8c7c13f462
Autor:
Mariana M Chaves, Debora A Sinflorio, Maria Luiza Thorstenberg, Monique Daiane Andrade Martins, Aline Cristina Abreu Moreira-Souza, Thuany Prado Rangel, Claudia L M Silva, Maria Bellio, Claudio Canetti, Robson Coutinho-Silva
Publikováno v:
PLoS Pathogens, Vol 15, Iss 6, p e1007887 (2019)
Leishmaniasis is a neglected tropical disease affecting millions of individuals worldwide. P2X7 receptor has been linked to the elimination of Leishmania amazonensis. Biological responses evoked by P2X7 receptor activation have been well-documented,
Externí odkaz:
https://doaj.org/article/aa7a275cf206443fb3d0cc99fa606955
Autor:
Suellen Darc Oliveira, Ejehi Erewele, Maricela Castellon, Andrew Schwartz, David L. Williams, Nick Morrell, Marcelo Bonini, Claudia L. M. Silva, Sarah E. Lutz, Serpil Erzurum, Richard D Minshall
Publikováno v:
Arteriosclerosis, Thrombosis, and Vascular Biology. 42
Introduction: Schistosomiasis-associated Pulmonary Arterial Hypertension (Sch-PAH) is a life-threatening complication of chronic S. mansoni infection, which can evolve to heart failure and death. During PAH, hyperproliferation of apoptosis-resistant
Autor:
Suellen D S Oliveira, Luis E M Quintas, Luciana S Amaral, François Noël, Sandra H Farsky, Claudia L M Silva
Publikováno v:
PLoS ONE, Vol 6, Iss 8, p e23547 (2011)
Background and aimsSchistosomiasis is an intravascular parasitic disease associated with inflammation. Endothelial cells control leukocyte transmigration and vascular permeability being modulated by pro-inflammatory mediators. Recent data have shown
Externí odkaz:
https://doaj.org/article/0d2b3802548846bea575904cc9fa62b0