Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Claudia Eggstein"'
Autor:
Alice Bernard, Claudia Eggstein, Linyan Tang, Marius Keller, Andreas Körner, Valbona Mirakaj, Peter Rosenberger
Publikováno v:
Journal of Biomedical Science, Vol 31, Iss 1, Pp 1-15 (2024)
Abstract Background Intracellular sensing of lipopolysaccharide (LPS) is essential for the immune response against gram-negative bacteria and results in activation of caspase-11 and pyroptotic cell death with fatal consequences in sepsis. We found th
Externí odkaz:
https://doaj.org/article/b54e832e9aff4c64af350d560fb3652c
Autor:
Andreas Körner, David Köhler, Mariella Schneider, Judith M. Roth, Tiago F. Granja, Claudia Eggstein, Valbona Mirakaj, Peter Rosenberger
Publikováno v:
Frontiers in Immunology, Vol 14 (2023)
IntroductionThe study explores the role of endothelial Semaphorin 7A (SEMA7A) in inflammatory processes. SEMA7A is known for enhancing inflammation during tissue hypoxia and exhibiting anti-inflammatory properties in the intestinal system during coli
Externí odkaz:
https://doaj.org/article/8f3c4ab1b0174265a23ba3ac949c4888
Autor:
David Köhler, Tiago Granja, Julia Volz, Michael Koeppen, Harald F. Langer, Georg Hansmann, Ekaterina Legchenko, Tobias Geisler, Tamam Bakchoul, Claudia Eggstein, Helene A. Häberle, Bernhard Nieswandt, Peter Rosenberger
Publikováno v:
Nature Communications, Vol 11, Iss 1, Pp 1-12 (2020)
Reperfusion injury following myocardial ischemia is aggravated by inflammation and platelet–neutrophil complex formation. Here the authors show that semaphorin 7A binds to platelet GPIb, enhancing platelet–neutrophil interaction and increasing po
Externí odkaz:
https://doaj.org/article/2aada106ea4f4ec3b1d24ebbcb4e19ee
Autor:
Tiago F. Granja, David Köhler, Veronika Leiss, Claudia Eggstein, Bernd Nürnberg, Peter Rosenberger, Sandra Beer-Hammer
Publikováno v:
Cells, Vol 11, Iss 19, p 3009 (2022)
Ischemic events are associated with severe inflammation and are here referred to as ischemic inflammatory response (IIR). Recent studies identified the formation of platelet–neutrophil complexes (PNC) as key players in IIR. We investigated the role
Externí odkaz:
https://doaj.org/article/a5326fc780354cf685969ba73c38d277
Autor:
Kristian-Christos Ngamsri, Christoph Jans, Rizki A. Putri, Katharina Schindler, Jutta Gamper-Tsigaras, Claudia Eggstein, David Köhler, Franziska M. Konrad
Publikováno v:
Frontiers in Immunology, Vol 11 (2020)
Our previous studies revealed a pivotal role of the chemokine stromal cell-derived factor (SDF)-1 and its receptors CXCR4 and CXCR7 on migratory behavior of polymorphonuclear granulocytes (PMNs) in pulmonary inflammation. Thereby, the SDF-1-CXCR4/CXC
Externí odkaz:
https://doaj.org/article/17897d4a7c6c493099f754d04a1d2840
Autor:
Beer-Hammer, Tiago F. Granja, David Köhler, Veronika Leiss, Claudia Eggstein, Bernd Nürnberg, Peter Rosenberger, Sandra
Publikováno v:
Cells; Volume 11; Issue 19; Pages: 3009
Ischemic events are associated with severe inflammation and are here referred to as ischemic inflammatory response (IIR). Recent studies identified the formation of platelet–neutrophil complexes (PNC) as key players in IIR. We investigated the role
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=multidiscipl::a0f6f5fc8a5db7687114b979e1c84b42
Autor:
Claudia Eggstein, Helene A. Häberle, Julia Volz, Tobias Geisler, Michael Koeppen, Georg Hansmann, Tamam Bakchoul, Peter Rosenberger, Ekaterina Legchenko, Tiago Granja, Harald F. Langer, David Köhler, Bernhard Nieswandt
Publikováno v:
Nature Communications, Vol 11, Iss 1, Pp 1-12 (2020)
Nature Communications
Nature Communications
Myocardial ischemia is one of the leading health problems worldwide. Therapy consists of the restitution of coronary perfusion which is followed by myocardial inflammation. Platelet–neutrophil interaction is a crucial process during inflammation, y
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8a645f3bcc5a07f2088b93319fba7f48
https://doaj.org/article/2aada106ea4f4ec3b1d24ebbcb4e19ee
https://doaj.org/article/2aada106ea4f4ec3b1d24ebbcb4e19ee