Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Clara Stanko"'
Autor:
Katjana Schwab, Konstantin Riege, Luis Coronel, Clara Stanko, Silke Förste, Steve Hoffmann, Martin Fischer
Publikováno v:
Cell Death Discovery, Vol 10, Iss 1, Pp 1-8 (2024)
Abstract The transcription factor regulatory factor X 7 (RFX7) has been identified as a tumor suppressor that is recurrently mutated in lymphoid cancers and appears to be dysregulated in many other cancers. RFX7 is activated by the well-known tumor s
Externí odkaz:
https://doaj.org/article/e534e98200654b9791d782f12a46e429
Autor:
Sijia Ren, Feng Bai, Viviane Schnell, Clara Stanko, Muriel Ritsch, Tino Schenk, Emanuel Barth, Manja Marz, Bin Wang, Xin-Hai Pei, Holger Bierhoff
Publikováno v:
Cell Reports, Vol 43, Iss 1, Pp 113644- (2024)
Summary: Extensive remodeling of the female mammary epithelium during development and pregnancy has been linked to cancer susceptibility. The faithful response of mammary epithelial cells (MECs) to hormone signaling is key to avoiding breast cancer d
Externí odkaz:
https://doaj.org/article/1c72a64a8d864b5791a60d74a4984cb5
Publikováno v:
Scientific Reports, Vol 7, Iss 1, Pp 1-11 (2017)
Abstract High risk human papilloma viruses cause several types of cancer. The HPV oncoproteins E6 and E7 are essential for oncogenic cell transformation. E6 mediates the degradation of the tumor suppressor p53, and E7 can form complexes with the reti
Externí odkaz:
https://doaj.org/article/8835ae107aaf44df83dbe8f3c6cd940b
Autor:
Sven Stengel, Kevin R. Petrie, Yordan Sbirkov, Clara Stanko, Faezeh Ghazvini Zadegan, Veronica Gil, Rafał Skopek, Paweł Kamiński, Łukasz Szymański, Annamaria Brioli, Arthur Zelent, Tino Schenk
Publikováno v:
British Journal of Haematology. 198:338-348
Autor:
Sven, Stengel, Kevin R, Petrie, Yordan, Sbirkov, Clara, Stanko, Faezeh, Ghazvini Zadegan, Veronica, Gil, Rafał, Skopek, Paweł, Kamiński, Łukasz, Szymański, Annamaria, Brioli, Arthur, Zelent, Tino, Schenk
Publikováno v:
British journal of haematologyREFERENCES. 198(2)
Aberrant activity of the phosphatidylinositol-3 kinase/protein kinase B/mammalian target of rapamycin (PI3K/AKT/mTOR [PAM]) pathway, as well as suppressed retinoic acid signalling, contribute to enhanced proliferation and the differentiation blockade