Zobrazeno 1 - 10
of 157
pro vyhledávání: '"Claes, Hellerström"'
Autor:
Bo Hellman, Claes Hellerström
Publikováno v:
Acta Pathologica Microbiologica Scandinavica. 45:113-122
Publikováno v:
Acta Pathologica Microbiologica Scandinavica. 76:184-192
Autor:
Bo Hellman, Claes Hellerström
Publikováno v:
Acta Pathologica Microbiologica Scandinavica. 55:385-394
Publikováno v:
Acta Pathologica Microbiologica Scandinavica. 54:1-8
Publikováno v:
Acta Pathologica Microbiologica Scandinavica Section A Pathology. :285-288
Ligation of the pancreatic duct causes atrophy of the acinar cells but leaves the endocrine tissue intact. In the present study, a partial ligation of the pancreatic duct was performed in the rat, and the in vitro insulin response to glucose was comp
Publikováno v:
Acta Pathologica Microbiologica Scandinavica. 54:365-372
Autor:
Leif Jansson, Anders Hallberg, Steven J. Sandler, Claes Hellerström, Agneta Andersson, Bo Nilsson, Birger Petersson, Ingemar Swenne, L. Reibring
Publikováno v:
Acta Medica Scandinavica. 208:43-48
Transplantation of the endocrine pancreas : a new approach towards treatment of diabetes mellitus.
Publikováno v:
European Journal of Endocrinology. :169-178
The aim of the study was to investigate if hypertension affects pancreatic islet blood flow and endocrine function. For this purpose, spontaneously hypertensive rats (SHR) were compared with normotensive control Wistar-Kyoto rats (WKY). Both islet si
Autor:
Per Westermark, Donald F. Steiner, Niles Fox, Gunilla T. Westermark, Arne Andersson, Claes Hellerström, Decio L. Eizirik
Publikováno v:
Metabolism. 48:448-454
Islet amyloid polypeptide (IAPP)-derived amyloid is frequently deposited in the islets of Langerhans in patients with chronic non-insulin-dependent diabetes mellitus (NIDDM). When human islets were implanted under the renal capsule in nude mice, amyl
Publikováno v:
The Journal of Clinical Endocrinology & Metabolism. 82:2660-2663
We have previously observed that human islets, transplanted under the kidney capsule of hyperglycemic nude mice, show a longlasting impairment in glucose-induced insulin release. To investigate the cause(s) of this phenomenon, we transplanted human i