Zobrazeno 1 - 10
of 22
pro vyhledávání: '"Cinzia Cancellieri"'
Autor:
Alessandro Vacchini, Elisa Maffioli, Dario Di Silvestre, Cinzia Cancellieri, Samantha Milanesi, Simona Nonnis, Sabrina Badanai, Pierluigi Mauri, Armando Negri, Massimo Locati, Gabriella Tedeschi, Elena Monica Borroni
Publikováno v:
Frontiers in Molecular Biosciences, Vol 9 (2022)
ACKR2 is an atypical chemokine receptor which is structurally uncoupled from G proteins and is unable to activate signaling pathways used by conventional chemokine receptors to promote cell migration. Nonetheless, ACKR2 regulates inflammatory and imm
Externí odkaz:
https://doaj.org/article/bde15963e014423c9585dbe4db42066b
Autor:
Federica Banfi, Alicia Rubio, Mattia Zaghi, Luca Massimino, Giulia Fagnocchi, Edoardo Bellini, Mirko Luoni, Cinzia Cancellieri, Anna Bagliani, Chiara Di Resta, Camilla Maffezzini, Angelo Ianielli, Maurizio Ferrari, Rocco Piazza, Luca Mologni, Vania Broccoli, Alessandro Sessa
Publikováno v:
Nature Communications, Vol 12, Iss 1, Pp 1-21 (2021)
Schinzel-Giedion syndrome (SGS) is a fatal developmental syndrome characterized by severe intellectual and physical deficits due, at least in part, to early neurodegeneration. Here the authors introduce a human SGS model that displays disease-relevan
Externí odkaz:
https://doaj.org/article/9f82d3e4675e4ed291494e6ac2e0dfe7
Autor:
Anna Cozzi, Daniel I. Orellana, Paolo Santambrogio, Alicia Rubio, Cinzia Cancellieri, Serena Giannelli, Maddalena Ripamonti, Stefano Taverna, Giulia Di Lullo, Ermanna Rovida, Maurizio Ferrari, Gian Luca Forni, Chiara Fiorillo, Vania Broccoli, Sonia Levi
Publikováno v:
Stem Cell Reports, Vol 13, Iss 5, Pp 832-846 (2019)
Summary: Neuroferritinopathy (NF) is a movement disorder caused by alterations in the L-ferritin gene that generate cytosolic free iron. NF is a unique pathophysiological model for determining the direct consequences of cell iron dysregulation. We es
Externí odkaz:
https://doaj.org/article/1a80b9ac5a774715806543d0e61b3993
Autor:
Angelo Iannielli, Simone Bido, Lucrezia Folladori, Alice Segnali, Cinzia Cancellieri, Alessandra Maresca, Luca Massimino, Alicia Rubio, Giuseppe Morabito, Leonardo Caporali, Francesca Tagliavini, Olimpia Musumeci, Giuliana Gregato, Erwan Bezard, Valerio Carelli, Valeria Tiranti, Vania Broccoli
Publikováno v:
Cell Reports, Vol 22, Iss 8, Pp 2066-2079 (2018)
Summary: Dysfunctions in mitochondrial dynamics and metabolism are common pathological processes associated with Parkinson’s disease (PD). It was recently shown that an inherited form of PD and dementia is caused by mutations in the OPA1 gene, whic
Externí odkaz:
https://doaj.org/article/e74ec4bf3763430d970714f46ab17f1c
Autor:
Pietro Giuseppe Mazzara, Luca Massimino, Marta Pellegatta, Giulia Ronchi, Alessandra Ricca, Angelo Iannielli, Serena Gea Giannelli, Marco Cursi, Cinzia Cancellieri, Alessandro Sessa, Ubaldo Del Carro, Angelo Quattrini, Stefano Geuna, Angela Gritti, Carla Taveggia, Vania Broccoli
Publikováno v:
Nature Communications, Vol 8, Iss 1, Pp 1-13 (2017)
Schwann cells (SCs) myelinate peripheral nerve axons and offer opportunities for the treatment of injuries and demyelinating diseases but reliable and renewable sources of these cells are hard to come by. Here the authors reprogram rat, mouse and hum
Externí odkaz:
https://doaj.org/article/3e5822abc9d6496093cbde7fb6236024
Autor:
Alessandro Vacchini, Cinzia Cancellieri, Samantha Milanesi, Sabrina Badanai, Benedetta Savino, Francesco Bifari, Massimo Locati, Raffaella Bonecchi, Elena Monica Borroni
Publikováno v:
Vaccines, Vol 8, Iss 3, p 542 (2020)
The atypical chemokine receptor ACKR2, formerly named D6, is a scavenger chemokine receptor with a non-redundant role in the control of inflammation and immunity. The scavenging activity of ACKR2 depends on its trafficking properties, which require a
Externí odkaz:
https://doaj.org/article/d13f68dbc089452082d753d6d63f170e
Autor:
Angelo Ianielli, Mattia Zaghi, Chiara Di Resta, Federica Banfi, Edoardo Bellini, Mirko Luoni, Luca Massimino, Rocco Piazza, Alicia Rubio, Alessandro Sessa, Anna Bagliani, Vania Broccoli, Giulia Fagnocchi, Cinzia Cancellieri, Maurizio Ferrari, Luca Mologni, Camilla Maffezzini
Publikováno v:
Nature Communications, Vol 12, Iss 1, Pp 1-21 (2021)
Nature Communications
Nature Communications
The investigation of genetic forms of juvenile neurodegeneration could shed light on the causative mechanisms of neuronal loss. Schinzel-Giedion syndrome (SGS) is a fatal developmental syndrome caused by mutations in the SETBP1 gene, inducing the acc
Autor:
Alicia Rubio, Serena Giannelli, Vania Broccoli, Maurizio Ferrari, Chiara Fiorillo, Daniel Orellana, Ermanna Rovida, Giulia Di Lullo, Paolo Santambrogio, Anna Cozzi, Cinzia Cancellieri, Stefano Taverna, Gian Luca Forni, Maddalena Ripamonti, Sonia Levi
Publikováno v:
Stem Cell Reports, Vol 13, Iss 5, Pp 832-846 (2019)
Stem Cell Reports
Stem Cell Reports
Summary Neuroferritinopathy (NF) is a movement disorder caused by alterations in the L-ferritin gene that generate cytosolic free iron. NF is a unique pathophysiological model for determining the direct consequences of cell iron dysregulation. We est
Autor:
Samantha Milanesi, Massimo Locati, Raffaella Bonecchi, Alessandro Vacchini, Sabrina Badanai, Cinzia Cancellieri, Francesco Bifari, Elena Monica Borroni, Benedetta Savino
Publikováno v:
Vaccines, Vol 8, Iss 542, p 542 (2020)
Vaccines
Volume 8
Issue 3
Vaccines
Volume 8
Issue 3
The atypical chemokine receptor ACKR2, formerly named D6, is a scavenger chemokine receptor with a non-redundant role in the control of inflammation and immunity. The scavenging activity of ACKR2 depends on its trafficking properties, which require a
Autor:
Simone Bido, Alice Segnali, Valeria Tiranti, Vania Broccoli, Francesca Tagliavini, Olimpia Musumeci, Lucrezia Folladori, Cinzia Cancellieri, Giuliana Gregato, Erwan Bezard, Leonardo Caporali, Alicia Rubio, Valerio Carelli, Angelo Iannielli, Luca Massimino, Giuseppe Morabito, Alessandra Maresca
Publikováno v:
Cell Reports
Cell Reports, Vol 22, Iss 8, Pp 2066-2079 (2018)
Cell Reports, Vol 22, Iss 8, Pp 2066-2079 (2018)
Summary Dysfunctions in mitochondrial dynamics and metabolism are common pathological processes associated with Parkinson’s disease (PD). It was recently shown that an inherited form of PD and dementia is caused by mutations in the OPA1 gene, which