Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Chuan-lai Du"'
Autor:
Jun Li, Cheng-gui Miao, Chuan-lai Du, Xiao-lin Zhang, Tong-wen Song, You-yi Xiong, Wei-jing Shi, Mei-song Qin, Bing Zhang, Hao Yu
Publikováno v:
Immunology Letters. 166:45-54
Rheumatoid arthritis (RA) is a symmetrical polyarticular autoimmune disease of unknown etiology. In this present study, we observed that the adenomatous polyposis coli (APC) expression is down-regulated and the expression of microRNA (miR)-663 increa
Autor:
Chuan-lai Du, Xiao-lin Zhang, Cheng-gui Miao, Mei-song Qin, Tong-wen Song, You-yi Xiong, Hao Yu
Publikováno v:
International immunopharmacology. 28(1)
Systemic sclerosis (SSc) is an autoimmune disease characterized by immune disorders, vascular obliteration, excessive extracellular matrix deposition, skin fibrosis, and further pathological change of internal organs. To date, the exact etiology of t
Autor:
You-yi Xiong, Jun Li, Chuan-lai Du, Hua Ye, Xiao-lin Zhang, Cheng-gui Miao, Cheng Huang, Ma Shitang, Tong-wen Song, Wei-jing Shi, Yan Huang, Dou Jinfeng, Jian-zhong Chen, Fang Yanxi, Zhou Guoliang, Liu Hanzhen, Bing Zhang, Mei-song Qin, Xu He, Hao Yu, Dan Qin
Publikováno v:
International immunopharmacology. 28(1)
In previous study, we identified that microRNA (miR)-152 expression was down-regulated in RA model rats, and overexpression of miR-152 inhibited the canonical Wnt signaling through the DNA methyltransferase (DNMT1) inhibition. However, the exact mole
miR-375 regulates the canonical Wnt pathway through FZD8 silencing in arthritis synovial fibroblasts
Autor:
Tong-wen Song, Chuan-lai Du, Xiao-lin Zhang, Hao Yu, Cheng-gui Miao, Jun Li, You-yi Xiong, Wei-jing Shi, Mei-song Qin
Publikováno v:
Immunology letters. 164(1)
Whether the rheumatoid arthritis (RA) pathogenesis is regulated by microRNA (miRNA) is not entirely clear. In this study, we found that miR-375 was down-regulated significantly in fibroblast-like synoviocytes (FLS) in adjuvant-induced arthritis (AIA)
Publikováno v:
Biochimie. 106
Rheumatoid arthritis (RA) is an autoimmune and progressive systemic disease of unknown etiology. Research shows that fibroblast-like synoviocytes (FLS) participate in the cartilage erosion, synovial hyperplasia, inflammatory cytokine secretion and su