Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Chuan-Yi Zheng"'
Publikováno v:
Oncology Letters
Glioma is the most common type of primary tumor of the central nervous system. The present study aimed to demonstrate the role of miR-101 and cyclooxygenase-2 (COX-2) in the initiation and development of glioma. The expression of miR-101 and COX-2 in
Publikováno v:
Oncology Letters
It has been reported previously that the expression of glucose transporter member 3 (GLUT3) is increased in malignant glioma cells compared with normal glial cells. However, the regulating mechanism that causes this phenomenon remains unknown. The pr
Autor:
Quan-Hong Ma, Zhi-Qiang Wu, Jing Wang, Zhong-Min Wen, Chuan-Yi Zheng, Ruxiang Xu, He-Qing Zhao, Ke Yan, Li-Hua Chen
Publikováno v:
Neuroscience Letters. 569:131-136
TAR DNA-binding protein 43 (TDP-43), an essential pathological protein in both amyotrophic later sclerosis (ALS) and frontotemporal lobar degeneration (FTLD), is expressed abnormally in Alzheimer's disease (AD). However, whether and how TDP-43 contri
Amniotic Mesenchymal Stem Cells Decrease Aβ Deposition and Improve Memory in APP/PS1 Transgenic Mice
Autor:
Qing-Shan Deng, Ruxiang Xu, Mou Gao, Ke-En Chen, Qiang Fu, Jun Lv, Shanshan Wang, Zhong-jie Yan, Xiang-Yu Wang, Xing-Yu Dong, Mao-Ying Zhang, Yu You, Chuan-Yi Zheng, Qian-Quan Wan, Hong-Long Zhou, Xiao-Yu Zheng, Hui Yao
Publikováno v:
Neurochemical research. 42(8)
Transplantation of human amniotic mesenchymal stem cells (hAM-MSCs) seems to be a promising strategy for the treatment of neurodegenerative disorders, including Alzheimer's disease (AD). However, the clinical therapeutic effects of hAM-MSCs and their
Autor:
Zheng-Gang Chen, Chuan-Yi Zheng, Wang-Qing Cai, Da-Wei Li, Fu-Yue Ye, Jian Zhou, Ran Wu, Kun Yang
Publikováno v:
Oncology Research; 2019, Vol. 27 Issue 2, p147-155, 9p
Autor:
Ming-Ming Zou, Mao-Ying Zhang, Quan-Hong Ma, Ruxiang Xu, Jian-Wei Zhu, Shao Li, Jing Wang, Zhi-Cheng Xiao, Yan Zhang, Chuan-Yi Zheng, Chun-Feng Liu, Qi-Fa Li
Publikováno v:
Neurobiology of aging. 35(12)
Hyperactivity and its compensatory mechanisms may causally contribute to synaptic and cognitive deficits in Alzheimer's disease (AD). Blocking the overexcitation of the neural network, with levetiracetam (LEV), a sodium channel blocker applied in the