Zobrazeno 1 - 10
of 19
pro vyhledávání: '"Chu-Wei Huang"'
Autor:
Hsin-Hsiung Chen, Hao-Yu Lu, Chao-Hsin Chang, Shih-Hao Lin, Chu-Wei Huang, Po-Han Wei, Yi-Wen Chen, Yi-Rou Lin, Hsien-Sung Huang, Pei-Yu Wang, Yeou-Ping Tsao, Show-Li Chen
Publikováno v:
Stem Cell Research & Therapy, Vol 13, Iss 1, Pp 1-20 (2022)
Abstract Background Breast carcinoma-amplified sequence 2 (BCAS2) regulates β-catenin gene splicing. The conditional knockout of BCAS2 expression in the forebrain (BCAS2 cKO) of mice confers impaired learning and memory along with decreased β-caten
Externí odkaz:
https://doaj.org/article/ff43f430a06d49728dfe6ec84acac4b6
Autor:
Meng-Hsuan Chou, Yi-Chen Hsieh, Chu-Wei Huang, Po-Han Chen, Shih-Peng Chan, Yeou-Ping Tsao, Hsiu-Hsiang Lee, June-Tai Wu, Show-Li Chen
Publikováno v:
PLoS ONE, Vol 10, Iss 6, p e0130706 (2015)
Previously, we showed that BCAS2 is essential for Drosophila viability and functions in pre-mRNA splicing. In this study, we provide strong evidence that BCAS2 regulates the activity of Delta-Notch signaling via Delta pre-mRNA splicing. Depletion of
Externí odkaz:
https://doaj.org/article/7fcb61702acb458784f82e1c81a45f07
Autor:
Chu-Wei Huang, 黃楚崴
105
Part I: Breast carcinoma amplified sequence 2 (BCAS2) is a 26 kDa nuclear protein.Recent studies demonstrated that there were at least two BCAS2 mechanisms involved in carcinogenesis: (1) BCAS2 is a negative regulator of p53: Deprivation of
Part I: Breast carcinoma amplified sequence 2 (BCAS2) is a 26 kDa nuclear protein.Recent studies demonstrated that there were at least two BCAS2 mechanisms involved in carcinogenesis: (1) BCAS2 is a negative regulator of p53: Deprivation of
Externí odkaz:
http://ndltd.ncl.edu.tw/handle/wu4qx2
Autor:
Hsin-Hsiung Chen, Hao-Yu Lu, Chao-Hsin Chang, Shih-Hao Lin, Chu-Wei Huang, Po-Han Wei, Yi-Wen Chen, Yi-Rou Lin, Hsien-Sung Huang, Pei-Yu Wang, Yeou-Ping Tsao, Show-Li Chen
Publikováno v:
Stem cell researchtherapy. 13(1)
Background Breast carcinoma-amplified sequence 2 (BCAS2) regulates β-catenin gene splicing. The conditional knockout of BCAS2 expression in the forebrain (BCAS2 cKO) of mice confers impaired learning and memory along with decreased β-catenin expres
Autor:
Victoria Arevalo, Tamille Rhynes, Keiko Yabuno-Nakagawa, Oleh Krupa, Suriya Thompson, Naoki Nakagawa, Jingjun Li, Chu-Wei Huang, Zoltán Molnár, Jason L. Stein, Janice Lee, Amelia Lee, Aditi Adhikari, Charlotte Plestant, Ali Badache, Eva S. Anton
Publikováno v:
Neuron
Neuron, Elsevier, 2019, 103 (5), pp.836-852.e5. ⟨10.1016/j.neuron.2019.05.049⟩
Neuron, 2019, 103 (5), pp.836-852.e5. ⟨10.1016/j.neuron.2019.05.049⟩
Neuron, Elsevier, 2019, 103 (5), pp.836-852.e5. ⟨10.1016/j.neuron.2019.05.049⟩
Neuron, 2019, 103 (5), pp.836-852.e5. ⟨10.1016/j.neuron.2019.05.049⟩
International audience; Polarized, non-overlapping, regularly spaced, tiled organization of radial glial cells (RGCs) serves as a framework to generate and organize cortical neuronal columns, layers, and circuitry. Here, we show that mediator of cell
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::234b12b6c69d3c35d116f686322514f0
https://hal.archives-ouvertes.fr/hal-02530383
https://hal.archives-ouvertes.fr/hal-02530383
Autor:
Lee Ci, Chu-Wei Huang, Chung Yp, Chang Hw, Ming-Shyue Lee, Show-Li Chen, Ping-Chang Kuo, Chiun-Sheng Huang, Hsieh Sw, Tsao Lp, Yeou-Ping Tsao
Publikováno v:
British Journal of Cancer
Background: We showed previously that breast carcinoma amplified sequence 2 (BCAS2) functions as a negative regulator of p53. We also found that BCAS2 is a potential AR-associated protein. AR is essential for the growth and survival of prostate carci
Autor:
Chun Liang Lo, Chorng Shyan Chern, Hsin-Cheng Chiu, Shih Hong Chen, Wen Chia Huang, Wen-Hsuan Chiang, Chu Wei Huang
Publikováno v:
Biomacromolecules. 17(12)
A novel drug delivery strategy featured with enhanced uptake of nanoparticles (NPs) by targeted tumor cells and subsequent intratumoral cellular hitchhiking of chemotherapy to deep tumor regions was described. The NP delivery system was obtained from
Autor:
June-Tai Wu, Li-Jen Lee, Po-Han Chen, Pei-Yu Wang, Chu-Wei Huang, Show-Li Chen, Meng-Hsuan Chou, Yi Wen Chen, Yi-Rou Lin, Yeou-Ping Tsao
Publikováno v:
Scientific Reports
Breast carcinoma amplified sequence 2 (BCAS2) is a core component of the hPrP19 complex that controls RNA splicing. Here, we performed an exon array assay and showed that β-catenin is a target of BCAS2 splicing regulation. The regulation of dendrite