Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Christopher W. Benner"'
Autor:
Nicholas K. Vinckier, Nisha A. Patel, Ryan J. Geusz, Allen Wang, Jinzhao Wang, Ileana Matta, Austin R. Harrington, Matthew Wortham, Nichole Wetton, Jianxun Wang, Ulupi S. Jhala, Michael G. Rosenfeld, Christopher W. Benner, Hung-Ping Shih, Maike Sander
Publikováno v:
Nature Communications, Vol 11, Iss 1, Pp 1-15 (2020)
How epigenetic regulation affects pancreatic development is unclear. Here, the authors show that the histone demethylase LSD1 regulates the epigenetic state of developmental enhancers during pancreatic specification and controls how these enhancers r
Externí odkaz:
https://doaj.org/article/219e2bb5225f43dcb77d228c096f4de8
Autor:
Xiaojing Yang, André A. Weber, Elvira Mennillo, Miles Paszek, Samantha Wong, Sabrina Le, Jia Ying Ashley Teo, Max Chang, Christopher W. Benner, Robert H. Tukey, Shujuan Chen
Publikováno v:
Journal of Biological Chemistry. 299:102955
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::4650db3da71e8d70123f15bd9c78368d
https://doi.org/10.1016/j.jbc.2023.102955
https://doi.org/10.1016/j.jbc.2023.102955
Autor:
Michael Tun Yin Lam, Sascha H. Duttke, Mazen F. Odish, Hiep D. Le, Emily A. Hansen, Celina T. Nguyen, Samantha Trescott, Roy Kim, Shaunak Deota, Max W. Chang, Arjun Patel, Mark Hepokoski, Mona Alotaibi, Mark Rolfsen, Katherine Perofsky, Anna S. Warden, Jennifer Foley, Sydney I. Ramirez, Jennifer M. Dan, Robert K. Abbott, Shane Crotty, Laura E. Crotty Alexander, Atul Malhotra, Satchidananda Panda, Christopher W. Benner, Nicole G. Coufal
Publikováno v:
Cell Reports Medicine. 4:100935
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::b1b1f70a482dbec00bff4d741b909db2
https://doi.org/10.1016/j.xcrm.2023.100935
https://doi.org/10.1016/j.xcrm.2023.100935
Autor:
Wolfgang H Dillmann, Majid Ghassemian, Christopher W Benner, Jorge A Suarez, Tanja Diemer, Anzhi Dai, Sven Heinz, Darren E. Casteel, Jorge Suarez, Brian T Scott, Federico Cividini, Ada Admin
The contribution of altered mitochondrial Ca2+ handling to metabolic and functional defects in type 2 diabetic (T2D) mouse hearts is not well understood. Here, we show that the T2D heart is metabolically inflexible and almost exclusively dependent on
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1df2a6cba74393b03d680a7ecf6cd09c
https://doi.org/10.2337/figshare.13342229
https://doi.org/10.2337/figshare.13342229
