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of 10
pro vyhledávání: '"Christine Kratzel"'
Autor:
Achim Thomzig, Walter Schulz-Schaeffer, Arne Wrede, Wilhelm Wemheuer, Bertram Brenig, Christine Kratzel, Karin Lemmer, Michael Beekes
Publikováno v:
PLoS Pathogens, Vol 3, Iss 5, p e66 (2007)
Prion infectivity and its molecular marker, the pathological prion protein PrP(Sc), accumulate in the central nervous system and often also in lymphoid tissue of animals or humans affected by transmissible spongiform encephalopathies. Recently, PrP(S
Externí odkaz:
https://doaj.org/article/095245e813094dbe87522181b8ad4164
Autor:
Marion Joncic, Muhsin Oezel, Michael Beekes, Georg Pauli, Christine Kratzel, Karin Lemmer, Martin Mielke
Publikováno v:
Journal of General Virology. 89:348-358
The unusual resistance of agents causing transmissible spongiform encephalopathies (TSEs) to chemical or thermal inactivation requires special decontamination procedures in order to prevent accidental transmission of these pathogens by surgical instr
Publikováno v:
Journal of General Virology. 88:3479-3485
Prions, the putative causative agents of transmissible spongiform encephalopathies, are neurotropic pathogens that spread to the central nervous system via synaptically linked neural conduits upon peripheral infection. Axons and their transport proce
Publikováno v:
Journal of Neuro-Oncology. 72:209-215
Implanted drug carrier systems for retarded chemotherapy against gliomas are mainly based upon polymers containing nitrosoureas. The authors have developed an intracavitary carrier system of biodegradable liquid crystalline cubic phases encapsulating
Publikováno v:
Journal of Clinical Investigation. 113:1465-1472
Recently, pathological prion protein PrP(Sc), the putative key constituent of infectious agents causing transmissible spongiform encephalopathies (TSEs), was found in muscles of rodents experimentally infected with scrapie and in patients with Creutz
Publikováno v:
Veterinary Research
Veterinary Research, BioMed Central, 2007, 38 (1), pp.127-139. ⟨10.1051/vetres:2006047⟩
Veterinary Research, BioMed Central, 2007, 38 (1), pp.127-139. ⟨10.1051/vetres:2006047⟩
International audience; As is known from various animal models, the spread of agents causing transmissible spongiform encephalopathies (TSE) after peripheral infection affects peripheral nerves before reaching the central nervous system (CNS) and lea
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c97c2b0cbc8b1951190ed7e426d6b6ff
https://hal.archives-ouvertes.fr/hal-00902843/document
https://hal.archives-ouvertes.fr/hal-00902843/document
Scrapie, bovine spongiform encephalopathy and chronic wasting disease are orally communicable, transmissible spongiform encephalopathies (TSEs). As zoonotic transmissions of TSE agents may pose a risk to human health, the identification of reservoirs
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fd40dd7478f22e091b1730f174a661c1
https://europepmc.org/articles/PMC1319182/
https://europepmc.org/articles/PMC1319182/
Publikováno v:
BMC Veterinary Research
BMC Veterinary Research, Vol 3, Iss 1, p 22 (2007)
BMC Veterinary Research, Vol 3, Iss 1, p 22 (2007)
Background The exact role of the lymphoreticular system in the spread of peripheral prion infections to the central nervous system still needs further elucidation. Against this background, the influence of the regional lymph node (Ln. popliteus) on t
Autor:
Wilhelm Wemheuer, Michael Beekes, Christine Kratzel, Bertram Brenig, Walter J. Schulz-Schaeffer, Karin Lemmer, Arne Wrede, Achim Thomzig
Publikováno v:
PLoS Pathogens, Vol 3, Iss 5, p e66 (2007)
PLoS Pathogens
PLoS Pathogens
Prion infectivity and its molecular marker, the pathological prion protein PrPSc, accumulate in the central nervous system and often also in lymphoid tissue of animals or humans affected by transmissible spongiform encephalopathies. Recently, PrPSc w
Autor:
Gerhard Schmidmaier, Camilla Bergmann, Britt Wildemann, Georg N. Duda, Stefan Greiner, Christine Kratzel
Publikováno v:
BMC Musculoskeletal Disorders
BMC Musculoskeletal Disorders, Vol 9, Iss 1, p 135 (2008)
BMC Musculoskeletal Disorders, Vol 9, Iss 1, p 135 (2008)
Background Delayed union or nonunion are frequent and feared complications in fracture treatment. Animal models of impaired bone healing are rare. Moreover, specific descriptions are limited although understanding of the biological course of pathogen