Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Christine A. Kostek"'
Autor:
Jeffrey Boone Miller, Magdalena Ardelt, Mary Lou Beermann, Janice A. Dominov, Amanda J. Kravetz, Christine A. Kostek
Publikováno v:
Human Molecular Genetics. 14:1029-1040
To examine the role of apoptosis in neuromuscular disease progression, we have determined whether pathogenesis in dystrophin-deficient (mdx) and laminin alpha2-deficient (Lama2-null) mice is ameliorated by overexpression of the anti-apoptosis protein
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::62ddd44942489b87d85acefb8e9e247b
https://doi.org/10.1093/hmg/ddi095
https://doi.org/10.1093/hmg/ddi095
Autor:
Christine A. Kostek, Linda C. Burkly, Jeffrey A. Winkles, Pascal Schneider, Shawn Weng, Sharron A.N. Brown, Norm Allaire, Yen-Ming Hsu, Mahasweta Girgenrath, Richard A. Flavell, Timothy S. Zheng, Monica Wang, Martin L. Scott, Beth Browning, Hideo Yagita, Jennifer S. Michaelson, Jeffrey Boone Miller
Publikováno v:
EMBO Journal, vol. 25, no. 24, pp. 5826-5839
Inflammation participates in tissue repair through multiple mechanisms including directly regulating the cell fate of resident progenitor cells critical for successful regeneration. Upon surveying target cell types of the TNF ligand TWEAK, we observe
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::708b6dbd28ba8dfca62e27c87e25899b
https://pubmed.ncbi.nlm.nih.gov/17124496
https://pubmed.ncbi.nlm.nih.gov/17124496
The most common form of human congenital muscular dystrophy (CMD) is caused by mutations in the laminin-alpha2 gene. Loss of laminin-alpha2 function in this autosomal recessive type 1A form of CMD results in neuromuscular dysfunction and, often, earl
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::27258dda672490f9147105ba4a3fdaf9
https://europepmc.org/articles/PMC529286/
https://europepmc.org/articles/PMC529286/
We found that up-regulation of major histocompatibility complex (MHC) class I expression accompanies, but is not required for, appearance of spontaneous myopathy in SJL/J mice. In some neuromuscular diseases, MHC class I expression is markedly up-reg
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0effe317cee4172333a08e613aa265a4
https://europepmc.org/articles/PMC1867159/
https://europepmc.org/articles/PMC1867159/
Publikováno v:
BMC Neurology
BMC Neurology, Vol 5, Iss 1, p 7 (2005)
BMC Neurology, Vol 5, Iss 1, p 7 (2005)
Background Multiple types of mononucleate cells reside among the multinucleate myofibers in skeletal muscles and these mononucleate cells function in muscle maintenance and repair. How neuromuscular disease might affect different types of muscle mono
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ea2ec65047ee49c6b4bb84a99f32afc2
http://europepmc.org/articles/PMC1084348
http://europepmc.org/articles/PMC1084348
Autor:
Jonathan Malowitz, Christine A. Kostek, Amanda J. Kravetz, Jonathan A. Nowak, Janice A. Dominov, Mahasweta Girgenrath, Jeffrey Boone Miller
Publikováno v:
BMC Cell Biology. 5:1
Upon serial passaging of mouse skeletal muscle cells, a small number of cells will spontaneously develop the ability to proliferate indefinitely while retaining the ability to differentiate into multinucleate myotubes. Possible gene changes that coul
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::97cf6b931675bea6ecdad2f4d0cf2ce2
https://doi.org/10.1186/1471-2121-5-1
https://doi.org/10.1186/1471-2121-5-1