Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Christina Meinken"'
Autor:
Thomas Pabst, Beatrice U. Mueller, Christina Meinken, Marie-Ange Bozzini, Miguel Teixera Marques, Katja Seipel
Fig. S1 Inactivation of CEBPA, KLF4 and p53 in AML blasts and normal HSCs.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::53e569426eb795d6492823cd61e28cc2
https://doi.org/10.1158/1078-0432.22459964
https://doi.org/10.1158/1078-0432.22459964
Autor:
Thomas Pabst, Beatrice U. Mueller, Christina Meinken, Marie-Ange Bozzini, Miguel Teixera Marques, Katja Seipel
Fig. S2 Sensitivity of AML cells to conventional induction treatment and small molecule p53 activators.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1fb80d8e2677d019d7f7e597a6a6e2b0
https://doi.org/10.1158/1078-0432.22459961.v1
https://doi.org/10.1158/1078-0432.22459961.v1
Autor:
Thomas Pabst, Beatrice U. Mueller, Christina Meinken, Marie-Ange Bozzini, Miguel Teixera Marques, Katja Seipel
Table S2: Assessment of cell cycle stages, apoptosis and differentiation markers in OCI-AML3 cells after 24hrs of treatment (�lls).
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::239e9d3c0419bc151a09f508c79bb6b9
https://doi.org/10.1158/1078-0432.22459952.v1
https://doi.org/10.1158/1078-0432.22459952.v1
Autor:
Thomas Pabst, Beatrice U. Mueller, Christina Meinken, Marie-Ange Bozzini, Miguel Teixera Marques, Katja Seipel
Table S1: Molecular and karyotype analysis of AML patients
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::305ac831ba2e3b5cbfe25bba6cf9454d
https://doi.org/10.1158/1078-0432.22459955.v1
https://doi.org/10.1158/1078-0432.22459955.v1
Autor:
Thomas Pabst, Beatrice U. Mueller, Christina Meinken, Marie-Ange Bozzini, Miguel Teixera Marques, Katja Seipel
Purpose: In acute myeloid leukemia (AML), the transcription factors CEBPA and KLF4 as well as the universal tumor suppressor p53 are frequently deregulated. Here, we investigated the extent of dysregulation, the molecular interactions, and the mechan
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::56f69e058eb044d689fb94a86194be0d
https://doi.org/10.1158/1078-0432.c.6524360
https://doi.org/10.1158/1078-0432.c.6524360
Autor:
Thomas Pabst, Beatrice U. Mueller, Christina Meinken, Marie-Ange Bozzini, Miguel Teixera Marques, Katja Seipel
Supplementary Material Figure legend to Figure S1 and Figure S2
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::77e4e7f6b11a325921801179e537bb61
https://doi.org/10.1158/1078-0432.22459958.v1
https://doi.org/10.1158/1078-0432.22459958.v1
Autor:
Marie-Ange Bozzini, Katja Seipel, Miguel A.T. Marques, Thomas Pabst, Christina Meinken, Beatrice U. Mueller
Publikováno v:
Clinical cancer research : an official journal of the American Association for Cancer Research. 22(3)
Purpose: In acute myeloid leukemia (AML), the transcription factors CEBPA and KLF4 as well as the universal tumor suppressor p53 are frequently deregulated. Here, we investigated the extent of dysregulation, the molecular interactions, and the mechan