Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Christian Willenbockel"'
Publikováno v:
Deutsche Zeitschrift für Akupunktur. 50:14-18
Zusammenfassung Die Yamamoto neue Schadelakupunktur (YNSA) ist, wie anhand von vier Fallbeispielen gezeigt wird, eine gute Therapieoption bei chronischer Innenohrschwerhorigkeit und chronischem Tinnitus, wenn ein gleichzeitig bestehendes Halswirbelsa
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::d1537282f6de5173de703be859c7027b
https://doi.org/10.1078/0415-6412-00238
https://doi.org/10.1078/0415-6412-00238
Autor:
Christian Willenbockel, Helmut Drexler, Franz Hofmann, Suzanne M. Lohmann, Kai C. Wollert, Robert Feil, Beate Fiedler, Albert Smolenski
Publikováno v:
Journal of Biological Chemistry. 281:32831-32840
Cardiac myocyte apoptosis during ischemia and reperfusion (I/R) is tightly controlled by a complex network of stress-responsive signaling pathways. One pro-apoptotic pathway involves the interaction of the scaffold protein TAB1 with p38 mitogen-activ
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::31ffc1142112ca30e60747406e55a942
https://doi.org/10.1074/jbc.m603416200
https://doi.org/10.1074/jbc.m603416200
Autor:
Klaus-Dieter Schlüter, Torben Brod, Jörg Heineke, Tibor Kempf, Mortimer Korf-Klingebiel, Peter H. Sugden, Helmut Drexler, Kai C. Wollert, Franziska Jantzen, Ralf P. Brandes, Christian Willenbockel, Jeffery D. Molkentin, Volker J. Schmidt
Publikováno v:
Circulation. 123(5)
Background— Fibroblast growth factor 9 (FGF9) is secreted from bone marrow cells, which have been shown to improve systolic function after myocardial infarction (MI) in a clinical trial. FGF9 promotes cardiac vascularization during embryonic develo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::36e0c287b683d840e711d36d1395b6ba
https://pubmed.ncbi.nlm.nih.gov/21262993
https://pubmed.ncbi.nlm.nih.gov/21262993
Autor:
Tibor Kempf, Christian Widera, Udo Bavendiek, Christian Willenbockel, Mortimer Korf-Klingebiel, Anna Kanwischer, Kai C Wollert
Publikováno v:
Circulation. 116
Growth-differentiation factor (GDF)-15 is a stress-responsive TGF-ß related cytokine. GDF-15 plasma levels are increased and provide independent prognostic information in patients with acute coronary syndrome. After coronary ligation in mice, GDF-15
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::3a41534cae0e66ec79eeb534d91538e8
https://doi.org/10.1161/circ.116.suppl_16.ii_165-a
https://doi.org/10.1161/circ.116.suppl_16.ii_165-a
Autor:
Stefan Donath, Rüdiger von Harsdorf, Volkmar Gross, Michael Bader, Nidal Al-Saadi, Rainer Dietz, Ulrich Martin, Christian Willenbockel, Johann Bauersachs, Thomas E. Willnow, Kai C. Wollert, Peifeng Li
Publikováno v:
Circulation. 113(9)
Background— Ischemic heart disease and heart failure are associated with an increased loss of cardiomyocytes due to apoptosis. Whether cardiomyocyte apoptosis plays a causal role in the pathogenesis of heart failure remains enigmatic. The apoptosis
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4f7070f1bb775127f99b1096034498e6
https://pubmed.ncbi.nlm.nih.gov/16505176
https://pubmed.ncbi.nlm.nih.gov/16505176
Autor:
Marian Naguib, Kai C. Wollert, Jörn Tongers, Helmut Drexler, Tibor Kempf, Matthias Eden, Christian Willenbockel, Jeffery D. Molkentin, Daniel Kotlarz, Jörg Heineke, Hans W.M. Niessen, Jian Xu, Jens Strelau
Publikováno v:
Kempf, T, Eden, M, Strelau, J, Naguib, M, Willenbockel, C, Tongers, J, Heineke, J, Kotlarz, D, Xu, J, Molkentin, J D, Niessen, H W, Drexler, H & Wollert, K C 2006, ' The transforming growth factor-beta superfamily member growth-differentiation factor-15 protects the heart from ischemia/reperfusion injury ', Circulation Research, vol. 98, no. 3, pp. 351-60 . https://doi.org/10.1161/01.RES.0000202805.73038.48
Circulation Research, 98(3), 351-60. Lippincott Williams and Wilkins
Circulation Research, 98(3), 351-60. Lippincott Williams and Wilkins
Data from the Women’s Health Study show that serum levels of growth-differentiation factor-15 (GDF-15), a distant member of the transforming growth factor-β superfamily, are an independent risk indicator for adverse cardiovascular events. However,
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::74d6b2680a9b2580d23fc38648781362
https://research.vumc.nl/en/publications/f92160cd-d360-4362-8e94-5a633fb669bf
https://research.vumc.nl/en/publications/f92160cd-d360-4362-8e94-5a633fb669bf
Autor:
Denise Hilfiker-Kleiner, Christian Willenbockel, Tibor Kempf, Sandra C. Gross, Pico Caroni, Marian Naguib, Joerg Heineke, Robert A. Kaiser, Hartmut Ruetten, Helmut Drexler, Theresia Kraft, Arnd Schaefer, Kai C. Wollert, Jeffery D. Molkentin
Adverse left ventricular (LV) remodeling after myocardial infarction (MI) is a major cause for heart failure. Molecular modifiers of the remodeling process remain poorly defined. Patients with heart failure after MI have reduced LV expression levels
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::333aad078087822301a456905ab5cdf2
https://europepmc.org/articles/PMC547821/
https://europepmc.org/articles/PMC547821/