Zobrazeno 1 - 10
of 89
pro vyhledávání: '"Christian S. Haas"'
Publikováno v:
Scientific Reports, Vol 14, Iss 1, Pp 1-7 (2024)
Abstract Cryopyrin-associated periodic syndromes (CAPS) are orphan hereditary auto-inflammatory diseases with various phenotypes, including chronic kidney disease (CKD). Current therapies inhibit interleukin-1 (IL-1) to achieve clinical and serologic
Externí odkaz:
https://doaj.org/article/347a47ff0dfc460c838a5fc516c0e003
Autor:
Ioannis Tomazos, Vasileios Nikolaou, Christoph Licht, Spero R. Cataland, Benjamin Miller, Christian S. Haas, Janet Green, Rita D. Swinford, Elena Román-Ortiz, Imad Al-Dakkak, Larry A. Greenbaum, Hae Il Cheong, Lisa Sartz
Publikováno v:
Kidney International Reports. 5:1161-1171
Introduction Atypical hemolytic uremic syndrome (aHUS) is a progressive and potentially life-threatening disease characterized by complement-mediated thrombotic microangiopathy. Patients with aHUS may experience fatigue, which can negatively impact t
Autor:
Katja Evert, Claudius Speer, Oliver A. Cornely, Elham Khatamzas, Jessica Seeßle, Matthias Lubnow, Uta Merle, Christopher Behrens, Christian S. Haas, Igor Wolfgang Blau, Rosanne Sprute, Oliver Kurzai, Danila Seidel, Philipp Enghard, Joerg Steinmann, Michaela Simon
Publikováno v:
Mycoses
Background Most COVID‐19‐associated mucormycosis (CAM) cases are reported from India and neighbouring countries. Anecdotally cases from Europe have been presented. Objective To estimate the disease burden and describe the clinical presentation of
Background: The erythrocyte sedimentation rate (ESR) is a simple laboratory diagnostic tool for estimating systemic inflammation. It remains unclear, if renal function affects ESR, thereby compromising its validity. This pilot study aims to compare p
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8b37792c7f7cdfd275374a65c8596a52
https://doi.org/10.21203/rs.3.rs-609915/v1
https://doi.org/10.21203/rs.3.rs-609915/v1
Publikováno v:
Dtsch Arztebl Int
Autor:
Hanns-Martin Lorenz, Hans-Peter Tony, Norbert Blank, Jürgen Rech, Karoline Krause, Martin Nitschke, Ina Kötter, Dorothee Kaudewitz, Birgit Maria Köhler, Martin Krusche, Christian S Haas
Background: To investigate the clinical presentation and genetic variants in patients of the German anti-IL1 registry for autoinflammatory orphan diseases (GARROD) between 2013-2019.Methods: Multicenter, retrospective analysis of demographic, clinica
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::8a455a9c289ce8d98f6d4526d35ece5a
https://doi.org/10.21203/rs.3.rs-293402/v1
https://doi.org/10.21203/rs.3.rs-293402/v1
Publikováno v:
Clinical Kidney Journal
Background Autosomal dominant polycystic kidney disease (ADPKD) is the most common inherited kidney disease worldwide. The renal phenotype is characterized by progressive cystic enlargement of the kidneys leading to a decline in renal function, hyper
Publikováno v:
Endocrine, Metabolic & Immune Disorders-Drug Targets. 16:249-253
Background: Patients with hypothalamic-pituitary disorders (HPD) may be of increased risk to develop overweight and obesity, thereby fostering cardiovascular events. However, it remains unclear if patients with pituitary dysfunctions per se have an i
Autor:
Michael Jeglitsch, Ulf Schönermarck, Alexander Mellmann, Steffen Mitzner, Malgorzata Dunaj-Kazmierowska, Bernd Schröppel, Wolfgang Ries, Nadezda Basara, Volker Burst, Lars Pape, Rita Dittmer, Michael Starck, Daniel Schmidbauer, Christian S. Haas
Publikováno v:
Clinical Kidney Journal
Background Data are lacking on the relative incidence of thrombotic thrombocytopenic purpura (TTP), haemolytic uraemic syndrome (HUS) caused by Shiga toxin–producing Escherichia coli (STEC) and atypical HUS (aHUS) in patients presenting with thromb
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8f7186f8671f82a4ddb577fd61cd6b50
https://publica.fraunhofer.de/handle/publica/267530
https://publica.fraunhofer.de/handle/publica/267530
Publikováno v:
Journal of Nephrology
Atypical hemolytic uremic syndrome (aHUS) is a rare, genetic, progressive, life-threatening form of thrombotic microangiopathy (TMA) predominantly caused by dysregulation of the alternative pathway of the complement system. Complement-amplifying cond