Zobrazeno 1 - 10
of 86
pro vyhledávání: '"Christen P Dahl"'
Autor:
Kine Andenæs, Ida G Lunde, Naiyereh Mohammadzadeh, Christen P Dahl, Jan Magnus Aronsen, Mari E Strand, Sheryl Palmero, Ivar Sjaastad, Geir Christensen, Kristin V T Engebretsen, Theis Tønnessen
Publikováno v:
PLoS ONE, Vol 13, Iss 7, p e0201422 (2018)
Pressure overload of the heart leads to cardiac remodeling that may progress into heart failure, a common, morbid and mortal condition. Increased mechanistic insight into remodeling is instrumental for development of novel heart failure treatment. Ca
Externí odkaz:
https://doaj.org/article/d6548e337d7d4de287ebce437f1298b6
Autor:
Arne O Melleby, Mari E Strand, Andreas Romaine, Kate M Herum, Biljana Skrbic, Christen P Dahl, Ivar Sjaastad, Arnt E Fiane, Jorge Filmus, Geir Christensen, Ida G Lunde
Publikováno v:
PLoS ONE, Vol 11, Iss 10, p e0165079 (2016)
Pressure overload is a frequent cause of heart failure. Heart failure affects millions of patients worldwide and is a major cause of morbidity and mortality. Cell surface proteoglycans are emerging as molecular players in cardiac remodeling, and incr
Externí odkaz:
https://doaj.org/article/e2ee85575a0a400eaae840ee535e1ffa
Autor:
Alexandra Vanessa Finsen, Thor Ueland, Ivar Sjaastad, Trine Ranheim, Mohammed S Ahmed, Christen P Dahl, Erik T Askevold, Svend Aakhus, Cathrine Husberg, Arnt E Fiane, Martin Lipp, Lars Gullestad, Geir Christensen, Pål Aukrust, Arne Yndestad
Publikováno v:
PLoS ONE, Vol 9, Iss 11, p e112172 (2014)
BACKGROUND: CCL21 acting through CCR7, is termed a homeostatic chemokine. Based on its role in concerting immunological responses and its proposed involvement in tissue remodeling, we hypothesized that this chemokine could play a role in myocardial r
Externí odkaz:
https://doaj.org/article/6bebe3d6c6d44d9685a23fab6a865f52
Autor:
Ida Gregersen, Mona Skjelland, Sverre Holm, Kirsten B Holven, Kirsten Krogh-Sørensen, David Russell, Erik T Askevold, Christen P Dahl, Stein Ørn, Lars Gullestad, Tom E Mollnes, Thor Ueland, Pål Aukrust, Bente Halvorsen
Publikováno v:
PLoS ONE, Vol 8, Iss 8, p e72769 (2013)
OBJECTIVE: Atherosclerosis is a chronic inflammatory disorder that involves a range of inflammatory mediators. Although interleukin (IL)-9 has been related to inflammation, there are at present no data on its role in atherosclerosis. Here we have exa
Externí odkaz:
https://doaj.org/article/1409a647bb9f4850a1f6d76defc5c705
Autor:
Zoltán Prohászka, Lea Munthe-Fog, Thor Ueland, Timea Gombos, Arne Yndestad, Zsolt Förhécz, Mikkel-Ole Skjoedt, Zoltan Pozsonyi, Alice Gustavsen, Lívia Jánoskuti, István Karádi, Lars Gullestad, Christen P Dahl, Erik T Askevold, George Füst, Pål Aukrust, Tom E Mollnes, Peter Garred
Publikováno v:
PLoS ONE, Vol 8, Iss 4, p e60976 (2013)
BACKGROUND: Inflammatory mechanisms involving complement activation has been shown to take part in the pathophysiology of congestive heart failure, but the initiating mechanisms are unknown. We hypothesized that the main initiator molecules of the le
Externí odkaz:
https://doaj.org/article/d9aa4c2e045c444997483c0f62bd0674
Autor:
Arne Yndestad, Alexandra Vanessa Finsen, Thor Ueland, Cathrine Husberg, Christen P Dahl, Erik Øie, Leif Erik Vinge, Ivar Sjaastad, Øystein Sandanger, Trine Ranheim, Kenneth Dickstein, John Kjekshus, Jan Kristian Damås, Arnt E Fiane, Denise Hilfiker-Kleiner, Martin Lipp, Lars Gullestad, Geir Christensen, Pål Aukrust
Publikováno v:
PLoS ONE, Vol 7, Iss 3, p e33038 (2012)
BACKGROUND: CCL19 and CCL21, acting through CCR7, are termed homeostatic chemokines. Based on their role in concerting immunological responses and their proposed involvement in tissue remodeling, we hypothesized that these chemokines could play a pat
Externí odkaz:
https://doaj.org/article/fa097ed2b7474920a2599b5b31aabb1c
Autor:
Liv I Bjoner Sikkeland, Christen P Dahl, Thor Ueland, Arne K Andreassen, Einar Gude, Thor Edvardsen, Torbjørn Holm, Arne Yndestad, Lars Gullestad, Johny Kongerud, Pål Aukrust, Erik Øie
Publikováno v:
PLoS ONE, Vol 7, Iss 5, p e36815 (2012)
BACKGROUND: Pathophysiological interactions between heart and lungs in heart failure (HF) are well recognized. We investigated whether expression of different factors known to be increased in the myocardium and/or the circulation in HF is also increa
Externí odkaz:
https://doaj.org/article/2838d20c947644c4b1582a2473e986d9
Autor:
Anne Waehre, Bente Halvorsen, Arne Yndestad, Cathrine Husberg, Ivar Sjaastad, Ståle Nygård, Christen P Dahl, M Shakil Ahmed, Alexandra V Finsen, Henrik Reims, William E Louch, Denise Hilfiker-Kleiner, Leif E Vinge, Borghild Roald, Håvard Attramadal, Martin Lipp, Lars Gullestad, Pål Aukrust, Geir Christensen
Publikováno v:
PLoS ONE, Vol 6, Iss 4, p e18668 (2011)
RATIONALE: Inflammatory mechanisms have been suggested to play a role in the development of heart failure (HF), but a role for chemokines is largely unknown. Based on their role in inflammation and matrix remodeling in other tissues, we hypothesized
Externí odkaz:
https://doaj.org/article/adb8103069744d68b9d1120c5ecdafbe
Autor:
Athiramol Sasi, Andreas Romaine, Pugazendhi Murugan Erusappan, Arne Olav Melleby, Almira Hasic, Christen Peder Dahl, Kaspar Broch, Vibeke Marie Almaas, Rosa Doñate Puertas, H. Llewelyn Roderick, Ida Gjervold Lunde, Ivar Sjaastad, Maria Vistnes, Geir Christensen
Publikováno v:
Matrix Biology Plus, Vol 19, Iss , Pp 100135- (2023)
Cardiac fibrosis is a central pathological feature in several cardiac diseases, but the underlying molecular players are insufficiently understood. The extracellular matrix proteoglycan versican is elevated in heart failure and suggested to be a targ
Externí odkaz:
https://doaj.org/article/e019eb64945b4076a1c582261916bafe
Autor:
Chloe Rixon, Kristine Andreassen, Xin Shen, Pugazendhi Murugan Erusappan, Vibeke Marie Almaas, Sheryl Palmero, Christen Peder Dahl, Thor Ueland, Ivar Sjaastad, William Edward Louch, Mathis Korseberg Stokke, Theis Tønnessen, Geir Christensen, Ida Gjervold Lunde
Publikováno v:
ESC Heart Failure, Vol 10, Iss 2, Pp 858-871 (2023)
Abstract Aims Familial hypertrophic cardiomyopathy (HCM) is the most common form of inherited cardiac disease. It is characterized by myocardial hypertrophy and diastolic dysfunction, and can lead to severe heart failure, arrhythmias, and sudden card
Externí odkaz:
https://doaj.org/article/1ff7ce53c6d9413785c03244390f1f0d