Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Chris H, Dreyer"'
Autor:
Michael L.C. Beagan, Chris H. Dreyer, Louise K. Jensen, Henrik E. Jensen, Thomas E. Andersen, Soeren Overgaard, Ming Ding
Publikováno v:
Journal of Orthopaedic Translation, Vol 45, Iss , Pp 120-131 (2024)
Background: Reliable animal models are critical for preclinical research and should closely mimic the disease. With respect to route of infection, pathogenic agent, disease progression, clinical signs, and histopathological changes. Sheep have simila
Externí odkaz:
https://doaj.org/article/a40cb9f48d584c60ac5cf26b37e24828
Publikováno v:
Journal of Orthopaedic Translation, Vol 24, Iss , Pp 46-57 (2020)
Background: To achieve optimal bone formation one of the most influential parameters has been mentioned to be adequate blood supply. Vascular endothelial growth factor (VEGF) is hereby of particular interest in bone regeneration, because of its prima
Externí odkaz:
https://doaj.org/article/ed535086a7b24c4a92a545637fba9f2d
Autor:
Kristian Kjærgaard, Chris H. Dreyer, Nicholas Ditzel, Christina M. Andreasen, Li Chen, Søren P. Sheikh, Søren Overgaard, Ming Ding
Publikováno v:
Stem Cells International, Vol 2016 (2016)
Background. Scaffolds for bone tissue engineering (BTE) can be loaded with stem and progenitor cells (SPC) from different sources to improve osteogenesis. SPC can be found in bone marrow, adipose tissue, and other tissues. Little is known about osteo
Externí odkaz:
https://doaj.org/article/f8704dadfc494a51afeb4683e5c89d99
Autor:
Chris H, Dreyer, Kristian, Kjaergaard, Nicholas, Ditzel, Niklas R, Jørgensen, Søren, Overgaard, Ming, Ding
Publikováno v:
Journal of biomedical materials research. Part A. 105(12)
Insufficient blood supply may limit bone regeneration in bone defects. Vascular endothelial growth factor (VEGF) promotes angiogenesis by increasing endothelial migration. This outcome, however, could depend on time of application. Sheep mesenchymal
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::a77c672a6c547e0127ec8235b2e82092
https://pubmed.ncbi.nlm.nih.gov/28879669
https://pubmed.ncbi.nlm.nih.gov/28879669