Zobrazeno 1 - 10
of 19
pro vyhledávání: '"Chisen Takeuchi"'
Autor:
Makiko Shinomoto, Chisen Takeuchi, Harutsugu Tatebe, Fukiko Kitani-Morii, Takuma Ohmichi, Yuzo Fujino, Kanako Menjo, Naoto Terada, Miho Osako, Yoko Mochizuki, Satoshi Teramukai, Takahiko Tokuda, Toshiki Mizuno, Takashi Kasai
Publikováno v:
PLoS ONE, Vol 19, Iss 12, p e0311878 (2024)
The Dementia Screening Questionnaire for Individuals with Intellectual Disabilities (DSQIID) is an appropriate screening tool for detecting dementia in Down's syndrome patients. However, whether this questionnaire reflects the neuropsychiatric signs
Externí odkaz:
https://doaj.org/article/2f3332b4b8da446ea564629addcf1097
Autor:
Mika Hayakawa, Tomoyasu Matsubara, Yoko Mochizuki, Chisen Takeuchi, Motoyuki Minamitani, Masayuki Imai, Kenjiro Kosaki, Tomio Arai, Shigeo Murayama
Publikováno v:
BMC Neurology, Vol 22, Iss 1, Pp 1-9 (2022)
Abstract Background The detailed neuropathological features of patients with autosomal recessive hereditary spastic paraplegia with a thin corpus callosum (TCC) and SPG11 mutations are poorly understood, as only a few autopsies have been reported. He
Externí odkaz:
https://doaj.org/article/65737180a3ed4d4b824a504c07a51d85
Autor:
Yoko Kanbara1,2, Chisen Takeuchi3, Yoko Mochizuki3, Miho Osako3, Motoko Sasaki1,4, Hidehiko Miyake1,4,5 hide3yake@nms.ac.jp
Publikováno v:
Journal of Nippon Medical School. 2023, Vol. 90 Issue 2, p210-219. 10p.
Autor:
Hisato Suzuki, Simo Li, Tomoharu Tokutomi, Chisen Takeuchi, Miyuki Takahashi, Mamiko Yamada, Hironobu Okuno, Fuyuki Miya, Toshiki Takenouchi, Hironao Numabe, Kenjiro Kosaki, Toshio Ohshima
Publikováno v:
Human molecular genetics.
Collapsin response mediator protein 2 (Crmp2) is an evolutionarily well-conserved tubulin-binding cytosolic protein that plays critical roles in the formation of neural circuitry in model organisms including zebrafish and rodents. No clinical evidenc
Publikováno v:
Rinsho Shinkeigaku. 60:193-199
Three patients with neurodegenerative diseases who had developed repeated aspiration pneumonia underwent laryngeal closure, a surgical procedure at the larynx to prevent aspiration. None of these patients have developed aspiration pneumonia since the
Publikováno v:
Neurology: Clinical Practice. 13:e200130
Background and ObjectivesAlthough the importance of pediatric-to-adult health care transition (HCT) has been recognized, individuals with childhood-onset neurologic conditions often encounter challenges during pediatric-to-adult HCT, and HCT benefits
Publikováno v:
Rinsho Shinkeigaku. 59:279-281
We investigated the patients followed in our hospital's adult neurology department to evaluate issues during the transition from pediatric to adult health care for patients with special health-care needs for neurological diseases. There has been an i
Autor:
Takako Takemiya1 takemiya.takako@twmu.ac.jp, Chisen Takeuchi2 chisentakeuchi@gmail.com, Marumi Kawakami1 kawakami.marumi@twmu.ac.jp
Publikováno v:
International Journal of Molecular Sciences. Dec2017, Vol. 18 Issue 12, p2758. 18p.
Publikováno v:
International Journal of Molecular Sciences, Vol 19, Iss 11, p 3647 (2018)
International Journal of Molecular Sciences
Volume 19
Issue 11
International Journal of Molecular Sciences
Volume 19
Issue 11
Microsomal prostaglandin E synthetase-1 (mPGES-1) is an inducible terminal enzyme for the production of prostaglandin E2 (PGE2). In experimental autoimmune encephalomyelitis (EAE), an animal model of multiple sclerosis, mPGES-1 is induced in vascular
Publikováno v:
International Journal of Molecular Sciences; Volume 18; Issue 12; Pages: 2758
International Journal of Molecular Sciences, Vol 18, Iss 12, p 2758 (2017)
International Journal of Molecular Sciences
International Journal of Molecular Sciences, Vol 18, Iss 12, p 2758 (2017)
International Journal of Molecular Sciences
Microsomal prostaglandin synthetase-1 (mPGES-1) is an inducible terminal enzyme that produces prostaglandin E2 (PGE2). In our previous study, we investigated the role of mPGES-1 in the inflammation and demyelination observed in experimental autoimmun