Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Chiseko Ikenaga"'
Clinical utility of anti‐cytosolic 5’‐nucleotidase 1A antibody in idiopathic inflammatory myopathies
Autor:
Chiseko Ikenaga, Andrew R. Findlay, Namita A. Goyal, Sarah Robinson, Jonathan Cauchi, Yessar Hussain, Leo H. Wang, Joshua C. Kershen, Brent A. Beson, Michael Wallendorf, Robert C. Bucelli, Tahseen Mozaffar, Alan Pestronk, Conrad C. Weihl
Publikováno v:
Annals of Clinical and Translational Neurology, Vol 8, Iss 3, Pp 571-578 (2021)
Abstract Objective To define the clinicopathologic features and diagnostic utility associated with anti‐cytosolic 5′‐nucleotidase 1A (NT5C1A) antibody seropositivity in idiopathic inflammatory myopathies (IIMs). Methods Anti‐NT5C1A antibody s
Externí odkaz:
https://doaj.org/article/dbc8d0799a744d33a15998849a84520d
Autor:
Chiseko Ikenaga, Andrew R. Findlay, Michelle Seiffert, Allison Peck, Nathan Peck, Nicholas E. Johnson, Jeffrey M. Statland, Conrad C. Weihl
Publikováno v:
Orphanet Journal of Rare Diseases, Vol 15, Iss 1, Pp 1-9 (2020)
Abstract Background Dominant mutations in valosin-containing protein (VCP) gene cause an adult onset inclusion body myopathy, Paget’s disease of bone, and frontotemporal dementia also termed multisystem proteinopathy (MSP). The genotype-phenotype r
Externí odkaz:
https://doaj.org/article/a7d0cfa7337e4ee6a7193852a449c628
Publikováno v:
Tuesday, April 25.
Clinical utility of anti‐cytosolic 5’‐nucleotidase 1A antibody in idiopathic inflammatory myopathies
Autor:
Leo H. Wang, Namita Goyal, Alan Pestronk, Jonathan Cauchi, Conrad C. Weihl, Yessar Hussain, Sarah Robinson, Chiseko Ikenaga, Joshua C. Kershen, Michael Wallendorf, Robert C. Bucelli, Andrew R. Findlay, Tahseen Mozaffar, Brent A. Beson
Publikováno v:
Annals of Clinical and Translational Neurology, Vol 8, Iss 3, Pp 571-578 (2021)
Annals of clinical and translational neurology, vol 8, iss 3
Annals of Clinical and Translational Neurology
Annals of clinical and translational neurology, vol 8, iss 3
Annals of Clinical and Translational Neurology
Author(s): Ikenaga, Chiseko; Findlay, Andrew R; Goyal, Namita A; Robinson, Sarah; Cauchi, Jonathan; Hussain, Yessar; Wang, Leo H; Kershen, Joshua C; Beson, Brent A; Wallendorf, Michael; Bucelli, Robert C; Mozaffar, Tahseen; Pestronk, Alan; Weihl, Con
Autor:
Chiseko Ikenaga, Hidetoshi Date, Motoi Kanagawa, Jun Mitsui, Hiroyuki Ishiura, Jun Yoshimura, Iago Pinal‐Fernandez, Andrew L. Mammen, Thomas E. Lloyd, Shoji Tsuji, Jun Shimizu, Tatsushi Toda, Jun Goto
Publikováno v:
O2, repositorio institucional de la UOC
Universitat Oberta de Catalunya (UOC)
Universitat Oberta de Catalunya (UOC)
Objective: This study aimed to elucidate the molecular features of inclusion body myositis (IBM). Methods: We performed RNA sequencing analysis of muscle biopsy samples from 67 participants, consisting of 58 myositis patients with the pathological fi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f0116da8f19ebdd0c863d2f66ce66a03
Autor:
Kyla A. Britson, Jonathan P. Ling, Kerstin E. Braunstein, Janelle M. Montagne, Jenna M. Kastenschmidt, Andrew Wilson, Chiseko Ikenaga, William Tsao, Iago Pinal-Fernandez, Katelyn A. Russell, Nicole Reed, Tahseen Mozaffar, Kathryn R. Wagner, Lyle W. Ostrow, Andrea M. Corse, Andrew L. Mammen, S. Armando Villalta, H. Benjamin Larman, Philip C. Wong, Thomas E. Lloyd
Publikováno v:
Science translational medicine, vol 14, iss 628
Sci Transl Med
Sci Transl Med
Sporadic inclusion body myositis (IBM) is the most common acquired muscle disease in adults over age 50, yet it remains unclear whether the disease is primarily driven by T cell–mediated autoimmunity. IBM muscle biopsies display nuclear clearance a
Autor:
Chiseko Ikenaga, Jiang Zhu, Abdallah M. Eteleeb, Rolf Schröder, Andrew D. Sauerbeck, Carla M. Yuede, Jason D. Ulrich, Sara K. Pittman, Conrad C. Weihl, Khalid Arhzaouy, Feng Wang, Sydney J. Reitz, Oscar Harari, Terrance T. Kummer, Christoph S. Clemen, Abubakar Wani, Shan Li, Bruno A. Benitez, Carlos Cruchaga, Tsui-Fen Chou
Publikováno v:
Cell reports
SUMMARY The pathogenic mechanism by which dominant mutations in VCP cause multisystem proteinopathy (MSP), a rare neurodegenerative disease that presents as fronto-temporal lobar degeneration with TDP-43 inclusions (FTLD-TDP), remains unclear. To exp
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::16965f9cc536d611d8be1b4d52b3c971
https://resolver.caltech.edu/CaltechAUTHORS:20210811-212815828
https://resolver.caltech.edu/CaltechAUTHORS:20210811-212815828
Autor:
Allison Peck, Conrad C. Weihl, Jeffrey Statland, Michelle Seiffert, Nicholas E. Johnson, Nathan Peck, Chiseko Ikenaga, Andrew R. Findlay
Publikováno v:
Orphanet Journal of Rare Diseases
Orphanet Journal of Rare Diseases, Vol 15, Iss 1, Pp 1-9 (2020)
Orphanet Journal of Rare Diseases, Vol 15, Iss 1, Pp 1-9 (2020)
Background Dominant mutations in valosin-containing protein (VCP) gene cause an adult onset inclusion body myopathy, Paget’s disease of bone, and frontotemporal dementia also termed multisystem proteinopathy (MSP). The genotype-phenotype relationsh
Autor:
Naohiro Uchio, Kenichiro Taira, Chiseko Ikenaga, Atsushi Unuma, Masato Kadoya, Akatsuki Kubota, Tatsushi Toda, Jun Shimizu
Publikováno v:
Neurology® Neuroimmunology & Neuroinflammation; Jul2018, Vol. 5 Issue 4, p1-3, 3p