Utility of Glycosylated TIMP3 molecules: Inhibition of MMPs and TACE to improve cardiac function in rat myocardial infarct model.

Autor: Chintalgattu, Vishnu¹ (AUTHOR), Greenberg, Joanne¹ (AUTHOR), Singh, Shivani¹ (AUTHOR), Chiueh, Venice¹ (AUTHOR), Gilbert, Amy¹ (AUTHOR), O'Neill, Jason W.¹ (AUTHOR), Smith, Stephen¹ (AUTHOR), Jackson, Simon¹ (AUTHOR), Khakoo, Aarif Y.¹ (AUTHOR), Lee, TaeWeon¹ (AUTHOR) TAEWEONL@amgen.com

Publikováno v: Pharmacology Research & Perspectives. Dec2018, Vol. 6 Issue 6, p1-N.PAG. 14p.

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Cardiac myofibroblasts: a novel source of vascular endothelial growth factor (VEGF) and its receptors Flt-1 and KDR

Autor: Chintalgattu, Vishnu, Nair, Devi M, Katwa, Laxmansa C ^*

Publikováno v: In Journal of Molecular and Cellular Cardiology 2003 35(3):277-286

Identification, cloning and functional characterization of novel beta-defensins in the rat (Rattus norvegicus)

Autor: French Frank S, Radhakrishnan Yashwanth, Wingard Christopher J, Chintalgattu Vishnu, Yenugu Suresh, Hall Susan H

Publikováno v: Reproductive Biology and Endocrinology, Vol 4, Iss 1, p 7 (2006)

Abstract Background beta-defensins are small cationic peptides that exhibit broad spectrum antimicrobial properties. The majority of beta-defensins identified in humans are predominantly expressed in the male reproductive tract and have roles in non-

Externí odkaz: https://doaj.org/article/1b0266d8428f4b76ac637b9d0c8106f6

Cardiac myocyte p38α kinase regulates angiogenesis via myocyte-endothelial cell cross-talk during stress-induced remodeling in the heart

Autor: Rose, Beth A, Yokota, Tomohiro, Chintalgattu, Vishnu, Ren, Shuxun, Iruela-Arispe, Luisa, Khakoo, Aarif Y, Minamisawa, Susumu, Wang, Yibin

Publikováno v: The Journal of biological chemistry, vol 292, iss 31
Rose, Beth A; Yokota, Tomohiro; Chintalgattu, Vishnu; Ren, Shuxun; Iruela-Arispe, Luisa; Khakoo, Aarif Y; et al.(2017). Cardiac myocyte p38α kinase regulates angiogenesis via myocyte-endothelial cell cross-talk during stress-induced remodeling in the heart.. The Journal of biological chemistry, 292(31), 12787-12800. doi: 10.1074/jbc.M117.784553. UC Office of the President: Research Grants Program Office (RGPO). Retrieved from: http://www.escholarship.org/uc/item/8762r1zt

Stress-induced p38 mitogen-activated protein kinase (MAPK) activity is implicated in pathological remodeling in the heart. For example, constitutive p38 MAPK activation in cardiomyocytes induces pathological features, including myocyte hypertrophy, a

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=dedup_wf_001::0b4705526a490eee2a7cd8846119310f
https://escholarship.org/uc/item/8762r1zt