Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Chiara Starvaggi Cucuzza"'
Autor:
Majid Pahlevan Kakhki, Antonino Giordano, Chiara Starvaggi Cucuzza, Tejaswi Venkata S. Badam, Samudyata Samudyata, Marianne Victoria Lemée, Pernilla Stridh, Asimenia Gkogka, Klementy Shchetynsky, Adil Harroud, Alexandra Gyllenberg, Yun Liu, Sanjaykumar Boddul, Tojo James, Melissa Sorosina, Massimo Filippi, Federica Esposito, Fredrik Wermeling, Mika Gustafsson, Patrizia Casaccia, Jan Hillert, Tomas Olsson, Ingrid Kockum, Carl M. Sellgren, Christelle Golzio, Lara Kular, Maja Jagodic
Publikováno v:
Nature Communications, Vol 15, Iss 1, Pp 1-17 (2024)
Abstract Multiple Sclerosis (MS) is a heterogeneous inflammatory and neurodegenerative disease with an unpredictable course towards progressive disability. Treating progressive MS is challenging due to limited insights into the underlying mechanisms.
Externí odkaz:
https://doaj.org/article/335891136a424c99812fda12c3ca1d91
Autor:
Kamila Zondra Revendova, Chiara Starvaggi Cucuzza, Ali Manouchehrinia, Mohsen Khademi, Michal Bar, David Leppert, Elisabeth Sandberg, Russell Ouellette, Tobias Granberg, Fredrik Piehl
Publikováno v:
Brain and Behavior, Vol 13, Iss 1, Pp n/a-n/a (2023)
Abstract Background Neurofilament light (NfL) levels reflect inflammatory disease activity in multiple sclerosis (MS), but it is less clear if NfL also can serve as a biomarker for MS progression in treated patients without relapses and focal lesion
Externí odkaz:
https://doaj.org/article/20c0b2d7dd214ee28b9e64e30ad63797
Autor:
Mattia Pozzato, Emanuele Micaglio, Chiara Starvaggi Cucuzza, Alessandro Cagol, Daniela Galimberti, Daniela Calandrella, Claudia Cinnante, Carlo Pappone, Monica Zanussi, Giovanni Meola, Elio Scarpini, Nereo Bresolin, Filippo Martinelli Boneschi
Publikováno v:
Frontiers in Neurology, Vol 11 (2021)
Familial Mediterranean Fever (FMF) is a genetic autoinflammatory disease characterized by recurrent episodes of fever and serositis caused by mutations in the MEFV gene, while Multiple Sclerosis (MS) is an inflammatory demyelinating disease of the CN
Externí odkaz:
https://doaj.org/article/7f93f91c898e4da1b7875a87f5f2acd1
Autor:
Kamila Zondra Revendova, Chiara Starvaggi Cucuzza, Ali Manouchehrinia, Mohsen Khademi, Michal Bar, David Leppert, Elisabeth Sandberg, Russell Ouellette, Tobias Granberg, Fredrik Piehl
Publikováno v:
Brain and Behavior. 13
Neurofilament light (NfL) levels reflect inflammatory disease activity in multiple sclerosis (MS), but it is less clear if NfL also can serve as a biomarker for MS progression in treated patients without relapses and focal lesion accrual. In addition
Autor:
null Kamila Zondra Revendova, null Chiara Starvaggi Cucuzza, null Ali Manouchehrinia, null Mohsen Khademi, null Michal Bar, null David Leppert, null Elisabeth Sandberg, null Russell Ouellette, null Tobias Granberg, null Fredrik Piehl
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::063dcaa8274b2ec0728b476c5612cc3c
https://doi.org/10.1002/brb3.2873/v2/response1
https://doi.org/10.1002/brb3.2873/v2/response1
Autor:
Chiara, Starvaggi Cucuzza, Elisa, Longinetti, Nicolas, Ruffin, Björn, Evertsson, Ingrid, Kockum, Maja, Jagodic, Faiez, Al Nimer, Thomas, Frisell, Fredrik, Piehl
Publikováno v:
Neurology(R) neuroimmunologyneuroinflammation. 10(1)
B cell-depleting therapies are highly effective in relapsing-remitting multiple sclerosis (RRMS) but are associated with increased infection risk and blunted humoral vaccination responses. Extension of dosing intervals may mitigate such negative effe
Autor:
Majid Pahlevan Kakhki, Chiara Starvaggi Cucuzza, Antonino Giordano, Tejaswi Venkata S. Badam, Pernilla Strid, Klementy Shchetynsky, Adil Harroud, Alexandra Gyllenberg, Yun Liu, Sanjaykumar Boddul, Tojo James, Melissa Sorosina, Massimo Filippi, Federica Esposito, Fredrik Wermeling, Mika Gustafsson, Patrizia Casaccia, Ingrid Kockum, Jan Hillert, Tomas Olsson, Lara Kular, Maja Jagodic
Multiple Sclerosis (MS) is a heterogeneous inflammatory and neurodegenerative disease of the central nervous system with an unpredictable course toward progressive disability. Understanding and treating progressive MS remains extremely challenging du
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::8f1900f75e757f2e0255011b7e1f18e3
https://doi.org/10.1101/2022.05.06.22274611
https://doi.org/10.1101/2022.05.06.22274611
Autor:
Lawrence Rajendran, Alice Canzi, Laura Piccio, Valerio Zerbi, Matteo Tamborini, Rosa C. Paolicelli, Raffaella Morini, Stefania Vetrano, Fabio Perrucci, Marco Erreni, Elisabetta Menna, Francesca Cignarella, Bernadeta Michalski, Margaret Fahnestock, Karel Otero, Marco Genua, Irene Corradini, Fabia Filipello, Chiara Starvaggi-Cucuzza, Michela Matteoli, Marija Markicevic
Publikováno v:
Immunity
The triggering receptor expressed on myeloid cells 2 (TREM2) is a microglial innate immune receptor associated with a lethal form of early, progressive dementia, Nasu-Hakola disease, and with an increased risk of Alzheimer's disease. Microglial defec
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d04c02199d5667fe2850d50d17423dcf
https://www.zora.uzh.ch/id/eprint/167227/
https://www.zora.uzh.ch/id/eprint/167227/
Autor:
Antonino Natalello, Marco Rasile, Eliana Lauranzano, Sonia Mazzitelli, Davide Pozzi, Isabella Barajon, Matteo Tamborini, Chiara Starvaggi-Cucuzza, Fabia Filipello, Michela Matteoli, Toni Giorgino
Publikováno v:
Acta Neuropathologica Communications 4(1), 110 (2016). doi:10.1186/s40478-016-0381-9
Acta Neuropathologica Communications
Acta Neuropathologica Communications 4 (2016): 110. doi:10.1186/s40478-016-0381-9
info:cnr-pdr/source/autori:Mazzitelli S.; Filipello F.; Rasile M.; Lauranzano E.; Starvaggi-Cucuzza C.; Tamborini M.; Pozzi D.; Barajon I.; Giorgino T.; Natalello A.; Matteoli M./titolo:Amyloid-beta 1-24 C-terminal truncated fragment promotes amyloid-beta 1-42 aggregate formation in the healthy brain/doi:10.1186%2Fs40478-016-0381-9/rivista:Acta Neuropathologica Communications/anno:2016/pagina_da:110/pagina_a:/intervallo_pagine:110/volume:4
Scopus-Elsevier
Acta Neuropathologica Communications
Acta Neuropathologica Communications 4 (2016): 110. doi:10.1186/s40478-016-0381-9
info:cnr-pdr/source/autori:Mazzitelli S.; Filipello F.; Rasile M.; Lauranzano E.; Starvaggi-Cucuzza C.; Tamborini M.; Pozzi D.; Barajon I.; Giorgino T.; Natalello A.; Matteoli M./titolo:Amyloid-beta 1-24 C-terminal truncated fragment promotes amyloid-beta 1-42 aggregate formation in the healthy brain/doi:10.1186%2Fs40478-016-0381-9/rivista:Acta Neuropathologica Communications/anno:2016/pagina_da:110/pagina_a:/intervallo_pagine:110/volume:4
Scopus-Elsevier
Substantial data indicate that amyloid-? (A?), the major component of senile plaques, plays a central role in Alzheimer's Disease and indeed the assembly of naturally occurring amyloid peptides into cytotoxic aggregates is linked to the disease patho