Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Chiara Allocca"'
Publikováno v:
Thyroid (N.Y.N.Y.) (2020). doi:10.1089/thy.2019.0728
info:cnr-pdr/source/autori:Allocca, Chiara; Cirafici, Anna Maria; Laukkanen, Mikko O.; Castellone, Maria Domenica/titolo:Serine 897 Phosphorylation of EPHA2 Is Involved in Signaling of Oncogenic ERK1%2F2 Drivers in Thyroid Cancer Cells/doi:10.1089%2Fthy.2019.0728/rivista:Thyroid (N.Y.N.Y.)/anno:2020/pagina_da:/pagina_a:/intervallo_pagine:/volume
info:cnr-pdr/source/autori:Allocca, Chiara; Cirafici, Anna Maria; Laukkanen, Mikko O.; Castellone, Maria Domenica/titolo:Serine 897 Phosphorylation of EPHA2 Is Involved in Signaling of Oncogenic ERK1%2F2 Drivers in Thyroid Cancer Cells/doi:10.1089%2Fthy.2019.0728/rivista:Thyroid (N.Y.N.Y.)/anno:2020/pagina_da:/pagina_a:/intervallo_pagine:/volume
Background: Phosphorylation of the intracellular domain of the EPHA2 receptor tyrosine kinase (RTK) on serine 897 (S897) has been demonstrated to mediate EPHA2 oncogenic activity. Here, we show that in thyroid cancer cells harboring driver oncogenes
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0893828f2e3517cf7c816d0a96d2feec
http://www.cnr.it/prodotto/i/433839
http://www.cnr.it/prodotto/i/433839
Publikováno v:
Encyclopedia of Signaling Molecules ISBN: 9781461464389
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0433dfce1036a9c272b3ef6ff15e98ca
https://doi.org/10.1007/978-3-319-67199-4_101649
https://doi.org/10.1007/978-3-319-67199-4_101649
Autor:
Clara Ugolini, Mikko O. Laukkanen, Giancarlo Vecchio, Beatrice Cobucci-Ponzano, Maria Domenica Castellone, Nobuo Tsuchida, Marco Moracci, Alessia Parascandolo, Andrea Strazzulli, Fulvio Basolo, Chiara Allocca
Publikováno v:
Oncotarget
Oncotarget 8 (2017): 27075–27092. doi:10.18632/oncotarget.15635
info:cnr-pdr/source/autori:Vecchio G.; Parascandolo A.; Allocca C.; Ugolini C.; Basolo F.; Moracci M.; Strazzulli A.; Cobucci-Ponzano B.; Laukkanen M.O.; Castellone M.D.; Tsuchida N./titolo:Human ?-L-fucosidase-1 attenuates the invasive properties of thyroid cancer/doi:10.18632%2Foncotarget.15635/rivista:Oncotarget/anno:2017/pagina_da:27075/pagina_a:27092/intervallo_pagine:27075–27092/volume:8
Oncotarget 8 (2017): 27075–27092. doi:10.18632/oncotarget.15635
info:cnr-pdr/source/autori:Vecchio G.; Parascandolo A.; Allocca C.; Ugolini C.; Basolo F.; Moracci M.; Strazzulli A.; Cobucci-Ponzano B.; Laukkanen M.O.; Castellone M.D.; Tsuchida N./titolo:Human ?-L-fucosidase-1 attenuates the invasive properties of thyroid cancer/doi:10.18632%2Foncotarget.15635/rivista:Oncotarget/anno:2017/pagina_da:27075/pagina_a:27092/intervallo_pagine:27075–27092/volume:8
// Giancarlo Vecchio 1, 2, * , Alessia Parascandolo 3, * , Chiara Allocca 1 , Clara Ugolini 4 , Fulvio Basolo 5 , Marco Moracci 6, 7 , Andrea Strazzulli 6, 7 , Beatrice Cobucci-Ponzano 6 , Mikko O. Laukkanen 3 , Maria Domenica Castellone 8 , Nobuo Ts
Autor:
Niko Sahlberg, Alessia Parascandolo, Massimo Santoro, Maria Carmela Cantisani, Vidal Fey, Francesco Merolla, Olli Kallioniemi, Chiara Allocca, Maria Domenica Castellone, Merja Perälä, Mikko O. Laukkanen, Fulvio Basolo
Publikováno v:
Oncotarget
Cantisani, M C, Parascandolo, A, Perälä, M, Allocca, C, Fey, V, Sahlberg, N, Merolla, F, Basolo, F, Laukkanen, M O, Kallioniemi, O P, Santoro, M & Castellone, M D 2016, ' A loss-of-function genetic screening identifies novel mediators of thyroid cancer cell viability ', Oncotarget, vol. 7, no. 19, pp. 28510-28522 . https://doi.org/10.18632/oncotarget.8577
Cantisani, M C, Parascandolo, A, Perälä, M, Allocca, C, Fey, V, Sahlberg, N, Merolla, F, Basolo, F, Laukkanen, M O, Kallioniemi, O P, Santoro, M & Castellone, M D 2016, ' A loss-of-function genetic screening identifies novel mediators of thyroid cancer cell viability ', Oncotarget, vol. 7, no. 19, pp. 28510-28522 . https://doi.org/10.18632/oncotarget.8577
RET, BRAF and other protein kinases have been identified as major molecular players in thyroid cancer. To identify novel kinases required for the viability of thyroid carcinoma cells, we performed a RNA interference screening in the RET/PTC1(CCDC6-RE