Výsledky vyhledávání - "Cheng Y. Lin"

NK but Not CD1-Restricted NKT Cells Facilitate Systemic Inflammation during Polymicrobial Intra-Abdominal Sepsis

Autor: Anthony Etogo, Jesus Nunez, Edward R. Sherwood, Cheng Y. Lin, Tracy Toliver-Kinsky

Publikováno v: The Journal of Immunology. 180:6334-6345

Evidence suggests that NK and NKT cells contribute to inflammation and mortality during septic shock caused by cecal ligation and puncture (CLP). However, the specific contributions of these cell types to the pathogenesis of CLP-induced septic shock

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2e8b2c87e1fb578d589a3de74ef001fb
https://doi.org/10.4049/jimmunol.180.9.6334

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CD4+ T-CELL DEPLETION IS NOT ASSOCIATED WITH ALTERATIONS IN SURVIVAL, BACTERIAL CLEARANCE, AND INFLAMMATION AFTER CECAL LIGATION AND PUNCTURE

Autor: Scott H. Lin, Victor T. Enoh, Cheng Y. Lin, Anthony Etogo, Edward R. Sherwood

Publikováno v: Shock. 29:56-64

Our recent studies indicate that mice depleted of T cells that bear the alphabeta T-cell receptor (alphabeta T cells) show less inflammation, less physiological dysfunction, and improved survival after cecal ligation and puncture (CLP) compared with

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a0d9e565cf7ed1e56cdd73752191fec2
https://doi.org/10.1097/shk.0b013e318070c8b9

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MICE DEPLETED OF αβ BUT NOT γδ T CELLS ARE RESISTANT TO MORTALITY CAUSED BY CECAL LIGATION AND PUNCTURE

Autor: Victor T. Enoh, Cheng Y. Lin, Tracy Toliver-Kinsky, Scott H. Lin, E. D. Murphey, Edward R. Sherwood, Tushar K. Varma

Publikováno v: Shock. 27:507-519

The present study was undertaken to determine whether the mice depleted of alphabeta or gammadelta T cells show resistance to acute polymicrobial sepsis caused by cecal ligation and puncture (CLP). T-cell receptor beta knockout (betaTCRKO) and T-cell

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::8c9fde890f5bce283454a66371858062
https://doi.org/10.1097/shk.0b013e31802b5d9f

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Differential effect of imipenem treatment on wild-type and NK cell-deficient CD8 knockout mice during acute intra-abdominal injury

Autor: Cheng Y. Lin, Tushar K. Varma, Victor T. Enoh, Edward R. Sherwood, Chad D Fairchild

Publikováno v: American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 290:R685-R693

CD8 knockout mice depleted of natural killer (NK) cells by treatment with anti-asialoGM1 (CD8KO/alphaAsGM1 mice) are resistant to injury caused by cecal ligation and puncture (CLP). However, CLP-induced injury is complex. Potential sources of injury

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d085370e08b4ef17285923877ad7e226
https://doi.org/10.1152/ajpregu.00678.2005

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Differential effect of imipenem treatment on injury caused by cecal ligation and puncture in wild-type and NK cell-deficient β2-microgloblin knockout mice

Autor: Tushar K. Varma, Cheng Y. Lin, Edward R. Sherwood, Victor T. Enoh

Publikováno v: American Journal of Physiology-Gastrointestinal and Liver Physiology. 290:G277-G284

Our previous studies showed that beta(2)-microglobulin knockout mice treated with anti-asialoGM1 (beta2MKO/alphaAsGM1 mice) are resistant to injury caused by cecal ligation and puncture (CLP). However, CLP-induced injury is complex. Potential mechani

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9a63553b75c467c7611900f8940a9dfa
https://doi.org/10.1152/ajpgi.00338.2005

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Endotoxin Priming Improves Clearance ofPseudomonas aeruginosain Wild-Type and Interleukin-10 Knockout Mice

Autor: Zhiyu Huang, Tracy Toliver-Kinsky, Edward R. Sherwood, E. D. Murphey, Tushar K. Varma, Weihua Cui, Megan Durham, Cheng Y. Lin

Publikováno v: Infection and Immunity. 73:7340-7347

Endotoxin (lipopolysaccharide [LPS]) tolerance is an altered state of immunity caused by prior exposure to LPS, in which production of many cytokines, including gamma interferon (IFN-γ) and interleukin-12 (IL-12), are reduced but secretion of the an

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e0fc94ee4097e270128ca69688b05a2d
https://doi.org/10.1128/iai.73.11.7340-7347.2005

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Cardiovascular dysfunction caused by cecal ligation and puncture is attenuated in CD8 knockout mice treated with anti-asialoGM1

Autor: Peng Li, William E. Johnston, Victor T. Enoh, Weike Tao, Edward R. Sherwood, Cheng Y. Lin

Publikováno v: American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 289:R478-R485

The present study was designed to assess hemodynamics and myocardial function at 18 h after injury caused by cecal ligation and puncture (CLP) in CD8-knockout mice treated with anti-asialoGM1 (CD8KO/alphaAsGM1 mice). Arterial pressure was measured by

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2b442a5c121ac6c6018d56291dbc0292
https://doi.org/10.1152/ajpregu.00081.2005

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Mice depleted of CD8+ T and NK cells are resistant to injury caused by cecal ligation and puncture

Autor: E. D. Murphey, Cheng Y. Lin, Victor T. Enoh, Edward R. Sherwood

Publikováno v: Laboratory Investigation. 84:1655-1665

We previously showed that beta 2 microglobulin knockout mice depleted of NK cells by treatment with anti-asialoGM1 (beta2MKO/alphaAsGM1 mice) are resistant to sepsis caused by cecal ligation and puncture (CLP). beta2MKO mice possess multiple immunolo

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1c0c81b81e140ac796fa951e42d31a1a
https://doi.org/10.1038/labinvest.3700184

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