Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Chelsea M. Heisler"'
Autor:
Corinna A. Kulicke, Gwendolyn M. Swarbrick, Nicole A. Ladd, Meghan Cansler, Megan Null, Aneta Worley, Chance Lemon, Tania Ahmed, Joshua Bennett, Taylor N. Lust, Chelsea M. Heisler, Megan E. Huber, Jason R. Krawic, Laurisa M. Ankley, Savannah K. McBride, Fikadu G. Tafesse, Andrew J. Olive, William H. Hildebrand, Deborah A. Lewinsohn, Erin J. Adams, David M. Lewinsohn, Melanie J. Harriff
Publikováno v:
Communications Biology, Vol 7, Iss 1, Pp 1-13 (2024)
Abstract MR1-restricted T cells have been implicated in microbial infections, sterile inflammation, wound healing and cancer. Similar to other antigen presentation molecules, evidence supports multiple, complementary MR1 antigen presentation pathways
Externí odkaz:
https://doaj.org/article/a188a5d473ac493d8d7f9aa7fe4c7e44
Publikováno v:
American Journal of Respiratory Cell and Molecular Biology. 68:90-102
Chronic obstructive pulmonary disease (COPD) is associated with airway inflammation, increased infiltration by CD8
Chronic obstructive pulmonary disease (COPD) is associated with airway inflammation, increased infiltration by CD8+ T lymphocytes, and infection-driven exacerbations. COPD is most commonly caused by cigarette smoke (CS), however the mechanisms drivin
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::1539093eb09bf37dd6d4d7a4dbbae7c6
https://doi.org/10.1101/2022.02.28.482383
https://doi.org/10.1101/2022.02.28.482383
Autor:
Megan E. Huber, Fikadu G. Tafesse, Melanie J. Harriff, Chelsea M. Heisler, Elham Karamooz, Regina Kurapova
Publikováno v:
Scientific Reports
Mucosal-associated invariant T (MAIT) cells are an innate-like T cell subset important in the early response to bacterial and viral lung pathogens. MAIT cells recognize bacterial small molecule metabolites presented on the Class I-like molecule MR1.
Publikováno v:
The Journal of Immunology. 206:93.08-93.08
MAIT cells, a highly prevalent subset of T cells restricted by the MHC Class I-like molecule MR1, play a key role in early response to bacterial infection. Chronic obstructive pulmonary disease (COPD) pathogenesis is associated with airway inflammati