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Autor:
Tyler D. Bold, Justin L. Benoit, Timothy W. Schacker, Ryan A. Langlois, Faheem W. Guirgis, Helen Voelker, Dana Bryne, Joseph S. Koopmeiners, Matthew T. Aliota, Lisa H. Merck, Kartikeya Cherabuddi, Kenneth B. Beckman, James Galbraith, Ronald Reilkoff, Alan E. Jones, Chas Salmen, Lauren Page Black, Jeffrey G. Chipman, Alex Hall, Margaret Beyer, Michael A. Puskarich, Joseph Farhat, Brian W. Roberts, David A. Wacker, Andrew M South, David W. Wright, Michelle H. Biros, Brian E. Driver, Nicholas E. Ingraham, Andrew C. Nelson, Christopher J. Tignanelli, Courtney V. Fletcher, Christopher Lewandowski, Thomas A. Murray
BackgroundSARS-CoV-2 viral entry may disrupt angiotensin II (Ang II) homeostasis in part via ACE2 downregulation, potentially contributing to COVID-19 induced lung injury. Preclinical models of viral pneumonias that utilize ACE2 demonstrate Ang II ty
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::71e16121d4e69bf9372be3ecf1fb9089
https://doi.org/10.1101/2021.08.25.21262623
https://doi.org/10.1101/2021.08.25.21262623