Zobrazeno 1 - 10
of 12
pro vyhledávání: '"Charuta Ambardekar"'
Publikováno v:
PLoS Pathogens, Vol 13, Iss 5, p e1006417 (2017)
The RIG-I-like RNA helicase (RLR)-mediated interferon (IFN) response plays a pivotal role in the hepatic antiviral immunity. The hepatitis A virus (HAV) and the hepatitis C virus (HCV) counter this response by encoding a viral protease that cleaves t
Externí odkaz:
https://doaj.org/article/53922e82cb1d4422b5f1b3be95449a9d
Autor:
Chang Liu, Wei Cai, Xin Yin, Zimin Tang, Guiping Wen, Charuta Ambardekar, Xinlei Li, Dong Ying, Zongdi Feng, Zizheng Zheng, Ningshao Xia
Publikováno v:
Viruses, Vol 11, Iss 1, p 64 (2019)
Hepatitis E virus (HEV) is a common cause of acute hepatitis worldwide. Current methods for evaluating the neutralizing activity of HEV-specific antibodies include immunofluorescence focus assays (IFAs) and real-time PCR, which are insensitive and op
Externí odkaz:
https://doaj.org/article/61dea37e06e94119a78dd80d77fcd131
Autor:
Philipp Ralfs, Brantley Holland, Eduardo Salinas, Bill Bremer, Minghang Wang, Jingting Zhu, Charuta Ambardekar, Heather Blasczyk, Christopher M. Walker, Zongdi Feng, Arash Grakoui
Publikováno v:
Hepatology.
Autor:
Gulce Sari, Jingting Zhu, Charuta Ambardekar, Andre Boonstra, Xin Yin, Zongdi Feng, Thomas Vanwolleghem
Publikováno v:
Journal of Virology, 95(23):e00585-21. American Society for Microbiology
J Virol
Journal of virology
J Virol
Journal of virology
Hepatitis E virus (HEV), an enterically transmitted RNA virus, is a major cause of acute hepatitis worldwide. Additionally, HEV genotype 3 (gt3) can frequently persist in immunocompromised individuals with an increased risk for developing severe live
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e6061b57dadd45e87951b3bc16a3df7c
https://pure.eur.nl/en/publications/6a9ac4de-ee80-483b-9309-06cfcdc83c78
https://pure.eur.nl/en/publications/6a9ac4de-ee80-483b-9309-06cfcdc83c78
Autor:
Philipp Ralfs, Eduardo Salinas, Charuta Ambardekar, Bill Bremer, Heather Blasczyk, Jingting Zhu, Brantley Holland, Christopher M Walker, Zongdi Feng, Arash Grakoui
Publikováno v:
The Journal of Immunology. 206:20.34-20.34
The hepatitis E virus (HEV) is a small, positive-stranded RNA virus that is a major cause of acute viral hepatitis globally. Acute HEV infection is typically asymptomatic and resolves within 8–10 weeks. HEV encodes 2 forms of capsid protein. A cyto
Autor:
Dong Ying, Chang Liu, Zi-Min Tang, Zongdi Feng, Zizheng Zheng, Wei Cai, Gui-Ping Wen, Charuta Ambardekar, Xinlei Li, Xin Yin, Ningshao Xia
Publikováno v:
Viruses, Vol 11, Iss 1, p 64 (2019)
Viruses
Viruses
Hepatitis E virus (HEV) is a common cause of acute hepatitis worldwide. Current methods for evaluating the neutralizing activity of HEV-specific antibodies include immunofluorescence focus assays (IFAs) and real-time PCR, which are insensitive and op
Autor:
Takaharu G. Yamamoto, Charuta Ambardekar, Kanisha Kavdia, Kiran Kodali, Zeenia Kaul, Ruishan Wang, Risa Kitagawa, Anthony A. High
Publikováno v:
Development. 140:2149-2159
The anaphase inhibitor securin plays a crucial role in regulating the timing of sister chromatid separation during mitosis. When sister chromatid pairs become bioriented, the E3 ligase anaphase promoting complex/cyclosome (APC/C) ubiquitylates securi
Publikováno v:
PLoS Pathogens
PLoS Pathogens, Vol 13, Iss 5, p e1006417 (2017)
PLoS Pathogens, Vol 13, Iss 5, p e1006417 (2017)
The RIG-I-like RNA helicase (RLR)-mediated interferon (IFN) response plays a pivotal role in the hepatic antiviral immunity. The hepatitis A virus (HAV) and the hepatitis C virus (HCV) counter this response by encoding a viral protease that cleaves t
The hepatitis E virus (HEV) sheds into feces as nonenveloped virions but circulates in the blood in a membrane-associated, quasi-enveloped form (eHEV). Since the eHEV virions lack viral proteins on the surface, we investigated the entry mechanism for
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::223c65e782975c0d3487140db47f6288
https://europepmc.org/articles/PMC4810531/
https://europepmc.org/articles/PMC4810531/
Autor:
Charuta Ambardekar, Simge Akbulut, Barbara Canciani, Steven E. Quatela, Priya Aggarwal, Mark Phillips, Alagarsamy Lakku Reddi, Tomas Vilimas, Jacqueline M. Mason, M. Albert Basson, Samuel L. Collins, Jonathan D. Licht, Jonathan D. Powell, Matthew Lovatt, Laura M. Hix, Marianne K.H. Kim
Publikováno v:
Molecular Biology of the Cell
PLCγ03B3 binds Spry1 and Spry2. Overexpression of Spry decreased PLCγ03B3 activity and IP3 and DAG production, whereas Spry-deficient cells yielded more IP3. Spry overexpression inhibited T-cell receptor signaling and Spry1 null T-cells hyperprolif