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pro vyhledávání: '"Charles Schaub"'
Autor:
MATTHEW DICKERSON, PRASANNA DADI, KAROLINA E. ZABORSKA, ARYA Y. NAKHE, CHARLES SCHAUB, JORDYN DOBSON, NICOLE M. WRIGHT, JOSHUA LYNCH, CLAIRE SCOTT, DAVID JACOBSON
Publikováno v:
Diabetes. 71
Gi/o protein-coupled receptor (Gi/o-GPCR) inhibition of pancreatic β-cell Ca2+ entry limits islet insulin secretion; however, the mechanism has not been identified. As Gi/o-GPCRs activate G protein-gated inwardly rectifying K+ (GIRK) channels, we ex
Autor:
Charles Schaub, Russell Thomson, Joseph Verdi, Jayne Raper, Nada Terra, Penny Lee, Gina Limon
Publikováno v:
J Biol Chem
The human innate immunity factor apolipoprotein L-I (APOL1) protects against infection by several protozoan parasites, including Trypanosoma brucei brucei. Endocytosis and acidification of high-density lipoprotein–associated APOL1 in trypanosome en
Autor:
Joseph A Giovinazzo, Charles Schaub, Russell Thomson, Penny Lee, Nada Terra, Alisha Racho-Jansen, Jayne Raper
Publikováno v:
The Journal of Biological Chemistry
Apolipoprotein L-I (APOL1) is a channel-forming effector of innate immunity. The common human APOL1 variant G0 provides protection against infection with certain Trypanosoma and Leishmania parasite species, but it cannot protect against the trypanoso
Publikováno v:
Methods in molecular biology (Clifton, N.J.). 2116
Interest in trypanosome lytic factors (TLFs) and apolipoprotein L1, the ion channel-forming protein component of TLFs, has increased tenfold since 2010. This is due to the association of African variants of APOL1 with kidney disease such that interes
Publikováno v:
Methods in Molecular Biology ISBN: 9781071602935
Interest in trypanosome lytic factors (TLFs) and apolipoprotein L1, the ion channel-forming protein component of TLFs, has increased tenfold since 2010. This is due to the association of African variants of APOL1 with kidney disease such that interes
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::3b1d9f65890e5ba564a2824b48c56852
https://doi.org/10.1007/978-1-0716-0294-2_28
https://doi.org/10.1007/978-1-0716-0294-2_28
Conference
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