Zobrazeno 1 - 10
of 18
pro vyhledávání: '"Charles F. Pilati"'
Autor:
Michael B. Maron, Walter I. Horne, J. Gary Meszaros, Hans G. Folkesson, Vahagn Ohanyan, Charles F. Pilati, Shyny Koshy, Daniel J. Luther, Jon M. Walro, Tianbo Li
Publikováno v:
American Journal of Physiology-Lung Cellular and Molecular Physiology. 297:L487-L495
The alveolar epithelium plays a critical role in resolving pulmonary edema. We thus hypothesized that its function might be upregulated in rats with heart failure, a condition that severely challenges the lung's ability to maintain fluid balance. Hea
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7c5b83debaa7e9b9e989b7fbed219066
https://doi.org/10.1152/ajplung.90629.2008
https://doi.org/10.1152/ajplung.90629.2008
Publikováno v:
Neurotoxicology and Teratology. 24:267-273
Methamphetamine (MA)-related deaths and nigrostriatal dopaminergic (NSDA) neurotoxicity are greater in males. The exact basis for this gender difference is not known, but data, which show that estrogen (E) can function as a protectant of both the car
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::c3d2034ef549ec04a00cd7932e0dd2e8
https://doi.org/10.1016/s0892-0362(02)00187-3
https://doi.org/10.1016/s0892-0362(02)00187-3
Publikováno v:
Canadian Journal of Physiology and Pharmacology. 77:806-812
Experiments were undertaken to determine whether angiotensin (Ang) II concentration increases during massive sympathetic nervous system (SNS) activation and whether such an increase plays a role in the pathogenesis of SNS-induced left ventricular (LV
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::b73ff4c486f5341592b0eb32461db490
https://doi.org/10.1139/y99-070
https://doi.org/10.1139/y99-070
Publikováno v:
Journal of Applied Physiology. 78:1642-1650
This study was undertaken to determine whether endothelium-derived relaxing factor (EDRF) modulates the pulmonary and systemic hemodynamic responses to massive sympathetic nervous system (SNS) activation and, in so doing, also modulates the degree of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1da67464d5d10560cc7319c4245dcbc9
https://doi.org/10.1152/jappl.1995.78.5.1642
https://doi.org/10.1152/jappl.1995.78.5.1642
Publikováno v:
Canadian Journal of Physiology and Pharmacology. 72:693-700
Intense activation of the sympathetic nervous system (SNS) decreases the contractile state of the rabbit left ventricle (LV). In this study, we determined the time course of LV dysfunction after massive central activation of the SNS in dogs. Veratrin
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c27396ab558cdd95cf61297bc34cf399
https://doi.org/10.1139/y94-098
https://doi.org/10.1139/y94-098
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 263:H784-H791
We sought to determine whether catecholamines are responsible for the depressed left ventricular (LV) function that follows massive sympathetic nervous system (SNS) activation and whether the additional myocardial energy demands of SNS-induced hypert
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::84fb0bdc9d8fee1f75a76c5c96d5b2e1
https://doi.org/10.1152/ajpheart.1992.263.3.h784
https://doi.org/10.1152/ajpheart.1992.263.3.h784
Publikováno v:
Journal of Applied Physiology. 73:50-58
We evaluated the ability of the canine in situ left lower lobe (LLL) vasculature to sieve endogenous plasma proteins of various molecular radii (34–124 A) after LLL arterial pressure had been transiently elevated to 23.8 +/- 0.9 (control group, n =
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7ca8128a4b0cc733e85d35eb0a29812c
https://doi.org/10.1152/jappl.1992.73.1.50
https://doi.org/10.1152/jappl.1992.73.1.50
Autor:
Kathleen J. Doane, Charles F Pilati, Jennifer E. Naugle, John Koshy, J. Gary Meszaros, Erik R. Olson, Donald J. Costic, Cheryl M. Hodnichak
Publikováno v:
The FASEB Journal. 20
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::8751a75d6f1d93cf83bedf188024c4dd
https://doi.org/10.1096/fasebj.20.5.a1464-b
https://doi.org/10.1096/fasebj.20.5.a1464-b
Autor:
Michael B. Maron, Walter I. Horne, Hans G. Folkesson, Kathleen J. Doane, Xiaojin Zhang, Jennifer E. Naugle, Sharon E. Mase, J. Gary Meszaros, Charles F. Pilati, Erik R. Olson
Publikováno v:
American journal of physiology. Heart and circulatory physiology. 290(1)
Cardiac fibroblast (CF) proliferation and differentiation into hypersecretory myofibroblasts can lead to excessive extracellular matrix (ECM) production and cardiac fibrosis. In turn, the ECM produced can potentially activate CFs via distinct feedbac
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::45db119a2d6013c24ed8b4ba5de1d151
https://pubmed.ncbi.nlm.nih.gov/16143656
https://pubmed.ncbi.nlm.nih.gov/16143656
Autor:
Jennifer M. Smith, Charles F. Pilati
Publikováno v:
Experimental biology and medicine (Maywood, N.J.). 227(2)
Our previous work Indicates that myocardial Ischemia could be the mechanism responsible for the left ventricular (LV) dysfunction that frequently develops after massive sympathetic nervous system (SNS) activation. In this study, coronary blood flow (
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d90b34fc230a94085c24e566749b49c8
https://pubmed.ncbi.nlm.nih.gov/11815676
https://pubmed.ncbi.nlm.nih.gov/11815676