Zobrazeno 1 - 10
of 65
pro vyhledávání: '"Changxin Yin"'
Autor:
Jie Luo, Xin Du, Jin Lou, Jianwei Wu, Liping Ma, Jixian Huang, Liangtuo Wang, Chuanqing Tu, Zelin Liu, Liya Chen, Yaxian Tan, Dongmei Luo, Hanyin Liang, Changxin Yin, Rui Cao, Xuan Zhou, Qifa Liu, Xiaoli Liu, Na Xu
Publikováno v:
eJHaem, Vol 3, Iss 4, Pp 1220-1230 (2022)
Abstract Background: Long‐term treatment‐free remission (TFR) represents a new goal for chronic myeloid leukemia (CML). Optimizing dose of tyrosine kinase inhibitors (TKIs) in the CML treatment maybe a new challenge to maintain effective and impr
Externí odkaz:
https://doaj.org/article/ab4d04903c7a449d851b637ccf2a11cc
Autor:
Dan Xu, Yishan Chen, Ying Yang, Zhao Yin, Changfen Huang, Qiang Wang, Ling Jiang, Xuejie Jiang, Changxin Yin, Qifa Liu, Guopan Yu
Publikováno v:
Journal of Translational Medicine, Vol 20, Iss 1, Pp 1-12 (2022)
Abstract Background Autophagy plays a critical role in drug resistance in acute myeloid leukemia (AML), including the subtype with FLT3-ITD mutation. Yet how autophagy is activated and mediates resistance to FLT3 inhibitors in FLT3-ITD-positive AML r
Externí odkaz:
https://doaj.org/article/9c2e487cef8844008f8dc2bccf1f3379
Autor:
Xuejie Jiang, Ling Jiang, Jiaying Cheng, Fang Chen, Jinle Ni, Changxin Yin, Qiang Wang, Zhixiang Wang, Dan Fang, Zhengshan Yi, Guopan Yu, Qingxiu Zhong, Bing Z. Carter, Fanyi Meng
Publikováno v:
Journal of Translational Medicine, Vol 19, Iss 1, Pp 1-12 (2021)
Abstract Background Epigenetic dysregulation plays important roles in leukemogenesis and the progression of acute myeloid leukemia (AML). Histone acetyltransferases (HATs) and histone deacetylases (HDACs) reciprocally regulate the acetylation and dea
Externí odkaz:
https://doaj.org/article/e662629846894083a15f14a73bd9ccba
Autor:
Yiming Tian, Zoufang Huang, Zhixiang Wang, Changxin Yin, Lanlan Zhou, Lingxiu Zhang, Kaikai Huang, Hongsheng Zhou, Xuejie Jiang, Jinming Li, Libin Liao, Mo Yang, Fanyi Meng
Publikováno v:
PLoS ONE, Vol 9, Iss 1, p e84150 (2014)
Numerous factors impact on the prognosis of acute myeloid leukemia (AML), among which molecular genetic abnormalities are developed increasingly, however, accurate prediction for newly diagnosed AML patients remains unsatisfied. For further improving
Externí odkaz:
https://doaj.org/article/6ef8c19092c240e597b80ebbcde457bb
Autor:
Meiqing Wu, Fen Huang, Xinmiao Jiang, Zhiping Fan, Hongsheng Zhou, Can Liu, Qianli Jiang, Yu Zhang, Ke Zhao, Li Xuan, Xiao Zhai, Fuhua Zhang, Changxin Yin, Jing Sun, Ru Feng, Qifa Liu
Publikováno v:
PLoS ONE, Vol 8, Iss 10, p e77805 (2013)
Herpesvirus infections of the central nervous system (CNS) are associated with encephalitis/myelitis and lymphoproliferative diseases in immunocompromised individuals. As of now, data of herpesvirus-associated CNS diseases in transplant recipients is
Externí odkaz:
https://doaj.org/article/8197315d2ccf456ca4f7d1b2aa5a5193
Publikováno v:
Leukemia & Lymphoma. 64:856-864
Publikováno v:
Solid Earth Discussions; 10/24/2023, p1-17, 17p
Autor:
Guopan Yu, Weixiang Lu, Xiaofan Chen, Yanlin Li, Jiaxin Long, Zhongxin Zheng, Changxin Yin, Dan Xu
Publikováno v:
Leukemialymphoma. 63(11)
This study was to explore the role of NK cell subsets and gene expression in maintaining TFR status. We identified six types of NK cells in the PBMCs over both groups (healthy controls and patients with TFR). Gene Oncology analysis showed that up reg
Autor:
Jie Luo, Juan Wang, Liang Liu, Xuan Zhou, Lingling Zhou, Jiayue Qin, Changxin Yin, Xiaoli Liu, Na Xu, Jixian Huang, Yaxian Tan, Zhimin Li, Waner Wu
Publikováno v:
OncoTargets and therapy
Waner Wu,1,* Na Xu,1,* Xuan Zhou,1 Liang Liu,1 Yaxian Tan,1 Jie Luo,1 Jixian Huang,2 Jiayue Qin,3 Juan Wang,3 Zhimin Li,3 Changxin Yin,1 Lingling Zhou,1 Xiaoli Liu1 1Department of Hematology, Nanfang Hospital, Southern Medical University, Guangzhou 5
Autor:
Xuejie Jiang, Changfen Huang, Qiang Wang, Yishan Chen, Zhao Yin, Guopan Yu, Ying Yang, Qifa Liu, Dan Xu, Changxin Yin, Ling Jiang
Background: Autophagy plays a critical role in drug resistance in acute myeloid leukemia (AML), including the subtype with FLT3-ITD mutation. Yet how autophagy is activated and mediates resistance to FLT3 inhibitors in FLT3-ITD-positive AML remains u
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::9085e67f28a3673949a08130b4ea75c0
https://doi.org/10.21203/rs.3.rs-676343/v1
https://doi.org/10.21203/rs.3.rs-676343/v1