Zobrazeno 1 - 10
of 13
pro vyhledávání: '"Chanel Keoni"'
Publikováno v:
Neoplasia: An International Journal for Oncology Research, Vol 21, Iss 1, Pp 52-60 (2019)
KIAA1549-BRAF is the most frequently identified genetic mutation in sporadic pilocytic astrocytoma (PA), creating a fusion BRAF (f-BRAF) protein with increased BRAF activity. Fusion-BRAF-expressing neural stem cells (NSCs) exhibit increased cell grow
Externí odkaz:
https://doaj.org/article/bc5b33cd4e4c4bc8802a51ce728cea6a
Autor:
Ran Chen, Chanel Keoni, Christopher A. Waker, Robert M. Lober, Yi-Hsien Chen, David H. Gutmann
Publikováno v:
Neoplasia: An International Journal for Oncology Research, Vol 22, Iss 3, Pp e1- (2020)
Externí odkaz:
https://doaj.org/article/1bff6e2eb1f14956bda989cc7c1172f4
Autor:
Renee E. Albers, Sarah Min, Bryony V. Natale, Melissa R. Kaufman, Clintoria R. Williams, Tom Brown, Kashmira Kulkarni-Datar, Chanel Keoni, David R. C. Natale
Publikováno v:
Scientific Reports, Vol 9, Iss 1, Pp 1-13 (2019)
Scientific Reports
Scientific Reports
The placenta is an essential organ that is formed during pregnancy and its proper development is critical for embryonic survival. While several animal models have been shown to exhibit some of the pathological effects present in human preeclampsia, t
Autor:
Robert M. Lober, Ran Chen, David H. Gutmann, Chanel Keoni, Christopher A. Waker, Yi Hsien Chen
Publikováno v:
Neoplasia: An International Journal for Oncology Research, Vol 21, Iss 1, Pp 52-60 (2019)
Neoplasia (New York, N.Y.)
Neoplasia (New York, N.Y.)
KIAA1549-BRAF is the most frequently identified genetic mutation in sporadic pilocytic astrocytoma (PA), creating a fusion BRAF (f-BRAF) protein with increased BRAF activity. Fusion-BRAF-expressing neural stem cells (NSCs) exhibit increased cell grow
Autor:
Chanel Keoni, Tom Brown
Publikováno v:
Journal of Cell Death, Vol 2015, Iss 8, Pp 1-7 (2015)
Journal of Cell Death
Japanese Clinical Medicine, Vol 8 (2015)
Journal of Cell Death
Japanese Clinical Medicine, Vol 8 (2015)
Apoptosis is physiological cell death required for the cellular maintenance of homeostasis, and caspases play a major role in the execution of this process. Numerous disorders occur when levels of apoptosis within an organism are excessive, and sever
Autor:
Melissa R. Kaufman, Christopher A. Waker, Renee E. Albers, Tom Brown, Sarah Min, Michael Bottomley, David R. C. Natale, Chanel Keoni
Publikováno v:
Theriogenology. 107
The placenta is a complex and essential organ composed largely of fetal-derived cells, including several different trophoblast subtypes that work in unison to support nutrient transport to the fetus during pregnancy. Abnormal placental development ca
Publikováno v:
Neuro-Oncology. 20:i62-i63
Diffuse intrinsic pontine glioma (DIPG) are incurable tumors and the leading cause of pediatric brain tumor deaths. They exhibit low blood perfusion and regions of necrosis, indicative of a low-oxygen environment that supports activation of hypoxia-i
Autor:
Benjamin Southerland, Kashmira Kulkarni-Datar, Chanel Keoni, Rebecca Bricker, William C. Grunwald, Jr., Daniel M. Ketcha, Eugene Hern, David R. Cool, Thomas L. Brown
Publikováno v:
Journal of Cell Death, Vol 2010, Iss 3, Pp 33-40 (2010)
The broad-spectrum apoptosis (caspase) inhibitor, Q-VD-OPh, has been shown to have no side effects and is effective at a much lower concentration than other FMK-type caspase inhibitors. However, an appropriate negative control to use with this inhibi
Autor:
Tom Brown, David R. C. Natale, Bryony V. Natale, Chanel Keoni, Melissa R. Kaufman, Guarav Kaushik, Renee E. Albers
Publikováno v:
Placenta. 45:78-79
Autor:
Melissa R. Kaufman, Chanel Keoni, Tom Brown, Kashmira Kulkarni-Datar, Renee E. Albers, David R. C. Natale
The placenta is a unique and highly complex organ that develops only during pregnancy and is essential for growth and survival of the developing fetus. The placenta provides the vital exchange of gases and wastes, the necessary nutrients for fetal de
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a7c13b6f4e49dd9e2253157c651c2705
https://europepmc.org/articles/PMC4167466/
https://europepmc.org/articles/PMC4167466/