Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Chandra Khantwal"'
Autor:
Anghesom Ghebremedhin, Ahmad Bin Salam, Benjamin Adu-Addai, Steve Noonan, Richard Stratton, Md Shakir Uddin Ahmed, Chandra Khantwal, George R. Martin, Huixian Lin, Chris Andrews, Balasubramanyam Karanam, Udo Rudloff, Henry Lopez, Jesse Jaynes, Clayton Yates
Publikováno v:
Cells, Vol 12, Iss 9, p 1254 (2023)
Activated M2-polarized macrophages are drivers of pulmonary fibrosis in several clinical scenarios, including Idiopathic Pulmonary Fibrosis (IPF). In this study, we investigated the effects of targeting the CD206 receptor in M2-like macrophages with
Externí odkaz:
https://doaj.org/article/634b487e00244c5a87f649e520e2e17c
Autor:
Greg. A. Timblin, Kevin. M. Tharp, Johanna ten Hoeve, Ilayda Baydemir, Chandra Khantwal, Joshua N. Farahzad, Jorge Domínguez-Andrés, Russell E. Vance, Valerie M. Weaver
The magnitude and duration of Toll-like receptor (TLR)-dependent macrophage proinflammatory responses are tightly regulated. Chronic TLR signaling drives tolerance, an immunosuppressed state of innate immune memory. Understanding of this regulation i
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::3880129f358f992fcd59e7ee9683e08f
https://doi.org/10.1101/2022.08.30.505732
https://doi.org/10.1101/2022.08.30.505732
Autor:
Jesse M. Jaynes, George R. Martin, Chris Andrews, Anghesom Ghebremedhin, Hui-Xian Lin, Benjamin Adu-Addai, Steve Noonan, Ahmad Bin Salam, Shakir Uddin Ahmed, Richard Stratton, Henry Lopez, Udo Rudloff, Balasubramanyam Karanam, Chandra Khantwal, Clayton Yates
Publikováno v:
bioRxiv
Activated M2 polarized macrophages are drivers of pulmonary fibrosis in several clinical scenarios such as Acute Respiratory Disease Syndrome (ARDS) and Idiopathic Pulmonary Fibrosis (IPF), through the production of inflammatory and fibrosis-inducing
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::63abba64d7670ebb3fbb40ccf165a761
https://doi.org/10.1101/2020.07.27.218115
https://doi.org/10.1101/2020.07.27.218115