IFN-gamma signaling in the central nervous system controls the course of experimental autoimmune encephalomyelitis independently of the localization and composition of inflammatory foci

Autor: Athena M. Soulika, David E Pleasure, Sarah Chanamara, Eunyoung Lee

Publikováno v: Lee, Eunyoung; Chanamara, Sarah; Pleasure, David; & Soulika, Athena M. (2012). IFN-gamma signaling in the central nervous system controls the course of experimental autoimmune encephalomyelitis independently of the localization and composition of inflammatory foci. Journal of Neuroinflammation, 9(1), 7. doi: http://dx.doi.org/10.1186/1742-2094-9-7. Retrieved from: http://www.escholarship.org/uc/item/8pn8r1sv
Journal of Neuroinflammation
Journal of Neuroinflammation, Vol 9, Iss 1, p 7 (2012)

Background Murine experimental autoimmune encephalomyelitis (EAE), a model for multiple sclerosis, presents typically as ascending paralysis. However, in mice in which interferon-gamma (IFNγ) signaling is disrupted by genetic deletion, limb paralysi

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0605dce7e4d1a17dc296eb42b94d463b
https://doi.org/10.1186/1742-2094-9-7