Zobrazeno 1 - 8 of 8 pro vyhledávání: '"Cezary Tręda"'
1
Akademický článek
A way to understand idiopathic senescence and apoptosis in primary glioblastoma cells – possible approaches to circumvent these phenomena
Autor: Karolina Janik, Cezary Treda, Aneta Wlodarczyk, Joanna Peciak, Kamila Rosiak, Jolanta Zieba, Dagmara Grot, Adrianna Rutkowska, Roza Pawlowska, Waldemar Och, Piotr Rieske, Ewelina Stoczynska-Fidelus
Publikováno v: BMC Cancer, Vol 19, Iss 1, Pp 1-16 (2019)
Abstract Background Glioblastoma (GB) is considered one of the most lethal tumors. Extensive research at the molecular level may enable to gain more profound insight into its biology and thus, facilitate development and testing of new therapeutic app
Externí odkaz: https://doaj.org/article/e41314fc5a5142cb9707b47026fbd6e9
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2
Akademický článek
EGFR Activation Leads to Cell Death Independent of PI3K/AKT/mTOR in an AD293 Cell Line.
Autor: Cezary Treda, Marta Popeda, Magdalena Ksiazkiewicz, Dawid P Grzela, Maciej P Walczak, Mateusz Banaszczyk, Joanna Peciak, Ewelina Stoczynska-Fidelus, Piotr Rieske
Publikováno v: PLoS ONE, Vol 11, Iss 5, p e0155230 (2016)
The Epidermal Growth Factor Receptor (EGFR) and its mutations contribute in various ways to tumorigenesis and biology of human cancers. They are associated with tumor proliferation, progression, drug resistance and the process of apoptosis. There are
Externí odkaz: https://doaj.org/article/b73caa7c4ba242c1b4e4d45c7b3290ff
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3
Akademický článek
The failure in the stabilization of glioblastoma-derived cell lines: spontaneous in vitro senescence as the main culprit.
Autor: Ewelina Stoczynska-Fidelus, Sylwester Piaskowski, Michal Bienkowski, Mateusz Banaszczyk, Krystyna Hulas-Bigoszewska, Marta Winiecka-Klimek, Anna Radomiak-Zaluska, Waldemar Och, Maciej Borowiec, Jolanta Zieba, Cezary Treda, Piotr Rieske
Publikováno v: PLoS ONE, Vol 9, Iss 1, p e87136 (2014)
Cell line analysis is an important element of cancer research. Despite the progress in glioblastoma cell culturing, the cells isolated from the majority of specimens cannot be propagated infinitely in vitro. The aim of this study was to identify the
Externí odkaz: https://doaj.org/article/af19becb501d4644989ac74eeb8ba452
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4
Akademický článek
Regulation of CAR transgene expression to design semiautonomous CAR-T
Autor: Paweł Głowacki, Cezary Tręda, Piotr Rieske
Publikováno v: Molecular Therapy: Oncology, Vol 32, Iss 3, Pp 200833- (2024)
Effective transgene expression is critical for genetically engineered cell therapy. Therefore, one of CAR-T cell therapy’s critical areas of interest, both in registered products and next-generation approaches is the expression of transgenes. It tu
Externí odkaz: https://doaj.org/article/35d13350992645cfa17f4fed336c7620
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5
Phenotypical Flexibility of the EGFRvIII-Positive Glioblastoma Cell Line and the Multidirectional Influence of TGFβ and EGF on These Cells-EGFRvIII Appears as a Weak Oncogene
Autor: Aneta Włodarczyk, Cezary Tręda, Adrianna Rutkowska, Dagmara Grot, Weronika Dobrewa, Amelia Kierasińska, Marta Węgierska, Tomasz Wasiak, Tadeusz Strózik, Piotr Rieske, Ewelina Stoczyńska-Fidelus
Publikováno v: International Journal of Molecular Sciences; Volume 23; Issue 20; Pages: 12129
Background: The biological role of EGFRvIII (epidermal growth factor receptor variant three) remains unclear. Methods: Three glioblastoma DK-MG sublines were tested with EGF (epidermal growth factor) and TGFβ (transforming growth factor β). Subline
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::00fb8f5607f87d7265d3dc99a76e10e2
https://pubmed.ncbi.nlm.nih.gov/36292985
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6
Increased EGFRvIII Epitope Accessibility after Tyrosine Kinase Inhibitor Treatment of Glioblastoma Cells Creates More Opportunities for Immunotherapy
Autor: Cezary Tręda, Aneta Włodarczyk, Marcin Pacholczyk, Adrianna Rutkowska, Ewelina Stoczyńska-Fidelus, Amelia Kierasińska, Piotr Rieske
Publikováno v: International Journal of Molecular Sciences
Volume 24
Issue 5
Pages: 4350
The number of glioblastoma (GB) cases is increasing every year, and the currently available therapies remain ineffective. A prospective antigen for GB therapy is EGFRvIII, an EGFR deletion mutant containing a unique epitope that is recognized by the
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::98fc14c4b9f6389751ad990b93f9502c
https://doi.org/10.3390/ijms24054350
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