Modulation of monocytes by bioactive lipid anandamide in multiple sclerosis involves distinct Toll-like receptors

Autor: Chiurchu, V, Leuti, A, Cencioni, Mt, Centonze, D, Albanese, M, De Bardi, M, Battistini, L, Maccarrone, M

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=od______3983::27422d2ad1517015071b7e426bdde96d
http://hdl.handle.net/11697/155844

Studio della risposta T CD1 ristretta ai gangliosidi in malattie demielinizzanti

Autor: Cencioni, Mt

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=od______3667::39a07348a2f6de2d312afb7b115370b0
http://hdl.handle.net/2108/429

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Inflammation Triggers Synaptic Alteration and Degeneration in Experimental Autoimmune Encephalomyelitis

Autor: Gianvito Martino, Valentina De Chiara, Giorgio Bernardi, Silvia Rossi, Alessandra Bergami, Diego Centonze, Giancarlo Comi, Alessandra Musella, Andrea Bergamaschi, Francesca Cavasinni, Marcello D'Amelio, Alessandro Martorana, Roberto Furlan, Luca Muzio, Maria Teresa Cencioni, Adamo Diamantini, Erica Butti, Francesco Cecconi, Luca Battistini, Virve Cavallucci

Publikováno v: The Journal of Neuroscience. 29:3442-3452

Neurodegeneration is the irremediable pathological event occurring during chronic inflammatory diseases of the CNS. Here we show that, in experimental autoimmune encephalomyelitis (EAE), a mouse model of multiple sclerosis, inflammation is capable in

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::05cb36991b96922244249eb7823ed145
https://doi.org/10.1523/jneurosci.5804-08.2009

Identification of a caspase-derived N-terminal tau fragment in cellular and animal Alzheimer's disease models

Autor: Nadia Canu, T.T. Rohn, Antonino Cattaneo, Pietro Calissano, Anna Atlante, Maria Teresa Cencioni, Veronica Corsetti, Antonietta Gentile, G. Amadoro, Simona Capsoni, M.T. Ciotti

Publikováno v: Molecular and cellular neurosciences
38 (2008): 381–392. doi:10.1016/j.mcn.2008.03.011
info:cnr-pdr/source/autori:Corsetti b; G. Amadoro a, b; A. Gentile a; S. Capsoni c; M.T. Ciotti c; M.T. Cencioni e; A. Atlante d; N. Canu a, b; T.T. Rohn f; A. Cattaneo b; P. Calissano a, b/titolo:Identification of a caspase-derived N-terminal tau fragment in cellular and animal Alzheimer's disease models/doi:10.1016%2Fj.mcn.2008.03.011/rivista:Molecular and cellular neurosciences (Print)/anno:2008/pagina_da:381/pagina_a:392/intervallo_pagine:381–392/volume:38
info:cnr-pdr/source/autori:Corsetti V., Amadoro G., Gentile A., Capsoni S.,Ciotti M.T., Cencion M.T.i,Atlante A., Canu N., Rohn T.T.,Cattaneo A., Calissano P./titolo:Identification of a caspase-derived n-terminal tau fragment in cellular and animal alzheimer disease models/doi:10.1016%2Fj.mcn.2008.03.011/rivista:Molecular and cellular neurosciences (Print)/anno:2008/pagina_da:381/pagina_a:392/intervallo_pagine:381–392/volume:38

Biochemical modifications of tau proteins have been proposed to be among the earliest neurobiological changes in Alzheimer's disease (AD) and correlate better with cognitive symptoms than do beta-amyloid plaques. We have recently reported that adenov

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1265ea1fe825ac793db94fe0ad4672a2
http://hdl.handle.net/11392/2349791