Zobrazeno 1 - 10
of 17
pro vyhledávání: '"Cell Adhesion/immunology"'
Autor:
Marius Ringelstein, L. M. Yshii, Ilka Kleffner, Stephen W. Reddel, Brigitte Wildemann, Todd A. Hardy, Markus Schwaninger, Guillaume Martin-Blondel, Nicholas Schwab, Delphine Loussouarn, John Parratt, Marc Pawlitzki, Henrike Plate, Eduardo Beltrán, Catharina C. Gross, Anna R. Tröscher, Tanja Kuhlmann, Tilman Schneider-Hohendorf, Klaus Dornmair, Jan Dörr, Roland S. Liblau, Urvashi Bhatia, Hans Lassmann, Sven G. Meuth, Luisa Klotz, Sebastian Herich, Markus Kraemer, Jan Bauer, Céline Meyer, David-Axel Laplaud, Wolfgang Brück, Romana Höftberger, Michael Barnett, Michael E. Buckland, Andreas Schulte-Mecklenbeck, Heinz Wiendl
Publikováno v:
Nature Communications
Nature Communications, Nature Publishing Group, 2019, 10 (1), pp.5779. ⟨10.1038/s41467-019-13593-5⟩
Nature Communications, 2019, 10, pp.5779. ⟨10.1038/s41467-019-13593-5⟩
Nature Communications, Vol 10, Iss 1, Pp 1-19 (2019)
Nature Communications, Nature Publishing Group, 2019, 10 (1), pp.5779. ⟨10.1038/s41467-019-13593-5⟩
Nature Communications, 2019, 10, pp.5779. ⟨10.1038/s41467-019-13593-5⟩
Nature Communications, Vol 10, Iss 1, Pp 1-19 (2019)
International audience; Neuroinflammation is often associated with blood-brain-barrier dysfunction, which contributes to neurological tissue damage. Here, we reveal the pathophysiology of Susac syndrome (SuS), an enigmatic neuroinflammatory disease w
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ca645613d7be9f13495f97945a31e9dd
https://www.hal.inserm.fr/inserm-02448919/file/s41467-019-13593-5.pdf
https://www.hal.inserm.fr/inserm-02448919/file/s41467-019-13593-5.pdf
Autor:
T. Litzenburger, S. Noll, A. Popp, D. Waldherr, J. Haunschild, Falk Ochsendorf, M. Urban, Wolf-Henning Boehncke
Publikováno v:
British Journal of Dermatology, Vol. 153, No 4 (2005) pp. 758-766
BACKGROUND: Psoriasis is considered as a chronic immune-mediated disease characterized by inflammation and proliferation of the epidermis. OBJECTIVES: Targeting intercellular adhesion molecule 1 (ICAM-1) is an attractive therapeutic option as this mo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e588bf69693d36fc79fc1e146d33c2a8
https://doi.org/10.1111/j.1365-2133.2005.06657.x
https://doi.org/10.1111/j.1365-2133.2005.06657.x
Publikováno v:
Olsen, I, Boysen, P, Kulberg, S, Hope, J, Jungersen, G & Storset, A K 2005, ' Bovine NK cells can produce gamma interferon in response to the secreted mycobacterial proteins ESAT-6 and MPP14 but not in response to MPB70 ', Infection and Immunity, vol. 73, no. 9, pp. 5628-35 . https://doi.org/10.1128/IAI.73.9.5628-5635.2005
Bovine NK cells have recently been characterized and the present study describes the interaction between NK cells, antigen-presenting cells, and secreted mycobacterial proteins. Gamma interferon (IFN-γ) production by NK cells was seen in approximate
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d836a82653b76702376c14e6dfd5aa70
https://doi.org/10.1128/iai.73.9.5628-5635.2005
https://doi.org/10.1128/iai.73.9.5628-5635.2005
Autor:
Pier Luigi Meroni, Jean-Michel Dayer, Carlo Chizzolini, Cinzia Testoni, Maria Orietta Borghi, Roger Rezzonico, Roberto Mallone, Armando Gabrielli, Nicoletta Del Papa, Elena Raschi, Andrea Facchini
Publikováno v:
Arthritis and Rheumatism, Vol. 46, No 6 (2002) pp. 1602-13
Objective Fibroblasts play a major role in the development of systemic sclerosis (SSc), and the occurrence of serum autoantibodies reacting with fibroblast plasma membrane antigens in SSc has been reported. This study was undertaken to investigate wh
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0c8afeb20143febfb4e6ca246e50fc0a
https://doi.org/10.1002/art.10361
https://doi.org/10.1002/art.10361
Autor:
Patrick Y-K Wong, Charles N. Serhan
This volume constitutes. in part. the proceedings of the symposium on'Cell-Cell Interaction and Release of Inflammatory Mediators'organized by Drs. Patrick Y-K Wong and Charles N. Serhan and presented at the FASEB meeting in Washington. D.C. in April
Autor:
Jung Eun Jang, Daigo Hashimoto, Paul S. Frenette, Dachuan Zhang, Andrew Chow, Yuya Kunisaki, Miriam Merad, Daniel Lucas, Christoph Scheiermann
Publikováno v:
Immunity, Vol. 37, No 2 (2012) pp. 290-301
SummaryThe multistep sequence leading to leukocyte migration is thought to be locally regulated at the inflammatory site. Here, we show that broad systemic programs involving long-range signals from the sympathetic nervous system (SNS) delivered by a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0b96310f11bc064dc775f310aa5ab31c
https://archive-ouverte.unige.ch/unige:113710
https://archive-ouverte.unige.ch/unige:113710
Autor:
Soufiane, Ghannam, Jérôme, Pène, Gabriel, Moquet-Torcy, Gabriel, Torcy-Moquet, Christian, Jorgensen, Hans, Yssel
Publikováno v:
Journal of Immunology
Journal of Immunology, Publisher : Baltimore : Williams & Wilkins, c1950-. Latest Publisher : Bethesda, MD : American Association of Immunologists, 2010, 185 (1), pp.302--12. ⟨10.4049/jimmunol.0902007⟩
Journal of Immunology, Publisher : Baltimore : Williams & Wilkins, c1950-. Latest Publisher : Bethesda, MD : American Association of Immunologists, 2010, 185 (1), pp.302--12. ⟨10.4049/jimmunol.0902007⟩
Mesenchymal stem cells (MSCs) exert immunomodulatory properties via the inhibition of T cell activation and proliferation. Because of the deleterious role of Th17 cells in the pathogenesis of inflammatory disease, we investigated whether proinflammat
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2a8fb7b71915f1c3c4179188d2f82630
https://hal.archives-ouvertes.fr/hal-02193434
https://hal.archives-ouvertes.fr/hal-02193434
Autor:
F.E.J. Coenjaerts, Brenda R. Kwak, Marc Chanson, Marc Bacchetta, Lucile Miquerol, Isabelle Roth, Zhihong Yang, T. A. B. van Veen, Urban Deutsch, Christos Chadjichristos, Habo J. Jongsma, M.J.A. van Kempen, M. Z. Richani Sarieddine, Tecla Dudez, K.E.L. Scheckenbach, Hema Viswambharan, Bernard Foglia, C. de Wit
Publikováno v:
Circulation
Circulation, 2010, 121 (1), pp.123-131. ⟨10.1161/CIRCULATIONAHA.109.867176⟩
Circulation, American Heart Association, 2010, 121 (1), pp.123-131
Circulation, Vol. 121, No 1 (2010) pp. 123-131
Circulation, 2010, 121 (1), pp.123-131. ⟨10.1161/CIRCULATIONAHA.109.867176⟩
Circulation, American Heart Association, 2010, 121 (1), pp.123-131
Circulation, Vol. 121, No 1 (2010) pp. 123-131
Background— Endothelial dysfunction is the initiating event of atherosclerosis. The expression of connexin40 (Cx40), an endothelial gap junction protein, is decreased during atherogenesis. In the present report, we sought to determine whether Cx40
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7bc33fba1a1aaaf799eda2172df5013a
https://hal.science/hal-00566980
https://hal.science/hal-00566980
Autor:
Katja Hardt, Maurizio Podda, Heinfried H. Radeke, Roxana Bistrian, Sandra Diehl, Michael P. Schön, Max Hatting, Sentot Santoso, Reinhard Henschler, Josef Pfeilschifter, Roland Kaufmann, Wolf-Henning Boehncke, Ralf Ludwig
Publikováno v:
Immunology, Vol. 128, No 2 (2009) pp. 196-205
Junctional adhesion molecule-A (JAM-A), JAM-B and JAM-C have been implicated in leucocyte transmigration. As JAM-B binds to very late activation antigen (VLA)-4, a leucocyte integrin that contributes to rolling and firm adhesion of lymphocytes to end
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::cfbe915aea575bb848288f0c46f3b666
https://pubmed.ncbi.nlm.nih.gov/19740376
https://pubmed.ncbi.nlm.nih.gov/19740376
Publikováno v:
Xenotransplantation, Vol. 16, No 1 (2009) pp. 19-26
BACKGROUND: Human natural killer (NK) cell-mediated cytotoxicity represents a hurdle in pig-to-human xenotransplantation. It was previously reported that the expression of human major histocompatibility complex class I molecules, including HLA-B27, -
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c1d3b221b12f01882c82385c8a8d2510
https://pubmed.ncbi.nlm.nih.gov/19243557
https://pubmed.ncbi.nlm.nih.gov/19243557