Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Celia Martín-Cuevas"'
Autor:
Celia Martín-Cuevas, Víctor Ramos-Herrero, María Molina-Fossati, Benedicto Crespo-Facorro, Ana Sánchez-Hidalgo
Publikováno v:
IBRO Neuroscience Reports, Vol 15, Iss , Pp S606- (2023)
Externí odkaz:
https://doaj.org/article/d17e1e38510540c3a642d5efb41d576a
Autor:
Ana Sánchez-Hidalgo, Celia Martín-Cuevas, Álvaro Flores-Martínez, Víctor Ramos-Herrero, Isabel Cortines-Serrano, Benedicto Crespo-Facorro
Publikováno v:
IBRO Neuroscience Reports, Vol 15, Iss , Pp S640- (2023)
Externí odkaz:
https://doaj.org/article/7592212c7be648f68bd9690def2c8124
Autor:
Francisco Arias-Aragón, Enriqueta Tristán-Clavijo, Irene Martínez-Gallego, Estefanía Robles-Lanuza, Heriberto Coatl-Cuaya, Celia Martín-Cuevas, Ana C. Sánchez-Hidalgo, Antonio Rodríguez-Moreno, Amalia Martinez-Mir, Francisco G. Scholl
Publikováno v:
iScience, Vol 26, Iss 6, Pp 106868- (2023)
Summary: Alzheimer’s disease (AD) is characterized by memory impairments and age-dependent synapse loss. Experimental and clinical studies have shown decreased expression of the glutamatergic protein Neuroligin-1 (Nlgn1) in AD. However, the consequ
Externí odkaz:
https://doaj.org/article/5233b3de08fc4b01a50dadad8bc24dfc
Autor:
Ana C. Sánchez-Hidalgo, Celia Martín-Cuevas, Benedicto Crespo-Facorro, Nathalia Garrido-Torres
Publikováno v:
Frontiers in Neuroanatomy, Vol 16 (2022)
Reelin is an extracellular matrix glycoprotein reduced in brain regions (the prefrontal cortex and the hippocampus) of patients with schizophrenia. There are diverse rodent models of schizophrenia that mimic patient symptoms based on various causal t
Externí odkaz:
https://doaj.org/article/46ebc9041aee41279b0545643b2257c3
Autor:
Francisco Arias-Aragón, Francisco G. Scholl, M Teresa Romero-Barragán, Amalia Martinez-Mir, Celia Martín-Cuevas, Ana C. Sánchez-Hidalgo, José M. Delgado-García
Publikováno v:
idUS. Depósito de Investigación de la Universidad de Sevilla
instname
Digital.CSIC. Repositorio Institucional del CSIC
instname
Digital.CSIC. Repositorio Institucional del CSIC
Presenilins (PS) form the active subunit of the gamma-secretase complex, which mediates the proteolytic clearance of a broad variety of type-I plasma membrane proteins. Loss-of-function mutations in PSEN1/2 genes are the leading cause of familial Alz
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3316d02386a8949c29d1136f3321cd66
