Zobrazeno 1 - 10
of 38
pro vyhledávání: '"Cecilia H. A. Gouveia"'
Autor:
Clarissa R. Zaitune, Tatiana L. Fonseca, Luciane P. Capelo, Fatima R. Freitas, Eduardo H. Beber, José M. Dora, Charles C. Wang, Manuela Miranda-Rodrigues, Keico O. Nonaka, Ana L. Maia, Cecilia H. A. Gouveia
Publikováno v:
Frontiers in Endocrinology, Vol 10 (2019)
C3H/HeJ (C3H) mice are deficient of type I deiodinase (D1), an enzyme that activates thyroid hormone (TH), converting thyroxine (T4) to triiodothyronine (T3). Nevertheless, C3H mice present normal serum T3 and a gross euthyroid phenotype. To investig
Externí odkaz:
https://doaj.org/article/df5008aa1e0c46a7bd494380dca4c590
Autor:
Gisele M. Martins, Marília B. C. G. Teixeira, Marcos V. Silva, Bianca Neofiti-Papi, Manuela Miranda-Rodrigues, Patricia C. Brum, Cecilia H. A. Gouveia
Publikováno v:
Frontiers in Endocrinology, Vol 9 (2018)
Evidence shows that sympathetic nervous system (SNS) activation inhibits bone formation and activates bone resorption leading to bone loss. Because thyroid hormone (TH) interacts with the SNS to control several physiological processes, we raised the
Externí odkaz:
https://doaj.org/article/173b222035714d90b5100f0857fe0b93
Autor:
Vanda Jorgetti, Nathalia Juliana Nardelli Gonçalves, Cecilia H. A. Gouveia, Rudá Prestes e Albuquerque, Manuela Miranda-Rodrigues, Bianca Neofiti-Papi, Julio C.B. Ferreira, Patricia Chacur Brum
Publikováno v:
Repositório Institucional da USP (Biblioteca Digital da Produção Intelectual)
Universidade de São Paulo (USP)
instacron:USP
Universidade de São Paulo (USP)
instacron:USP
Background: Thyrotoxicosis increases bone turnover, resulting in net bone loss. Sympathetic nervous system (SNS) activation, via β2-adrenoceptor (β2-AR) signaling, also has osteopenic effects. Because thyroid hormones (TH) interact with the SNS to
Autor:
M. I. Sousa, Amélia Fonseca, João Pedro Silva, Cecilia H. A. Gouveia, D. Aragão, J. P. Fernandas, A. Pinheiro, António Câmara
Publikováno v:
Spatial Multimedia and Virtual Reality
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::acacbf4f4fb20d039427a898e1365630
https://doi.org/10.1201/9781003209614-8
https://doi.org/10.1201/9781003209614-8
Autor:
Dieter P. Reinhardt, Lygia da Veiga Pereira, Isabela Gerdes Gyuricza, Elisa Ito Kawahara, Renan B. Lemes, Cecilia H. A. Gouveia, Manuela Miranda-Rodrigues, Marilia Bianca Cruz Grecco Teixeira, Rodrigo Barbosa de Souza, Gustavo Fernandes, Bianca Neofiti-Papi, Luis Ernesto Farinha-Arcieri
Publikováno v:
Repositório Institucional da USP (Biblioteca Digital da Produção Intelectual)
Universidade de São Paulo (USP)
instacron:USP
Universidade de São Paulo (USP)
instacron:USP
Marfan syndrome (MFS) is an autosomal dominant disease affecting cardiovascular, ocular and skeletal systems. It is caused by mutations in the fibrillin-1 (FBN1) gene, leading to structural defects of connective tissue and increased activation of TGF
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::907e7097c5f475c580101f4c5e5eace5
Publikováno v:
Encyclopedia of Bone Biology ISBN: 9780128140826
Thyroid hormone (TH) has key roles in bone development and metabolism. There is evidence that TH regulates skeletal development and bone metabolism via direct and indirect mechanisms. The presence of all the factors necessary for local actions of TH
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::1e3115b9ff2ee16a9a7b675dc9352836
https://doi.org/10.1016/b978-0-12-801238-3.11227-9
https://doi.org/10.1016/b978-0-12-801238-3.11227-9
Publikováno v:
Vitamins and hormones. 106
Thyroid hormone (TH) is essential for skeletal development from the late fetal life to the onset of puberty. During this large window of actions, TH has key roles in endochondral and intramembranous ossifications and in the longitudinal bone growth.
Autor:
Manuela Miranda-Rodrigues, Arnaldo Amado Fereira Neto, Eduardo Angeli Malavolta, Leonardo Cavinatto, Rames Mattar Júnior, Luiza de Campos Moreira da Silva, Cecilia H. A. Gouveia, Cesar de Cesar Netto, César Augusto Martins Pereira
Publikováno v:
Repositório Institucional da USP (Biblioteca Digital da Produção Intelectual)
Universidade de São Paulo (USP)
instacron:USP
Universidade de São Paulo (USP)
instacron:USP
Background In the event of a traumatic rotator cuff tear, patients are routinely advised that early surgical intervention produces an optimal repair, despite a lack of direct evidence to support this recommendation. To address this knowledge gap, mas
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::207d9b0349a5a7ef898d9dcd2e9df174
Autor:
Tatiana L, Fonseca, Marilia B C G, Teixeira, Manuela, Miranda-Rodrigues, Manuela, Rodrigues-Miranda, Marcos V, Silva, Gisele M, Martins, Cristiane C, Costa, Danielle Y, Arita, Juliana D, Perez, Dulce E, Casarini, Patricia C, Brum, Cecilia H A, Gouveia
Publikováno v:
American Journal of Physiology-Endocrinology and Metabolism. 307:E408-E418
To investigate whether thyroid hormone (TH) interacts with the sympathetic nervous system (SNS) to modulate bone mass and structure, we studied the effects of daily T3 treatment in a supraphysiological dose for 12 wk on the bone of young adult mice w
Autor:
Ana Carolina Bernardini Moulatlet, Priscilla Morethson, Ambart E. Covarrubias, Keico Okino Nonaka, Marilia Bianca Cruz Grecco Teixeira, Telma Maria Tenório Zorn, Cristiane C. Costa, Eric Hesse, Charles Chenwei Wang, Patricia Chakur Brum, Dulce Elena Casarini, Fatima R. Freitas, Vanda Jorgetti, Ricardo Ferraz de Oliveira, Cecilia H. A. Gouveia, Tatiana L. Fonseca, Eduardo H Beber, Luciane P. Capelo
Publikováno v:
Journal of Bone and Mineral Research. 26:591-603
Evidence demonstrates that sympathetic nervous system (SNS) activation causes osteopenia via β(2)-adrenoceptor (β2-AR) signaling. Here we show that female mice with chronic sympathetic hyperactivity owing to double knockout of adrenoceptors that ne