Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Catherine E. Moffatt"'
Publikováno v:
Cellular Microbiology. 14:577-588
Porphyromonas gingivalis, a host-adapted opportunistic pathogen, produces a serine phosphatase, SerB, known to affect virulence, invasion and persistence within the host cell. SerB induces actin filament rearrangement in epithelial cells, but the mec
Publikováno v:
Molecular Oral Microbiology. 26:365-373
An association between the gram-positive anaerobe Filifactor alocis and periodontal disease has recently emerged; however, possible pathogenic mechanisms have not been investigated. In this study we examined the responses of primary cultures of gingi
Publikováno v:
Infection and Immunity. 79:2632-2637
Porphyromonas gingivalis is a pathogen in severe periodontal disease. Able to exploit an intracellular lifestyle within primary gingival epithelial cells (GECs), a reservoir of P. gingivalis can persist within the gingival epithelia. This process is
Autor:
Catherine E. Moffatt, Sarah E. Whitmore, T A Brown, Dylan Hagerty, Dana T. Graves, Brittany C. Dickinson, Richard J. Lamont
Publikováno v:
Molecular Oral Microbiology. 26:210-220
Primary gingival epithelial cells were cultured in multilayers as a model for the study of interactions with oral bacteria associated with health and periodontal disease. Multilayers maintained at an air-liquid interface in low calcium medium display
Autor:
Richard J. Lamont, Indraneel Bhattacharyya, Bilal Yehia, Christie Eastman, Lakshmyya Kesavalu, Raj K. Verma, Mercedes F. Rivera, Catherine E. Moffatt, Brian W. Bainbridge
Publikováno v:
Infection and Immunity. 78:4560-4569
Porphyromonas gingivalis secretes a serine phosphatase enzyme, SerB, upon contact with gingival epithelial cells in vitro . The SerB protein plays a critical role in internalization and survival of the organism in epithelial cells. SerB is also respo
Publikováno v:
Cellular microbiology. 14(4)
Porphyromonas gingivalis, a host-adapted opportunistic pathogen, produces a serine phosphatase, SerB, known to affect virulence, invasion and persistence within the host cell. SerB induces actin filament rearrangement in epithelial cells, but the mec