Zobrazeno 1 - 10
of 20
pro vyhledávání: '"Catherina A. Cuevas"'
Publikováno v:
Hoorn, E J, Gritter, M, Cuevas, C A & Fenton, R A 2020, ' Regulation of the Renal NaCl Cotransporter and Its Role in Potassium Homeostasis ', Physiological Reviews, vol. 100, no. 1, pp. 321-356 . https://doi.org/10.1152/physrev.00044.2018
Physiological Reviews, 100(1), 321-356. American Physiological Society
Physiological Reviews, 100(1), 321-356. American Physiological Society
Daily dietary potassium (K+) intake may be as large as the extracellular K+pool. To avoid acute hyperkalemia, rapid removal of K+from the extracellular space is essential. This is achieved by translocating K+into cells and increasing urinary K+excret
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::cd667d306b56aaf46ffd2d23cf19de28
https://doi.org/10.1152/physrev.00044.2018
https://doi.org/10.1152/physrev.00044.2018
Autor:
Rafael Kramann, Jürgen Floege, Turgay Saritas, Anne Babler, Sylvie Menzel, Jitske Jansen, Christoph Kuppe, Andrew S Kerker, Catherina A. Cuevas, Christian Bleilevens, Xiao-Tong Su, Katharina C Reimer, David H. Ellison, Lucas L. Falke, Peter Boor, Anne Rix, James A. McCormick, Lu Chen
Publikováno v:
Nephrology Dialysis Transplantation. 36
Background and Aims Chronic hypokalemia causes kidney fibrosis with cystic lesions and arterial hypertension. In contrast, potassium-rich diet lowers blood pressure. The acute effects of hypo- and hyperkalemia on heart and kidney are not well underst
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::b68d83e3b1da6ebacd443f35096810b6
https://doi.org/10.1093/ndt/gfab090.004
https://doi.org/10.1093/ndt/gfab090.004
Autor:
A.H. Jan Danser, David Severs, Eloísa Rubio-Beltrán, Dominique M Bovée, Jaap A. Joles, Estrellita Uijl, Catherina A. Cuevas, Ewout J. Hoorn, Richard van Veghel, Robert Zietse, Antoinette Maassen van den Brink
Publikováno v:
American Journal of Physiology-Renal Physiology, 320(4), F654-F668. American Physiological Society
Chronic kidney disease contributes to hypertension, but the mechanisms are incompletely understood. To address this, we applied the 5/6th nephrectomy rat model to characterize hypertension and the response to dietary salt and renin-Angiotensin inhibi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d4b0595d60f90a0eeae357db6e3cca29
https://pure.eur.nl/en/publications/0274968f-0996-444d-b3ec-fe857b008a84
https://pure.eur.nl/en/publications/0274968f-0996-444d-b3ec-fe857b008a84
Autor:
Ming Xiao Wang, Xiao Tong Su, Wen-Hui Wang, James A. McCormick, Peng Wu, Zhong Xiuzi Gao, Chao Ling Yang, Dao Hong Lin, Catherina A. Cuevas, David H. Ellison
Publikováno v:
Kidney International. 93:893-902
Kir4.1 in the distal convoluted tubule plays a key role in sensing plasma potassium and in modulating the thiazide-sensitive sodium-chloride cotransporter (NCC). Here we tested whether dietary potassium intake modulates Kir4.1 and whether this is ess
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::baea1829699686a2c5f1c9a3d047095d
https://doi.org/10.1016/j.kint.2017.10.023
https://doi.org/10.1016/j.kint.2017.10.023
Autor:
Alexis A. Gonzalez, Cristian Reyes-Martinez, Minolfa C. Prieto, Alex Gonzalez-Vergara, Leonardo Zamora, Catherina A. Cuevas, Nicolas Salinas-Parra, Stefanny M. Figueroa, Nicole Roldán
Publikováno v:
Clinical and Experimental Pharmacology and Physiology. 44:1134-1144
Recent studies suggested that activation of the PRR upregulates profibrotic markers through reactive oxygen species (ROS) formation; however, the exact mechanisms have not been investigated in CD cells. We hypothesized that activation of the PRR incr
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::318bcecc7faa976b93873c4601f4ac00
https://doi.org/10.1111/1440-1681.12813
https://doi.org/10.1111/1440-1681.12813
Autor:
Andrew S. Terker, Ming Xiao Wang, Xiao Tong Su, David H. Ellison, Catherina A. Cuevas, James A. McCormick, Wen-Hui Wang, Chao Ling Yang, Dao Hong Lin
Publikováno v:
Journal of the American Society of Nephrology. 28:1814-1825
The mammalian distal convoluted tubule (DCT) makes an important contribution to potassium homeostasis by modulating NaCl transport. The thiazide-sensitive Na+/Cl− cotransporter (NCC) is activated by low potassium intake and by hypokalemia. Coupled
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5ed61807eb39eb0b42ea4d60b4fcd5f2
https://doi.org/10.1681/asn.2016090935
https://doi.org/10.1681/asn.2016090935
Autor:
Christoph Kuppe, Marcus J. Moeller, Jeffrey D. Singer, Catherina A. Cuevas, Turgay Saritas, Rafael Kramann, Mohammed Z. Ferdaus, James A. McCormick, Jürgen Floege
Publikováno v:
Scientific Reports
Scientific Reports, 9:4596. Nature Publishing Group
Scientific Reports, Vol 9, Iss 1, Pp 1-14 (2019)
Scientific Reports, 9:4596. Nature Publishing Group
Scientific Reports, Vol 9, Iss 1, Pp 1-14 (2019)
Cullin 3 (CUL3) is part of the ubiquitin proteasomal system and controls several cellular processes critical for normal organ function including the cell cycle, and Keap1/Nrf2 signaling. Kidney tubule-specific Cul3 disruption causes tubulointerstitia
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::56d518651b19367ddac95a621d5f6d55
http://europepmc.org/articles/PMC6418206
http://europepmc.org/articles/PMC6418206
Autor:
Ryan J. Cornelius, Jonathan W. Nelson, Jinge Si, David H. Ellison, Ruggero Pardi, Catherina A. Cuevas, Chao Ling Yang, Brittany D.K. Gratreak
Background The familial hyperkalemic hypertension (FHHt) cullin 3 (CUL3) mutant does not degrade WNK kinases normally, thereby leading to thiazide-sensitive Na-Cl cotransporter (NCC) activation. CUL3 mutant (CUL3 Δ 9) does not bind normally to the C
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4ddba382ec11769690c55f79c485752d
https://jasn.asnjournals.org/content/jnephrol/29/11/2627.full.pdf
https://jasn.asnjournals.org/content/jnephrol/29/11/2627.full.pdf
Autor:
Carlos Cespedes, Cheril Tapia-Rojas, Catherina A. Cuevas, Carlos P. Vio, Nibaldo C. Inestrosa
Publikováno v:
BioMed Research International, Vol 2016 (2016)
In the article titled “β-Catenin-Dependent Signaling Pathway Contributes to Renal Fibrosis in Hypertensive Rats” [1] the authors request to change bcl-2 figure from Figure 6, since the Western blot corresponding to bcl-2 does not correspond to t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7d3db9287ff8033df74bea43fe061648
https://doaj.org/article/224c4990ffe24825b7012186e60d9e87
https://doaj.org/article/224c4990ffe24825b7012186e60d9e87
Autor:
Catherina A. Cuevas, Patricia A. Navarrete-Gómez, Alexis Parada-Bustamante, Horacio B. Croxatto, Mariana Ríos, Luis A. Velásquez, Pedro A. Orihuela, Manuel Villalón
Publikováno v:
Biology of Reproduction. 77:934-941
Estradiol (E 2 ) accelerates oviductal egg transport through intraoviductal nongenomic pathways in cyclic rats and through genomic pathways in pregnant rats. This shift in pathways, which we have provisionally designated as intracellular path shiftin
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::45a9fbf5a5a0303085c57e087ee472d1
https://doi.org/10.1095/biolreprod.107.061622
https://doi.org/10.1095/biolreprod.107.061622