Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Caterina Allegretta"'
Publikováno v:
Cells, Vol 12, Iss 13, p 1764 (2023)
The deletion of phenylalanine at position 508 (F508del) produces a misfolded CFTR protein that is retained in the ER and degraded. The lack of normal CFTR channel activity is associated with chronic infection and inflammation which are the primary ca
Externí odkaz:
https://doaj.org/article/e065606b5db847c29555e183a70b3565
Autor:
Onofrio Laselva, Maria Laura Criscione, Caterina Allegretta, Sante Di Gioia, Arcangelo Liso, Massimo Conese
Publikováno v:
Frontiers in Molecular Biosciences, Vol 9 (2022)
Cystic Fibrosis (CF) patients are prone to contracting bacterial lung infections with opportunistic pathogens, especially Pseudomonas aeruginosa. Prolonged P. aeruginosa infections have been linked to chronic inflammation in the CF lung, whose hallma
Externí odkaz:
https://doaj.org/article/6847cf4eaf9646f79f44232024cf0ffa
Publikováno v:
International Journal of Molecular Sciences, Vol 23, Iss 7, p 3829 (2022)
Autoimmune demyelinating diseases—including multiple sclerosis, neuromyelitis optica spectrum disorder, anti-myelin oligodendrocyte glycoprotein-associated disease, acute disseminated encephalomyelitis, and glial fibrillary acidic protein (GFAP)-as
Externí odkaz:
https://doaj.org/article/b0b1acac1b774d53891feff08809aa36
Publikováno v:
Cells, Vol 10, Iss 8, p 2132 (2021)
Cystic Fibrosis (CF) is caused by mutations on the CF transmembrane conductance regulator (CFTR) gene and is associated with chronic infection and inflammation. Recently, it has been demonstrated that LPS-induced CFTR dysfunction in airway epithelial
Externí odkaz:
https://doaj.org/article/976e6d12e7264dc6af2d853cb48d8f56
Autor:
Emanuele D’Amico, Aurora Zanghì, Virginia Manuti, Caterina Allegretta, Antonella Amoruso, Gaetano Serviddio, Carlo Avolio
Publikováno v:
Journal of Neurology. 270:1150-1153
Publikováno v:
Cells; Volume 12; Issue 13; Pages: 1764
The deletion of phenylalanine at position 508 (F508del) produces a misfolded CFTR protein that is retained in the ER and degraded. The lack of normal CFTR channel activity is associated with chronic infection and inflammation which are the primary ca
Autor:
EMANUELE D'AMICO, caterina allegretta, Aurora Zanghì, virginia manuti, antonella amoruso, carlo avolio
Background: Recently microRNAs (miRs) have been proposed as possible disease biomarkers in Multiple Sclerosis (MS) field. Among miRs, those secreted through microvesicles (MVs) may contribute to different disease phases. Methods: This is a case-contr
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9a20f88ed24477b986ee5984a0eae60c
https://doi.org/10.21203/rs.3.rs-1493864/v2
https://doi.org/10.21203/rs.3.rs-1493864/v2
Publikováno v:
Cells
Volume 10
Issue 8
Cells, Vol 10, Iss 2132, p 2132 (2021)
Volume 10
Issue 8
Cells, Vol 10, Iss 2132, p 2132 (2021)
Cystic Fibrosis (CF) is caused by mutations on the CF transmembrane conductance regulator (CFTR) gene and is associated with chronic infection and inflammation. Recently, it has been demonstrated that LPS-induced CFTR dysfunction in airway epithelial