Zobrazeno 1 - 10
of 14
pro vyhledávání: '"Catarina Carmo"'
Dissertação de mestrado Genética Molecular
As ataxias espinocerebelosas (SCAs) são doenças neurodegenerativas e autossómicas dominantes causadas por degeneração de neurónios cerebrais. Pacientes SCA são principalmente caracterizados po
As ataxias espinocerebelosas (SCAs) são doenças neurodegenerativas e autossómicas dominantes causadas por degeneração de neurónios cerebrais. Pacientes SCA são principalmente caracterizados po
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od_______307::a3d7838d1438cb97bb87ecaea12d80db
https://hdl.handle.net/1822/85328
https://hdl.handle.net/1822/85328
Publikováno v:
International Journal of Environmental Research and Public Health; Volume 19; Issue 19; Pages: 12923
(1) Background: Breast cancer (BC) shows significant epidemiological differences between Eastern and Western countries. These may arise from socio-cultural factors influencing how healthy young women perceive this condition, their risk of getting can
Structural maintenance of chromosomes (SMC) proteins are critical to maintain mitotic fidelity in all organisms. Over the last decades, acute inactivation of these complexes, together with the analysis of their dynamic binding to mitotic chromatin, h
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::48977fecaaf14bb7c028887d8c7d5325
https://doi.org/10.17504/protocols.io.bnx6mfre
https://doi.org/10.17504/protocols.io.bnx6mfre
Autor:
Flaviano Giorgini, Lígia Fão, Luana Naia, Jorge Valero, Susanna Campesan, Carla Lopes, Victoria E. Cotton, Catarina Carmo, Tatiana R. Rosenstock, A. Cristina Rego
Publikováno v:
Free radical biologymedicine. 163
SIRT3 is a major regulator of mitochondrial acetylome. Here we show that SIRT3 is neuroprotective in Huntington's disease (HD), a motor neurodegenerative disorder caused by an abnormal expansion of polyglutamines in the huntingtin protein (HTT). Prot
Autor:
Martins, Ana Catarina Carmo
Publikováno v:
Repositório Científico de Acesso Aberto de Portugal
Repositório Científico de Acesso Aberto de Portugal (RCAAP)
instacron:RCAAP
Repositório Científico de Acesso Aberto de Portugal (RCAAP)
instacron:RCAAP
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Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od______3056::5557941c5e8c9c12354883e162ebafcc
Publikováno v:
Methods in molecular biology (Clifton, N.J.). 2004
Structural maintenance of chromosomes (SMC) proteins are critical to maintain mitotic fidelity in all organisms. Over the last decades, acute inactivation of these complexes, together with the analysis of their dynamic binding to mitotic chromatin, h
Autor:
Michael R. Hayden, Mário N. Laço, Gladys L. Caldeira, A. Cristina Rego, Luana Naia, Ana Oliveira, Tatiana R. Rosenstock, Sofia I. Oliveira-Sousa, Catarina Carmo, Catarina R. Oliveira
Publikováno v:
Molecular Neurobiology. 54:5385-5399
Sirtuin 1 (SIRT1) is a nicotinamide adenine dinucleotide (NAD+)-dependent lysine deacetylase that regulates longevity and enhances mitochondrial metabolism. Both activation and inhibition of SIRT1 were previously shown to ameliorate neuropathological
Publikováno v:
Methods in molecular biology (Clifton, N.J.). 1780
Mitochondrial dysfunction has gained a preponderant role in the pathogenesis of Huntington's disease (HD). Mutant huntingtin (mHTT) directly interacts with mitochondria in a deleterious manner. As the central hub of the cell, not only mitochondrial b
Publikováno v:
Polyglutamine Disorders ISBN: 9783319717784
Mitochondrial dysfunction has been described as an early pathological mechanism delineating the selective neurodegeneration that occurs in Huntington's disease (HD), a polyglutamine-expansion disorder that largely affects the striatum and the cerebra
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::e1ec4b5e94c46bf947cf7e8977abc844
https://doi.org/10.1007/978-3-319-71779-1_3
https://doi.org/10.1007/978-3-319-71779-1_3
Publikováno v:
Methods in Molecular Biology ISBN: 9781493978243
Mitochondrial dysfunction has gained a preponderant role in the pathogenesis of Huntington's disease (HD). Mutant huntingtin (mHTT) directly interacts with mitochondria in a deleterious manner. As the central hub of the cell, not only mitochondrial b
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::d08bce161c34e11e79a5ffdaee09ce2f
https://doi.org/10.1007/978-1-4939-7825-0_19
https://doi.org/10.1007/978-1-4939-7825-0_19