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Autor:
Ching-Man A. Virbasius, Natalia Sharova, Alissa G. Bukrinskaya, Xiaochun Zhu, Mario Stevenson, Michael R. Green, Caryn DeHoratius, Stefan A. Dupont
Publikováno v:
Nature. 402:681-685
An important aspect of the pathophysiology of human immunodeficiency virus type-1 (HIV-1) infection is the ability of the virus to replicate in non-dividing cells1,2,3. HIV-1 matrix (MA), the amino-terminal domain of the Pr55 gag polyprotein (Pr55),